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Introduction: The Belt and Road Initiative (BRI) is an important cooperative framework that increasingly affects the global economy, trade, and emission patterns. However, most existing studies pay insufficient attention to consumption-based emissions, embodied emissions, and non-CO2 greenhouse gases (GHGs). This study constructs a GHG emissions database to study the trends and variations in production-based, consumption-based, and embodied emissions associated with BRI countries.Outcome: We find that the per capita GHG emissions of BRI countries are lower than the global average but show significant variation within this group. We also find that trade-embodied emissions between BRI countries and China are growing. As a group, BRI countries are anet exporter of GHGs, with a global share of net export emissions of about 20%. In 2011, nearly 80% of GHG export emissions from BRI countries flowed to non-BRI countries, and nearly 15% flowed to China; about 57% of GHG import emissions were from non-BRI countries, and about 38% were from China.Conclusion: Therefore, this study concludes that the BRI should be used to coordinate climate governance to accelerate and strengthen the dissemination and deployment of low-emissions technologies, strategies, and policies within the BRI so as to avoid a carbon-intensive lock-in effect. [ABSTRACT FROM AUTHOR]

Eco-cities have attracted international attention from governments, corporations, academics and other actors seeking to use sustainable urban planning to reduce urban environmental impacts. China has devoted significant political will and economic resources to the development of new-build eco-city projects, reflecting the Chinese government's goals to build a 'harmonious society' in which environmental sustainability and social stability are mutually reinforcing. We critically analyse the case of the Sino-Singapore Tianjin eco-city to demonstrate that the eco-city's ecologically modernizing visions of eco-urbanism construct a protective environment for its residents that constrains broader consideration of social sustainability. Through analysis of the marketing and presentation of specific domestic and other spaces of the eco-city, we examine the application of ecologically modernizing construction and technology to the design of the city. We argue that the eco-city is discursively constructed as ecologically beneficial for its inhabitants rather than for the broader socio-environmental landscape. Our analysis of residential spaces in Tianjin eco-city introduces the question of what 'eco' means when considering the construction of eco-urban environments for the city's residents. [ABSTRACT FROM AUTHOR]

BACKGROUND: Bis-(2-ethylhexyl) tetrabromophthalate (TBPH) is widely used as a replacement for polybrominated diphenyl ethers (PBDEs) in commercial flame retardant mixtures such as Firemaster 550. It is also used in a commercial mixture called DP 45. Mono-(2-ethyhexyl) tetrabromophthalate (TBMEHP) is a potentially toxic metabolite. OBJECTIVES: We used in vitro and rodent in vivo models to evaluate human exposure and the potential metabolism and toxicity of TBPH. METHODS: Dust collected from homes, offices, and cars was measured for TBPH by gas chromatography followed by mass spectrometry. Pregnant rats were gavaged with TBMEHP (200 or 500 mg/kg) or corn oil on gestational days 18 and 19, and dams and fetuses were evaluated histologically for toxicity. We also assessed TBMEHP for deiodinase inhibition using rat liver microsomes and for peroxisome proliferator-activated receptor (PPAR) Α and γ activation using murine FAO cells and NIH 3T3 L1 cells. RESULTS: TBPH concentrations in dust from office buildings (median, 410 ng/g) were higher than in main living areas in homes (median, 150 ng/g). TBPH was metabolized by purified porcine esterases to TBMEHP. Two days of TBMEHP exposure in the rat produced maternal hypothyroidism with markedly decreased serum T3 (3,3',5-triiodo-L-thyronine), maternal hepatotoxicity, and increased multinucleated germ cells (MNGs) in fetal testes without antiandrogenic effects. In vitro, TBMEHP inhibited deiodinase activity, induced adipocyte differentiation in NIH 3T3 L1 cells, and activated PPARΑ- and PPARγ-mediated gene transcription in NIH 3T3 L1 cells and FAO cells, respectively. CONCLUSIONS: TBPH a) is present in dust from indoor environments (implying human exposure) and b) can be metabolized by porcine esterases to TBMEHP, which c) elicited maternal thyrotoxic and hepatotoxic effects and d) induced MNGs in the fetal testes in a rat model. In mouse NIH 3T3 L1 preadipocyte cells, TBMEHP inhibited rat hepatic microsome deiodinase activity and was an agonist for PPARs in murine FAO and NIH 3T3 L1 cells. [ABSTRACT FROM AUTHOR]