324 results on '"Borén, Jan"'
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2. Adipocyte PI3K links adipostasis with baseline insulin secretion at fasting through an adipoincretin effect
3. Regulation of meiotic telomere dynamics through membrane fluidity promoted by AdipoR2-ELOVL2
4. Role of Perilipins in Oxidative Stress—Implications for Cardiovascular Disease
5. CD38 as a therapeutic target in pediatric Burkitt’s Lymphoma: insights from a comparative approach (Running title: Targeting CD38 in pediatric Burkitt's Lymphoma)
6. Metabolism of Triglyceride-Rich Lipoproteins
7. Contribution of intestinal triglyceride-rich lipoproteins to residual atherosclerotic cardiovascular disease risk in individuals with type 2 diabetes on statin therapy
8. Triglyceride-rich lipoprotein remnants, low-density lipoproteins, and risk of coronary heart disease: a UK Biobank study
9. AdipoR2 recruits protein interactors to promote fatty acid elongation and membrane fluidity
10. Longitudinal metabolomics analysis reveals the acute effect of cysteine and NAC included in the combined metabolic activators
11. Exploration of Novel Urolithin C Derivatives as Non-Competitive Inhibitors of Liver Pyruvate Kinase
12. Cerebrospinal fluid sulfatide isoforms lack diagnostic utility in separating progressive from relapsing-remitting multiple sclerosis
13. Data from Treatment with HIV-Protease Inhibitor Nelfinavir Identifies Membrane Lipid Composition and Fluidity as a Therapeutic Target in Advanced Multiple Myeloma
14. Supplementary Data from Treatment with HIV-Protease Inhibitor Nelfinavir Identifies Membrane Lipid Composition and Fluidity as a Therapeutic Target in Advanced Multiple Myeloma
15. Supplementary Movie S1C from Treatment with HIV-Protease Inhibitor Nelfinavir Identifies Membrane Lipid Composition and Fluidity as a Therapeutic Target in Advanced Multiple Myeloma
16. Table S4 from Treatment with HIV-Protease Inhibitor Nelfinavir Identifies Membrane Lipid Composition and Fluidity as a Therapeutic Target in Advanced Multiple Myeloma
17. Data from Treatment with HIV-Protease Inhibitor Nelfinavir Identifies Membrane Lipid Composition and Fluidity as a Therapeutic Target in Advanced Multiple Myeloma
18. Supplementary Data from Treatment with HIV-Protease Inhibitor Nelfinavir Identifies Membrane Lipid Composition and Fluidity as a Therapeutic Target in Advanced Multiple Myeloma
19. Supplementary Movie S1A from Treatment with HIV-Protease Inhibitor Nelfinavir Identifies Membrane Lipid Composition and Fluidity as a Therapeutic Target in Advanced Multiple Myeloma
20. Supplementary Movie S1B from Treatment with HIV-Protease Inhibitor Nelfinavir Identifies Membrane Lipid Composition and Fluidity as a Therapeutic Target in Advanced Multiple Myeloma
21. Cardiomyocyte-specific PCSK9 deficiency compromises mitochondrial bioenergetics and heart function
22. Tuning liver pyruvate kinase activity up or down with a new class of allosteric modulators
23. Replacing Foods with a High-Glycemic Index and High in Saturated Fat by Alternatives with a Low Glycemic Index and Low Saturated Fat Reduces Hepatic Fat, Even in Isocaloric and Macronutrient Matched Conditions
24. Combined metabolic activators improve metabolic functions in the animal models of neurodegenerative diseases
25. Cardiac Plin5 interacts with SERCA2 and promotes calcium handling and cardiomyocyte contractility
26. Combined metabolic activators improve cognitive functions in Alzheimer’s disease patients: a randomised, double-blinded, placebo-controlled phase-II trial
27. Cardiac arrhythmias and conduction abnormalities in patients with type 2 diabetes
28. Quantitative interactome proteomics reveals Plin5 as a novel partner of sarcoplasmic/endoplasmic reticulum Ca2+ATPase 2a (SERCA2a) in the regulation of calcium cycling
29. Sphingosine 1-phosphate mediates adiponectin receptor signaling essential for lipid homeostasis and embryogenesis
30. Postprandial metabolism of apolipoproteins B48, B100, C-III, and E in humans with APOC3 loss-of-function mutations
31. Cardiac Arrhythmias and Conduction Abnormalities in Patients with Type 2 Diabetes
32. Left-Sided Degenerative Valvular Heart Disease in Type 1 and Type 2 Diabetes
33. pH-Dependent Protonation of Histidine Residues Is Critical for Electrostatic Binding of Low-Density Lipoproteins to Human Coronary Arteries
34. The spoils of war and the long-term spoiling of health conditions of entire nations
35. Pathogenesis of atherosclerosis: lipid metabolism
36. Role of endogenous incretins in the regulation of postprandial lipoprotein metabolism
37. Modified lipid metabolism and cytosolic phospholipase A2 activation in mesangial cells under pro-inflammatory conditions
38. MO614: Modified Lipid Metabolism and Cytosolic Phospholipase A2 Activation in Mesangial Cells Under Pro-Inflammatory Conditions
39. Anthraquinone derivatives as ADP-competitive inhibitors of liver pyruvate kinase
40. Metabolism of triglyceride-rich lipoproteins in health and dyslipidaemia
41. Multiomics Analysis Reveals the Impact of Microbiota on Host Metabolism in Hepatic Steatosis
42. Palmitic acid causes increased dihydroceramide levels when desaturase expression is directly silenced or indirectly lowered by silencing AdipoR2
43. The role of circulating galectin-1 in type 2 diabetes and chronic kidney disease: evidence from cross-sectional, longitudinal and Mendelian randomisation analyses
44. A network-based approach reveals the dysregulated transcriptional regulation in non-alcoholic fatty liver disease
45. Intussusceptive Angiogenesis in Human Metastatic Malignant Melanoma
46. Combined Metabolic Activators Decrease Liver Steatosis by Activating Mitochondrial Metabolism in Hamsters Fed with a High-Fat Diet
47. Testosterone reduces metabolic brown fat activity in male mice
48. Combined metabolic activators therapy ameliorates liver fat in nonalcoholic fatty liver disease patients
49. Effects of PNPLA3 I148M on hepatic lipid and very‐low‐density lipoprotein metabolism in humans
50. Triglyceride-rich lipoproteins and their remnants: metabolic insights, role in atherosclerotic cardiovascular disease, and emerging therapeutic strategies—a consensus statement from the European Atherosclerosis Society
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