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3. Contributors

6. An enhanced IL17 and muted type I interferon nasal epithelial cell state characterizes severe COVID-19 with fungal coinfection

8. The Global Hidradenitis Suppurativa Atlas (GHiSA) Methodology: Combining Global Proportions in a Pooled Analysis

11. Tertiary lymphoid structures sustain cutaneous B cell activity in hidradenitis suppurativa

16. Global consensus process to establish a core dataset for hidradenitis suppurativa registries

21. Outcome Measures for the Evaluation of Treatment Response in Hidradenitis Suppurativa for Clinical Practice

22. Enhanced production of eicosanoids in plasma and activation of DNA damage pathways in PBMCs are correlated with the severity of ancestral COVID-19 infection

29. Transforming Safety and Quality in Nursing

30. Factors associated with disease-specific life impact in patients with hidradenitis suppurativa: results from the Global VOICE project

31. Tertiary Lymphoid Structures Sustain Cutaneous B cell Activity in Hidradenitis Suppurativa

32. Enhanced Production of Eicosanoids in Plasma and Activation of DNA Damage Pathways in Pbmcs are Correlated with the Severity of Ancestral COVID-19 Infection

35. Real-World Findings on the Characteristics and Treatment Exposures of Patients with Hidradenitis Suppurativa from US Claims Data

36. Biologic Therapy Is Not Associated with Increased COVID-19 Severity in Patients with Hidradenitis Suppurativa: Updated Findings from the Global Hidradenitis Suppurativa COVID-19 Registry

37. Adaptation of Remote Symptom Monitoring Using Electronic Patient-Reported Outcomes for Implementation in Real-World Settings

39. Anakinra for refractory pustular psoriasis: A phase II, open-label, dose-escalation trial

40. Diquinol Functionality Boosts the Superoxide Dismutase Mimicry of a Zn(II) Complex with a Redox-Active Ligand while Maintaining Catalyst Stability and Enhanced Activity in Phosphate Solution

41. Hidradenitis suppurativa and sleep: a systematic review

43. Severe COVID-19 is associated with fungal colonization of the nasopharynx and potent induction of IL-17 responses in the nasal epithelium

45. Immunopathogenesis of hidradenitis suppurativa and response to anti–TNF-α therapy

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