16 results on '"Beyersdorf, Niklas"'
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2. Quorum Regulation via Nested Antagonistic Feedback Circuits Mediated by the Receptors CD28 and CTLA-4 Confers Robustness to T Cell Population Dynamics
3. The secreted Candida albicans protein Pra1 disrupts host defense by broadly targeting and blocking complement C3 and C3 activation fragments
4. CD4+ Foxp3+ T-cells contribute to myocardial ischemia-reperfusion injury
5. The T cell-selective IL-2 mutant AIC284 mediates protection in a rat model of Multiple Sclerosis
6. CD8+ T cell help is required for efficient induction of EAE in Lewis rats
7. Superagonistic CD28 stimulation of allogeneic T cells protects from acute graft-versus-host disease
8. Protection from graft-versus-host disease with a novel B7 binding site–specific mouse anti–mouse CD28 monoclonal antibody
9. CD28 superagonists put a break on autoimmunity by preferentially activating CD4 +CD25 + regulatory T cells
10. Analyzing trogocytosis of T lymphocytes by flow cytometry and confocal microscopy
11. Reply to Kang and Brooks: Comment on the interpretation of binding of Pra1, the fungal immune evasion protein from Candida albicans to the human C3 and on the conformational changes of C3 upon activation: Kang and Brooks Optimization of biolayer-interferometry-based binding assay of he interaction between the Candida albicans protein Pra1 and complement protein C3
12. Wasp venom immunotherapy expands a subpopulation of CD4+CD25+ forkhead box protein 3–positive regulatory T cells expressing the T-cell receptor Vβ2 and Vβ5.1 chains
13. Candida albicans’ pH regulated antigen 1 (Pra1) preferentially targets and inactivates human complement C3 and C3 activation fragments to evade host immune attack
14. Candia Pra1 blocks human CD4 T cell activation by ligation of CD46
15. OR.93. Superagonistic Ex Vivo and In Vivo Stimulation of T Lymphocytes by Novel Human Cd28-specific Monoclonal Antibodies
16. Superagonistic Anti-CD28 Antibody TGN1412 as a Potential Immunotherapeutic for the Treatment of B Cell Chronic Lymphocytic Leukemia.
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