12 results on '"Beyer, Frauke"'
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2. Prebiotic diet changes neural correlates of food decision-making in overweight adults: a randomised controlled within-subject cross-over trial
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Medawar, Evelyn, Beyer, Frauke, Thieleking, Ronja, Haange, Sven-Bastiaan, Rolle-Kampczyk, Ulrike, Reinicke, Madlen, Chakaroun, Rima, von Bergen, Martin, Stumvoll, Michael, Villringer, Arno, and Witte, A Veronica
- Abstract
ObjectiveAnimal studies suggest that prebiotic, plant-derived nutrients could improve homoeostatic and hedonic brain functions through improvements in microbiome–gut–brain communication. However, little is known if these results are applicable to humans. Therefore, we tested the effects of high-dosed prebiotic fibre on reward-related food decision-making in a randomised controlled within-subject cross-over study and assayed potential microbial and metabolic markers.Design59 overweight young adults (19 females, 18–42 years, body mass index 25–30 kg/m2) underwent functional task MRI before and after 14 days of supplementary intake of 30 g/day of inulin (prebiotics) and equicaloric placebo, respectively. Short chain fatty acids (SCFA), gastrointestinal hormones, glucose/lipid and inflammatory markers were assayed in fasting blood. Gut microbiota and SCFA were measured in stool.ResultsCompared with placebo, participants showed decreased brain activation towards high-caloric wanted food stimuli in the ventral tegmental area and right orbitofrontal cortex after prebiotics (preregistered, family wise error-corrected p <0.05). While fasting blood levels remained largely unchanged, 16S-rRNA sequencing showed significant shifts in the microbiome towards increased occurrence of, among others, SCFA-producing Bifidobacteriaceae, and changes in >60 predicted functional signalling pathways after prebiotic intake. Changes in brain activation correlated with changes in Actinobacteriamicrobial abundance and associated activity previously linked with SCFA production, such as ABC transporter metabolism.ConclusionsIn this proof-of-concept study, a prebiotic intervention attenuated reward-related brain activation during food decision-making, paralleled by shifts in gut microbiota.Trial registration numberNCT03829189.
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- 2024
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3. Genetic variants for head size share genes and pathways with cancer
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Knol, Maria J., Poot, Raymond A., Evans, Tavia E., Satizabal, Claudia L., Mishra, Aniket, Sargurupremraj, Muralidharan, van der Auwera, Sandra, Duperron, Marie-Gabrielle, Jian, Xueqiu, Hostettler, Isabel C., van Dam-Nolen, Dianne H.K., Lamballais, Sander, Pawlak, Mikolaj A., Lewis, Cora E., Carrion-Castillo, Amaia, van Erp, Theo G.M., Reinbold, Céline S., Shin, Jean, Scholz, Markus, Håberg, Asta K., Kämpe, Anders, Li, Gloria H.Y., Avinun, Reut, Atkins, Joshua R., Hsu, Fang-Chi, Amod, Alyssa R., Lam, Max, Tsuchida, Ami, Teunissen, Mariël W.A., Aygün, Nil, Patel, Yash, Liang, Dan, Beiser, Alexa S., Beyer, Frauke, Bis, Joshua C., Bos, Daniel, Bryan, R. Nick, Bülow, Robin, Caspers, Svenja, Catheline, Gwenaëlle, Cecil, Charlotte A.M., Dalvie, Shareefa, Dartigues, Jean-François, DeCarli, Charles, Enlund-Cerullo, Maria, Ford, Judith M., Franke, Barbara, Freedman, Barry I., Friedrich, Nele, Green, Melissa J., Haworth, Simon, Helmer, Catherine, Hoffmann, Per, Homuth, Georg, Ikram, M. Kamran, Jack, Clifford R., Jahanshad, Neda, Jockwitz, Christiane, Kamatani, Yoichiro, Knodt, Annchen R., Li, Shuo, Lim, Keane, Longstreth, W.T., Macciardi, Fabio, Amouyel, Philippe, Arfanakis, Konstantinos, Aribisala, Benjamin S., Bastin, Mark E., Chauhan, Ganesh, Chen, Christopher, Cheng, Ching-Yu, de Jager, Philip L., Deary, Ian