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1. HIV-1 adapts to lost IP6 coordination through second-site mutations that restore conical capsid assembly.

2. Human coronaviruses activate and hijack the host transcription factor HSF1 to enhance viral replication.

3. A pan-SARS-CoV-2-specific soluble angiotensin-converting enzyme 2-albumin fusion engineered for enhanced plasma half-life and needle-free mucosal delivery.

4. Multivalent bicyclic peptides are an effective antiviral modality that can potently inhibit SARS-CoV-2.

5. IP6-stabilised HIV capsids evade cGAS/STING-mediated host immune sensing.

6. HIV-1 is dependent on its immature lattice to recruit IP6 for mature capsid assembly.

7. TRIM7 Restricts Coxsackievirus and Norovirus Infection by Detecting the C-Terminal Glutamine Generated by 3C Protease Processing.

8. Analysis of Serological Biomarkers of SARS-CoV-2 Infection in Convalescent Samples From Severe, Moderate and Mild COVID-19 Cases.

9. SARS-CoV-2 B.1.617.2 Delta variant replication and immune evasion.

10. Single-dose immunisation with a multimerised SARS-CoV-2 receptor binding domain (RBD) induces an enhanced and protective response in mice.

11. A functional assay for serum detection of antibodies against SARS-CoV-2 nucleoprotein.

12. Furin cleavage of SARS-CoV-2 Spike promotes but is not essential for infection and cell-cell fusion.

13. SARS-CoV-2 Infects the Brain Choroid Plexus and Disrupts the Blood-CSF Barrier in Human Brain Organoids.

14. Periphilin self-association underpins epigenetic silencing by the HUSH complex.

15. TASOR is a pseudo-PARP that directs HUSH complex assembly and epigenetic transposon control.

16. MAVS polymers smaller than 80 nm induce mitochondrial membrane remodeling and interferon signaling.

17. Dual Function of the pUL7-pUL51 Tegument Protein Complex in Herpes Simplex Virus 1 Infection.

18. HSV-1 Glycoproteins Are Delivered to Virus Assembly Sites Through Dynamin-Dependent Endocytosis.

19. Structural analysis of herpes simplex virus by optical super-resolution imaging.

20. Analysis of serine codon conservation reveals diverse phenotypic constraints on hepatitis C virus glycoprotein evolution.

21. Additional glycosylation within a specific hypervariable region of subtype 3a of hepatitis C virus protects against virus neutralization.

22. Role of low-density lipoprotein receptor in the hepatitis C virus life cycle.

23. (-)-Epigallocatechin-3-gallate is a new inhibitor of hepatitis C virus entry.

24. Griffithsin has antiviral activity against hepatitis C virus.

25. Hepatitis C patient-derived glycoproteins exhibit marked differences in susceptibility to serum neutralizing antibodies: genetic subtype defines antigenic but not neutralization serotype.

26. Identification of new functional regions in hepatitis C virus envelope glycoprotein E2.

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