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Overall, we observed 5 (3%) cystic fibrosis, 10 (5%) other genetic disorders, 13 (7%) congenital infections, 5 (3%) neonatal deaths, 12 (7%) intestinal anomalies, 24 (13%) other malformations, and 19 (10%) experienced IUGR. Fetal and neonatal demise, intrauterine growth retardation (IUGR), associated anomalies, genetic disorders (including cystic fibrosis) and congenital infection were evaluated. When persistent HB were present, an increase rate of congenital infection and bowel dilatation were observed (14% versus 4%; p = 0,03 and 52% versus 23%; p = 0,0007, respectively). [Extracted from the article]

It is reported that many infants with CDH have evidence of brain injury on postnatal brain magnetic resonance imaging (MRI). Results HT

Objective:With increasing use of routine prenatal ultrasound, prenatal counseling after diagnosis of congenital malformations is frequently offered to prospective parents. We aimed to assess if the communication of diagnosis of a congenital anomaly in the fetus meets American Psychiatric Association (APA) criteria for trauma in parents.Study Design:In the period ranging from 2003 to 2009 a preliminary investigation was conducted with 165 prospective mothers and 91 prospective fathers being interviewed after communication of diagnosis. Analysis of statements was made independently by two psychologists considering the APA definition of trauma.Result:A total of 145 mothers and 76 fathers experienced the communication of diagnosis in their fetus as a traumatic event. There was no correlation between type of malformation and trauma nor was there statistical difference between mother and father regarding the stressor.Conclusion:Communication of diagnosis of a fetal anomaly can be a traumatic event and should be dealt with consequently. Given the therapeutic value of sharing traumatic experience such practice should be encouraged as part of the consultation process. [ABSTRACT FROM AUTHOR]

The article present a case study of a male infant who developed vesicles and blisters on the face and diaper area after five days of his birth. Antibiotic therapy could not stop skin lesions and respiratory symptoms to worsen, he was diagnosed with linear IgA bullous dermatosis, and was treated with corticosteroids, immunoglobulins and dapsone.