J., Fleischman, Debra A., Gottesman, Rebecca F., Gudnason, Vilmundur, Hilal, Saima, Hofer, Edith, Janowitz, Deborah, Jukema, J. Wouter, Liewald, David C.M., Lopez, Lorna M., Lopez, Oscar, Luciano, Michelle, Martinez, Oliver, Niessen, Wiro J., Nyquist, Paul, Rotter, Jerome I., Rundek, Tatjana, Sacco, Ralph L., Schmidt, Helena, Tiemeier, Henning, Trompet, Stella, van der Grond, Jeroen, Völzke, Henry, Wardlaw, Joanna M., Yanek, Lisa, Yang, Jingyun, Agartz, Ingrid, Alhusaini, Saud, Almasy, Laura, Ames, David, Amunts, Katrin, Andreassen, Ole A., Armstrong, Nicola, Bernard, Manon, Blangero, John, Blanken, Laura M.E., Boks, Marco P., Boomsma, Dorret I., Brickman, Adam M., Brodaty, Henry, Buckner, Randy L., Buitelaar, Jan K., Cannon, Dara M., Carr, Vaughan J., Catts, Stanley V., Chakravarty, M. Mallar, Chen, Qiang, Ching, Christopher R.K., Corvin, Aiden, Crespo-Facorro, Benedicto, Curran, Joanne E., Davies, Gareth E., de Geus, Eco J.C., de Zubicaray, Greig I., den Braber, Anouk, Desrivières, Sylvane, Dillman, Allissa, Djurovic, Srdjan, Drevets, Wayne C., Duggirala, Ravi, Ehrlich, Stefan, Erk, Susanne, Espeseth, Thomas, Fedko, Iryna O., Fernández, Guillén, Fisher, Simon E., Foroud, Tatiana M., Ge, Tian, Giddaluru, Sudheer, Glahn, David C., Goldman, Aaron L., Green, Robert C., Greven, Corina U., Grimm, Oliver, Hansell, Narelle K., Hartman, Catharina A., Hashimoto, Ryota, Heinz, Andreas, Henskens, Frans, Hibar, Derrek P., Ho, Beng-Choon, Hoekstra, Pieter J., Holmes, Avram J., Hoogman, Martine, Hottenga, Jouke-Jan, Hulshoff Pol, Hilleke E., Jablensky, Assen, Jenkinson, Mark, Jia, Tianye, Jöckel, Karl-Heinz, Jönsson, Erik G., Kim, Sungeun, Klein, Marieke, Kochunov, Peter, Kwok, John B., Lawrie, Stephen M., Le Hellard, Stephanie, Lemaître, Hervé, Loughland, Carmel, Marquand, Andre F., Martin, Nicholas G., Martinot, Jean-Luc, Matarin, Mar, Mathalon, Daniel H., Mather, Karen A., Mattay, Venkata S., McDonald, Colm, McMahon, Francis J., McMahon, Katie L., E, Rebekah, McWhirter, Mecocci, Patrizia, Melle, Ingrid, Meyer-Lindenberg, Andreas, Michie, Patricia T., Milaneschi, Yuri, Morris, Derek W., Mowry, Bryan, Nho, Kwangsik, Nichols, Thomas E., Nöthen, Markus N., Olvera, Rene L., Oosterlaan, Jaap, Ophoff, Roel A., Pandolfo, Massimo, Pantelis, Christos, Pappa, Irene, Penninx, Brenda, Pike, G. Bruce, Rasser, Paul E., Rentería, Miguel E., Reppermund, Simone, Rietschel, Marcella, Risacher, Shannon L., Romanczuk-Seiferth, Nina, Rose, Emma Jane, Sachdev, Perminder S., Sämann, Philipp G., Saykin, Andrew J., Schall, Ulrich, Schofield, Peter R., Schramm, Sara, Schumann, Gunter, Scott, Rodney, Shen, Li, Sisodiya, Sanjay M., Soininen, Hilkka, Sprooten, Emma, Srikanth, Velandai, Steen, Vidar M., Strike, Lachlan T., Thalamuthu, Anbupalam, Toga, Arthur W., Tooney, Paul, Tordesillas-Gutiérrez, Diana, Turner, Jessica A., Valdés Hernández, Maria del C., van der Meer, Dennis, Van der Wee, Nic J.A., Van Haren, Neeltje E.M., van 't Ent, Dennis, Veltman, Dick J., Walter, Henrik, Weinberger, Daniel R., Weiner, Michael W., Wen, Wei, Westlye, Lars T., Westman, Eric, Winkler, Anderson M., Woldehawariat, Girma, Wright, Margaret J., Wu, Jingqin, Mäkitie, Outi, Mazoyer, Bernard, Medland, Sarah E., Miyamoto, Susumu, Moebus, Susanne, Mosley, Thomas H., Muetzel, Ryan, Mühleisen, Thomas W., Nagata, Manabu, Nakahara, Soichiro, Palmer, Nicholette D., Pausova, Zdenka, Preda, Adrian, Quidé, Yann, Reay, William R., Roshchupkin, Gennady V., Schmidt, Reinhold, Schreiner, Pamela J., Setoh, Kazuya, Shapland, Chin Yang, Sidney, Stephen, St Pourcain, Beate, Stein, Jason L., Tabara, Yasuharu, Teumer, Alexander, Uhlmann, Anne, van der Lugt, Aad, Vernooij, Meike W., Werring, David J., Windham, B. Gwen, Witte, A. Veronica, Wittfeld, Katharina, Yang, Qiong, Yoshida, Kazumichi, Brunner, Han G., Le Grand, Quentin, Sim, Kang, Stein, Dan J., Bowden, Donald W., Cairns, Murray J., Hariri, Ahmad R., Cheung, Ching-Lung, Andersson, Sture, Villringer, Arno, Paus, Tomas, Cichon, Sven, Calhoun, Vince D., Crivello, Fabrice, Launer, Lenore J., White, Tonya, Koudstaal, Peter J., Houlden, Henry, Fornage, Myriam, Matsuda, Fumihiko, Grabe, Hans J., Ikram, M. Arfan, Debette, Stéphanie, Thompson, Paul M., Seshadri, Sudha, and Adams, Hieab H.H.
