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Kiwifruit (Actinidia spp.) postharvest decay is common in China, which can cause serious economic losses to kiwifruit industry. In order to further clarify the pathogen of kiwifruit rot disease in Guizhou Province, the rotten fruits of kiwifruit (cultivar "Jinyan") were collected, and the pathogenic fungi were identified by isolation and purification, pathogenicity test, morphological characteristics, and analysis of rDNA-ITS sequences. The results showed that the pathogenic fungi of kiwifruit rot disease were Diaporthe phaseolorum and Fusarium tricinctum. Meanwhile, the results showed that all the tested agents had a certain inhibitory effect on Diaporthe phaseolorum and Fusarium tricinctum. Among them, 33.5% quinolone SC had the best inhibitory effect on Diaporthe phaseolorum with an EC50 value of 9.67 mg/L, and 25% fludioxonil SC had the best inhibitory effect on Fusarium tridentatus with the EC50 value of 13.13 mg/L. The results will provide a reference for the control of kiwifruit rot disease. [ABSTRACT FROM AUTHOR]

The precise pathophysiological mechanisms of preeclampsia (PE) and preventative strategies remain unknown. Laboratory markers which can help in identifying PE patients from pregnant women and assessing the severity of PE during pregnancy are worthy to be explored. In this study, a retrospective case-control study was designed to assess whether the serum levels of albumin (ALB), total protein (TP), prealbumin (PA), alkaline phosphatase (ALP), lactic dehydrogenase (LDH), D-dimer, fibrinogen (Fbg), platelet (PLT) count, mean platelet volume (MPV), and platelet distribution width (PDW) can help in assessing PE and evaluate its severity. 256 pregnant women were enrolled and classified into 3 groups: mild preeclampsia (mPE, n = 85), severe preeclampsia (sPE, n = 78), and healthy normotensive controls (control, n = 93). Our result showed that the serum levels of ALP, LDH, and D-dimer were significantly higher in mild or severe PE patients compared with the healthy controls (66 (52.5-76.5) vs. 168 (141.5-201.25) vs. 182.5 (120-191.5), 152 (139.75-166.25) vs. 183.5 (163.25-307) vs. 282 (215.25-306), 1.05 (0.65-1.57) vs. 3.05 (2.25-4.08) vs. 5.65 (2.29-7.71)), while ALB, TP, and PA are lower (38 (37-42) vs. 31.5 (25.5-34.5) vs. 28.5 (24-33), 65 (63-68.25) vs. 56.5 (52-61) vs. 51.5 (49-58), 219.14 ± 68.25 vs. 167.88 ± 52.21 vs. 143.22 ± 50.46). On the other hand, compared with the mPE group, the sPE group showed significantly lower PLT count but higher level of LDH, D-dimer, and Fbg. No significant differences in MPV or PDW were found between any of the two groups. In conclusion, the above markers except for the MPV and PDW may be correlated with PE severity in this patient cohort, indicating possible values of these potential biomarkers in auxiliary diagnosis and severity assessment of PE. [ABSTRACT FROM AUTHOR]