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6. A single-nucleus and spatial transcriptomic atlas of the COVID-19 liver reveals topological, functional, and regenerative organ disruption in patients

11. GCH1 Deficiency Activates Brain Innate Immune Response and Impairs Tyrosine Hydroxylase Homeostasis

12. COVID-19 tissue atlases reveal SARS-CoV-2 pathology and cellular targets

14. A single-cell and spatial atlas of autopsy tissues reveals pathology and cellular targets of SARS-CoV-2

16. Whole-genome sequencing reveals new Alzheimer’s disease-associated rare variants in loci related to synaptic function and neuronal development

18. Aβ-accelerated neurodegeneration caused by Alzheimer’s-associated ACE variant R1279Q is rescued by angiotensin system inhibition in mice

19. Meta-Analysis of the Alzheimer’s Disease Human Brain Transcriptome and Functional Dissection in Mouse Models

20. Deep phenotyping of peripheral tissue facilitates mechanistic disease stratification in sporadic Parkinson’s disease

21. Identification of Novel Alzheimer’s Disease Loci Using Sex-Specific Family-Based Association Analysis of Whole-Genome Sequence Data

26. In Vivo Profiling of Leukemic Stem Cell Fitness Identifies Therapeutically Actionable Determinants of Growth

31. Molecular, phenotypic, and sample-associated data to describe pluripotent stem cell lines and derivatives

32. A data-driven approach links microglia to pathology and prognosis in amyotrophic lateral sclerosis

36. Integrated Genomic Analysis of Diverse Induced Pluripotent Stem Cells from the Progenitor Cell Biology Consortium

37. P4-031: Integrative Network Analysis of Multiple Alzheimer's Disease Rnaseq Studies From the Accelerating Medicine Partnership-Alzheimer's Disease Consortium

38. Exome sequencing reveals recurrent germ line variants in patients with familial Waldenström macroglobulinemia

40. Mycoplasma Infection Alters Cancer Stem Cell Properties in Vitro

47. Clonal-Heterogeneity and Propensity for Bone Metastasis in Multiple Myeloma

48. Identification of a Gene Expression Signature Characterizing Clonal Fitness and Dominance in Vivo in a Murine Model of MLL-AF9 Leukemia

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