21 results on '"Klotz-Noack, