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9. Iron is a modifier of the phenotypes of JAK2-mutant myeloproliferative neoplasms

12. Real-world study of children and young adults with myeloproliferative neoplasms: identifying risks and unmet needs

21. JAK2-mutant hematopoietic cells display metabolic alterations that can be targeted to treat myeloproliferative neoplasms

25. Ruxolitinib-induced defects in DNA repair cause sensitivity to PARP inhibitors in myeloproliferative neoplasms

36. Genomic profiling for clinical decision making in myeloid neoplasms and acute leukemia

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