18 results on '"Zhou, Xu-jie"'
Search Results
2. Genome-Wide Association Study in Acute Tubulointerstitial Nephritis
3. Glomerulonephritis triggered by chronical aortic graft infection in a male with Loeys–Dietz syndrome: A case report
4. Plasma exchange and rituximab treatments in primary membranous nephropathy combined with crescentic glomerulonephritis: A case report
5. A case report of remission of refractory membranous nephropathy progressing to stage 4 chronic kidney disease using low-dose rituximab: A long-term follow-up
6. A Rare Variant (rs933717) at FBXO31‐MAP1LC3B in Chinese Is Associated With Systemic Lupus Erythematosus
7. Confirmation of five novel susceptibility loci for systemic lupus erythematosus (SLE) and integrated network analysis of 82 SLE susceptibility loci
8. Association of variants in CCR6 with susceptibility to lupus nephritis in Chinese
9. Biomarkers in assessing disease activity in patients with eosinophilic granulomatosis with polyangiitis
10. Human neutrophil peptide 1–3, a component of the neutrophil extracellular trap, as a potential biomarker of lupus nephritis
11. Brief Report: Identification of MTMR3 as a Novel Susceptibility Gene for Lupus Nephritis in Northern Han Chinese by Shared-Gene Analysis With IgA Nephropathy
12. Rituximab, a viable alternative for induction therapy of active lupus nephritis
13. Higher DEFB4 genomic copy number in SLE and ANCA-associated small vasculitis
14. Gene–gene interaction of BLK, TNFSF4, TRAF1, TNFAIP3, and REL in systemic lupus erythematosus
15. Rs548234 polymorphism at PRDM1-ATG5 region susceptible to rheumatoid arthritis in Caucasians is not associated with rheumatoid arthritis in Chinese Han population
16. Genetic association of PRDM1-ATG5 intergenic region and autophagy with systemic lupus erythematosus in a Chinese population
17. FCGR2B gene polymorphism rather than FCGR2A, FCGR3A and FCGR3B is associated with anti-GBM disease in Chinese
18. Copy number variation of FCGR3A rather than FCGR3B and FCGR2B is associated with susceptibility to anti-GBM disease
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