18 results on '"Abrignani S"'
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2. Molecular analysis of V HI + B lymphocytes in hepatitis C patients
3. The combination of IFN-lambda-4 and HLA-DPB1 polymorphisms accurately predict HBsAg loss in IFN treated genotype D HBeAg-negative patients with chronic hepatitis B
4. HLA DPB1 rs9277535 polymorphism strongly predicts HBsAg clearance in interferon treated genotype D HBeAg-negative patients with chronic hepatitis B
5. P.02.11 PHENOTYPIC AND FUNCTIONAL CHARACTERIZATION OF INFLAMMATORY CELL INFILTRATE IN ADULT-ONSET AUTOIMMUNE ENTEROPATHY AND ITS EVOLUTION WITH GLUCOCORTICOIDS
6. P.05.9 PHENOTYPIC AND FUNCTIONAL CHARACTERIZATION OF INTRAEPITHELIAL AND LAMINA PROPRIA INFLAMMATORY CELL INFILTRATE IN AUTOIMMUNE ENTEROPATHY
7. OC.06.1 INFLIXIMAB-INDUCED MUCOSAL HEALING ASSOCIATES WITH A DOWN-REGULATION OF IL23/TH17 IN INFLAMMATORY BOWEL DISEASES
8. CS15-7. Decrease of IL-17 and Gm-Csf in Intestinal Mucosa of Ibd Patients is Associated With Reduction of CD68+ Macrophages and Clinical Remission in Response to Anti-TNF Therapy
9. P.1.14: INFLIXIMAB MODULATES IL23-TH17 AXIS IN PATIENTS WITH INFLAMMATORY BOWEL DISEASES
10. OC.09.6: GENETIC VARIATION IN IL28B POLYMORPHISM MAY PREDICT HBSAG CLEARANCE IN GENOTYPE D, HBEAG NEGATIVE PATIENTS TREATED WITH INTERFERON ALFA
11. T-37 Distribution of the IL28B rs12979860 polymorphism and its influence on sustained virological response rates in the MIST trial
12. T-36 Fibrosis progression in patients with chronic hepatitis C is not influenced by IL28B polymorphisms
13. OC-2 Genetic variation in IL28B polymorphism may predict HBsAg clearance in genotype D, HBeAg negative patients treated with interferon alfa
14. OC-9 The presence of hepatitis C virus deletion mutants in chronic hepatitis C patients and their role during PEG-IFN/ribavirin therapy
15. P.62 INFLIXIMAB MODULATES MUCOSAL CYTOKINE PROFILE IN PATIENTS WITH INFLAMMATORY BOWEL DISEASES
16. The candidate HCV E1E2MF59 vaccine enhances 2nd phase viral decline in responders to PEG-IFN/ribavirin
17. Corrigendum to “Chlamydia pneumoniae genome sequence analysis and identification of HLA-A2-restricted CD8(+) T cell epitopes recognized by infection-primed T cells” [Vaccine 23 (2005) 5028–5037]
18. 61 BCR-independent activation of naive B cell via CD81: Possible pathogenic mechanism of HCV associated B-lymphocyte disorders
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