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The size of the human head is highly heritable, but genetic drivers of its variation within the general population remain unmapped. We perform a genome-wide association study on head size (N = 80,890) and identify 67 genetic loci, of which 50 are novel. Neuroimaging studies show that 17 variants affect specific brain areas, but most have widespread effects. Gene set enrichment is observed for various cancers and the p53, Wnt, and ErbB signaling pathways. Genes harboring lead variants are enriched for macrocephaly syndrome genes (37-fold) and high-fidelity cancer genes (9-fold), which is not seen for human height variants. Head size variants are also near genes preferentially expressed in intermediate progenitor cells, neural cells linked to evolutionary brain expansion. Our results indicate that genes regulating early brain and cranial growth incline to neoplasia later in life, irrespective of height. This warrants investigation of clinical implications of the link between head size and cancer.
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- 2024
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4. Higher BMI, but not obesity-related genetic polymorphisms, correlates with lower structural connectivity of the reward network in a population-based study
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Beyer, Frauke, Zhang, Rui, Scholz, Markus, Wirkner, Kerstin, Loeffler, Markus, Stumvoll, Michael, Villringer, Arno, and Witte, A. Veronica
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Background: Obesity is of complex origin, involving genetic and neurobehavioral factors. Genetic polymorphisms may increase the risk for developing obesity by modulating dopamine-dependent behaviors, such as reward processing. Yet, few studies have investigated the association of obesity, related genetic variants, and structural connectivity of the dopaminergic reward network. Methods: We analyzed 347 participants (age range: 20–59 years, BMI range: 17–38 kg/m
2 ) of the LIFE-Adult Study. Genotyping for the single nucleotid polymorphisms rs1558902 (FTO) and rs1800497 (near dopamine D2 receptor) was performed on a microarray. Structural connectivity of the reward network was derived from diffusion-weighted magnetic resonance imaging at 3 T using deterministic tractography of Freesurfer-derived regions of interest. Using graph metrics, we extracted summary measures of clustering coefficient and connectivity strength between frontal and striatal brain regions. We used linear models to test the association of BMI, risk alleles of both variants, and reward network connectivity. Results: Higher BMI was significantly associated with lower connectivity strength for number of streamlines (β= −0.0025, 95%—C.I.: [−0.004, −0.0008], p= 0.0042), and, to lesser degree, fractional anisotropy (β= −0.0009, 95%—C.I. [−0.0016, −0.00008], p= 0.031), but not clustering coefficient. Strongest associations were found for left putamen, right accumbens, and right lateral orbitofrontal cortex. As expected, the polymorphism rs1558902 in FTO was associated with higher BMI (F= 6.9, p< 0.001). None of the genetic variants was associated with reward network structural connectivity. Conclusions: Here, we provide evidence that higher BMI correlates with lower reward network structural connectivity. This result is in line with previous findings of obesity-related decline in white matter microstructure. We did not observe an association of variants in FTO or near DRD2 receptor with reward network structural connectivity in this population-based cohort with a wide range of BMI and age. Future research should further investigate the link between genetics, obesity and fronto-striatal structural connectivity.- Published
