38 results on '"Keck, Mathilde"'
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2. A New Derivative of Retro-2 Displays Antiviral Activity against Respiratory Syncytial Virus
3. A New Derivative of Retro-2 Displays Antiviral Activity against Respiratory Syncytial Virus
4. One-year post-exposure assessment of 14C-few-layer graphene biodistribution in mice: single versus repeated intratracheal administration
5. From a Cone Snail Toxin to a Competitive MC4R Antagonist
6. C910 chemical compound inhibits the traffiking of several bacterial AB toxins with cross-protection against influenza virus
7. QGC606: A Best-in-Class Orally Active Centrally Acting Aminopeptidase A Inhibitor Prodrug for Treating Heart Failure Following Myocardial Infarction
8. Monitoring In Vivo Performances of Protein–Drug Conjugates Using Site-Selective Dual Radiolabeling and Ex Vivo Digital Imaging
9. A new Kunitz‐type snake toxin family associated with an original mode of interaction with the vasopressin 2 receptor
10. First evidence that emerging pinnatoxin-G, a contaminant of shellfish, reaches the brain and crosses the placental barrier
11. A screening pipeline identifies a broad-spectrum inhibitor of bacterial AB toxins with cross protection against influenza A virus H1N1 and SARS-CoV-2
12. LIT01-196, a Metabolically Stable Apelin-17 Analog, Normalizes Blood Pressure in Hypertensive DOCA-Salt Rats via a NO Synthase-dependent Mechanism
13. A new Kunitz-type snake toxin family associated with an original mode of interaction with the vasopressin 2 receptor.
14. A variant of ASIC2 mediates sodium retention in nephrotic syndrome
15. BRAIN-PENETRATING AMINOPEPTIDASE A INHIBITORS FOR NEW TREATMENT OF HYPERTENSION AND HEART FAILURE
16. A new variant of ASIC2 mediates sodium retention in nephrotic syndrome
17. A metabolically stable apelin-17 analog decreases AVP-induced antidiuresis and improves hyponatremia
18. Analysis of CLCNKB mutations at dimer‐interface, calcium‐binding site, and pore reveals a variety of functional alterations in ClC‐Kb channel leading to Bartter syndrome
19. NI956/QGC006, a Potent Orally Active, Brain-Penetrating Aminopeptidase A Inhibitor for Treating Hypertension
20. Orally Active Aminopeptidase A Inhibitor Prodrugs: Current State and Future Directions
21. Cardiac inflammatory CD11b/c cells exert a protective role in hypertrophied cardiomyocyte by promoting TNFR2- and Orai3- dependent signaling
22. Brain renin-angiotensin system blockade with orally active aminopeptidase A inhibitor prevents cardiac dysfunction after myocardial infarction in mice
23. Clinical and Genetic Spectrum of Bartter Syndrome Type 3
24. The ClC-K2 Chloride Channel Is Critical for Salt Handling in the Distal Nephron
25. ClC-K chloride channels: emerging pathophysiology of Bartter syndrome type 3
26. TNFR2‐mediated Survival via Orai1‐3‐dependent Calcium Influx in Compensated Cardiac Hypertrophy
27. Emergence of Orai3 activity during cardiac hypertrophy
28. CLCNKB mutations causing mild Bartter syndrome profoundly alter the pH and Ca2+ dependence of ClC-Kb channels
29. Characterization of the mouse ClC-K1/Barttin chloride channel
30. Mutations in SLC2A2 Gene Reveal hGLUT2 Function in Pancreatic β Cell Development
31. Abstract 198: Stim/orai Channel Complex In Normal And Hypertrophied Adult Cardiac Myocytes
32. Abstract 313: STIM1 Silencing Reverses Pressure-overload Induced Cardiac Hypertrophy In Mice
33. Abstract 312: STIM1 And The Development Of Pressure-overload Induced Cardiac Hypertrophy In Rodents
34. NovelCLCNKBMutations Causing Bartter Syndrome Affect Channel Surface Expression
35. Genotype‐phenotype relationship in patients affected by novel CLCNKB mutations
36. Renal phenotype in mice lacking the Kir5.1 ( Kcnj16 ) K + channel subunit contrasts with that observed in SeSAME/EAST syndrome
37. Functional analysis of novel CLCNKB mutations in patients with Bartter syndrome
38. Heterogeneity in the processing of CLCN5 mutants related to Dent disease
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