- 2021
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5. Atypical brain aging and its association with working memory performance in major depressive disorder
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Ho, Natalie C.W., Bethlehem, Richard AI., Seidlitz, Jakob, Nogovitsyn, Nikita, Metzak, Paul, Ballester, Pedro L., Hassel, Stefanie, Rotzinger, Susan, Poppenk, Jordan, Lam, Raymond W., Taylor, Valerie H., Milev, Roumen, Adamson, Chris, Adler, Sophie, Alexander-Bloch, Aaron F., Anagnostou, Evdokia, Anderson, Kevin M., Areces-Gonzalez, Ariosky, Astle, Duncan E., Auyeung, Bonnie, Ayub, Muhammad, Bae, Jong Bin, Ball, Gareth, Baron-Cohen, Simon, Beare, Richard, Bedford, Saashi A., Benegal, Vivek, Bethlehem, Richard A.I., Beyer, Frauke, Blangero, John, Blesa Cábez, Manuel, Boardman, James P., Borzage, Matthew, Bosch-Bayard, Jorge F., Bourke, Niall, Bullmore, Edward T., Calhoun, Vince D., Chakravarty, Mallar M., Chen, Christina, Chertavian, Casey, Chetelat, Gaël, Chong, Yap S., Corvin, Aiden, Costantino, Manuela, Courchesne, Eric, Crivello, Fabrice, Cropley, Vanessa L., Crosbie, Jennifer, Crossley, Nicolas, Delarue, Marion, Delorme, Richard, Desrivieres, Sylvane, Devenyi, Gabriel, Di Biase, Maria A., Dolan, Ray, Donald, Kirsten A., Donohoe, Gary, Dorfschmidt, Lena, Dunlop, Katharine, Edwards, Anthony D., Elison, Jed T., Ellis, Cameron T., Elman, Jeremy A., Eyler, Lisa, Fair, Damien A., Fletcher, Paul C., Fonagy, Peter, Franz, Carol E., Galan-Garcia, Lidice, Gholipour, Ali, Giedd, Jay, Gilmore, John H., Glahn, David C., Goodyer, Ian M., Grant, P.E., Groenewold, Nynke A., Gudapati, Shreya, Gunning, Faith M., Gur, Raquel E., Gur, Ruben C., Hammill, Christopher F., Hansson, Oskar, Hedden, Trey, Heinz, Andreas, Henson, Richard N., Heuer, Katja, Hoare, Jacqueline, Holla, Bharath, Holmes, Avram J., Huang, Hao, Ipser, Jonathan, Jack, Clifford R., Jackowski, Andrea P., Jia, Tianye, Jones, David T., Jones, Peter B., Kahn, Rene S., Karlsson, Hasse, Karlsson, Linnea, Kawashima, Ryuta, Kelley, Elizabeth A., Kern, Silke, Kim, Ki-Woong, Kitzbichler, Manfred G., Kremen, William S., Lalonde, François, Landeau, Brigitte, Lerch, Jason, Lewis, John D., Li, Jiao, Liao, Wei, Liston, Conor, Lombardo, Michael V., Lv, Jinglei, Mallard, Travis T., Marcelis, Machteld, Mathias, Samuel R., Mazoyer, Bernard, McGuire, Philip, Meaney, Michael J., Mechelli, Andrea, Misic, Bratislav, Morgan, Sarah E., Mothersill, David, Ortinau, Cynthia, Ossenkoppele, Rik, Ouyang, Minhui, Palaniyappan, Lena, Paly, Leo, Pan, Pedro M., Pantelis, Christos, Park, Min Tae M., Paus, Tomas, Pausova, Zdenka, Paz-Linares, Deirel, Pichet Binette, Alexa, Pierce, Karen, Qian, Xing, Qiu, Anqi, Raznahan, Armin, Rittman, Timothy, Rodrigue, Amanda, Rollins, Caitlin K., Romero-Garcia, Rafael, Ronan, Lisa, Rosenberg, Monica D., Rowitch, David H., Salum, Giovanni A., Satterthwaite, Theodore D., Schaare, H. Lina, Schabdach, Jenna, Schachar, Russell J., Schöll, Michael, Schultz, Aaron P., Seidlitz, Jakob, Sharp, David, Shinohara, Russell T., Skoog, Ingmar, Smyser, Christopher D., Sperling, Reisa A., Stein, Dan J., Stolicyn, Aleks, Suckling, John, Sullivan, Gemma, Thyreau, Benjamin, Toro, Roberto, Traut, Nicolas, Tsvetanov, Kamen A., Turk-Browne, Nicholas B., Tuulari, Jetro J., Tzourio, Christophe, Vachon-Presseau, Étienne, Valdes-Sosa, Mitchell J., Valdes-Sosa, Pedro A., Valk, Sofie L., van Amelsvoort, Therese, Vandekar, Simon N., Vasung, Lana, Vértes, Petra E., Victoria, Lindsay W., Villeneuve, Sylvia, Villringer, Arno, Vogel, Jacob W., Wagstyl, Konrad, Wang, Yin-Shan S., Warfield, Simon K., Warrier, Varun, Westman, Eric, Westwater, Margaret L., Whalley, Heather C., White, Simon R., Witte, A. Veronica, Yang, Ning, Yeo, B.T. Thomas, Yun, Hyuk Jin, Zalesky, Andrew, Zar, Heather J., Zettergren, Anna, Zhou, Juan H., Ziauddeen, Hisham, Zimmerman, Dabriel, Zugman, Andre, Zuo, Xi-Nian N., Bullmore, Edward T., Alexander-Bloch, Aaron F., Frey, Benicio N., Harkness, Kate L., Addington, Jean, Kennedy, Sidney H., and Dunlop, Katharine
- Abstract
Patients with major depressive disorder (MDD) can present with altered brain structure and deficits in cognitive function similar to aging. Yet, the interaction between age-related brain changes and brain development in MDD remains understudied. In a cohort of adolescents and adults with and without MDD, we assessed brain aging differences and associations through a newly developed tool quantifying normative neurodevelopmental trajectories.
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- 2024
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6. Baseline associations of cardiometabolic risk factors, executive function, and white matter microstructure in the DIRECT‐PLUS trial.
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Kaplan, Alon, Beyer, Frauke, Witte, A. Veronica, and Shai, Iris
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Background: Cardiometabolic risk factors including mid‐life obesity, diabetes and hypertension are associated with accelerated cognitive decline, e.g. in executive function, and brain aging, e.g. alterations in white matter (WM) microstructure [1]. Method: 290 participants (34 females, mean age= 49 y (30 – 79 y) of the DIRECT‐PLUS trial (clinicaltrials.gov ID: NCT03020186, [2]) participated in diffusion‐weighted imaging on a 3T Philips. Tract‐based spatial statistics provided hemisphere‐averaged fractional anisotropy (FA) of 9 fiber tracts (genu/body of corpus callosum, body and crus of fornix, (hippocampal) cingulum, inferior/superior longitudinal fasciculus (i/sLF) and uncinate fasciculus. We tested the association of age, sex, five risk factors (body mass index (BMI), systolic blood pressure (SBP), glycated hemoglobin, high and low‐density lipoprotein), four measures of executive function from the brief executive function battery and average FA of these WM tracts with correlations and linear regression models, adjusting for age and sex, in R version 3.6.1, corrected for multiple testing using false discovery rate (FDR). Result: Age was associated with lower FA in the cingulum (r=‐0.56, p<0.001), uncinate fasciculus (r=‐0.51, p<0.001), hippocampus cingulum (r=‐0.39, p<0.001), iLF (‐0.19, p=0.001) and sLF (r=‐0.18, p=0.001). FA in the fornix, hippocampus cingulum and iLF were higher in men as compared to women (p<0.05 for all). Among cardiometabolic risk factors, higher BMI was associated with significantly lower FA in iILF (β = ‐0.002, p=0.0009, padj=0.035), and higher SBP was associated with lower FA in uncinate fasciculus (β= ‐0.0006, p=0.0012, padj=0.035)None of the executive function measures was significantly related to FA of WM tracts after adjusting for age and sex and controlling for FDR. Conclusion: Our findings corroborate previous findings of the adverse impacts of cardiometabolic risk on WM microstructure. Upcoming longitudinal investigations in this dataset will show whether improved cardiometabolic function, induced by a dietary intervention, will modify the trajectories of WM microstructure and cognitive performance. Figure 1: Higher BMI and higher SBP were significantly associated with lower FA in two major WM tracts (blue: ILF, green: uncinate fasciculus)[1] Wassenaar, TM., et al. NBA (2019) [2] Tsaban, G, et al. Heart (2020). [ABSTRACT FROM AUTHOR]
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- 2021
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7. P2‐156: EFFECTS OF LEPTIN ON HIPPOCAMPUS VOLUME AND COGNITIVE PERFORMANCE.
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Huhn, Sebastian, Beyer, Frauke, Zhang, Rui, Lampe, Leonie, Luck, Tobias, Riedel-Heller, Steffi G., Kratzsch, Jürgen, Schroeter, Matthias L., Loeffler, Markus, Stumvoll, Michael, Villringer, Arno, and Witte, A. Veronica
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- 2018
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8. The Obesity-Susceptibility Gene TMEM18Promotes Adipogenesis through Activation of PPARG
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Landgraf, Kathrin, Klöting, Nora, Gericke, Martin, Maixner, Nitzan, Guiu-Jurado, Esther, Scholz, Markus, Witte, A. Veronica, Beyer, Frauke, Schwartze, Julian T., Lacher, Martin, Villringer, Arno, Kovacs, Peter, Rudich, Assaf, Blüher, Matthias, Kiess, Wieland, and Körner, Antje
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TMEM18is the strongest candidate for childhood obesity identified from GWASs, yet as for most GWAS-derived obesity-susceptibility genes, the functional mechanism remains elusive. We here investigate the relevance of TMEM18for adipose tissue development and obesity. We demonstrate that adipocyte TMEM18expression is downregulated in children with obesity. Functionally, downregulation of TMEM18impairs adipocyte formation in zebrafish and in human preadipocytes, indicating that TMEM18is important for adipocyte differentiation in vivoand in vitro. On the molecular level, TMEM18 activates PPARG, particularly upregulating PPARG1promoter activity, and this activation is repressed by inflammatory stimuli. The relationship between TMEM18and PPARG1is also evident in adipocytes of children and is clinically associated with obesity and adipocyte hypertrophy, inflammation, and insulin resistance. Our findings indicate a role of TMEM18as an upstream regulator of PPARGsignaling driving healthy adipogenesis, which is dysregulated with adipose tissue dysfunction and obesity.
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- 2020
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9. Association of peripheral blood pressure with gray matter volume in 19- to 40-year-old adults.
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Schaare, H. Una, Masouleh, Shahrzad Kharabian, Beyer, Frauke, Schaare, H Lina, Kharabian Masouleh, Shahrzad, Kumral, Deniz, Uhlig, Marie, Reinelt, Janis D, Reiter, Andrea M F, Lampe, Leonie, Babayan, Anahit, Erbey, Miray, Roebbig, Josefin, Schroeter, Matthias L, Okon-Singer, Hadas, Müller, Karsten, Mendes, Natacha, Margulies, Daniel S, Witte, A Veronica, and Gaebler, Michael
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- 2019
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10. ASSOCIATION OF HIPPOCAMPAL VOLUMES WITH COGNITIVE TASKS IN A LARGE POPULATION-BASED COHORT.
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Huhn, Sebastian, Beyer, Frauke, Zhang, Rui, Lampe, Leonie, Luck, Tobias, Riedel-Heller, Steffi G., Schroeter, Mathias L., Loeffler, Markus, Stumvoll, Michael, Villringer, Arno, and Witte, Veronica A.
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- 2017
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11. IMMUNE CELL POPULATIONS ARE ASSOCIATED WITH HUMAN HIPPOCAMPUS VOLUME.
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Witte, A. Veronica, Beyer, Frauke, Huhn, Sebastian, Zhang, Rui, Lampe, Leonie, Luck, Tobias, Riedel-Heller, Steffi G., Stumvoll, Michael, Schroeter, Matthias L., Melzer, Susanne, Tárnok, Attila, and Villringer, Arno
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- 2017
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12. FTO IS NOT RELATED TO IMAGING PARAMETERS OF THE HIPPOCAMPUS: A VOLUMETRIC AND DIFFUSION TENSOR IMAGING STUDY.
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Huhn, Sebastian, Rui, Zhang, Beyer, Frauke, Masouleh, Shahrzad Kharabian, Lampe, Leonie, Luck, Tobias, Riedel-Heller, Steffi G., Schroeter, Mathias L., Scholz, Markus, Burkhardt, Ralph, Stumvoll, Michael, Loeffler, Markus, Villringer, Arno, and Witte, Veronica A.
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- 2016
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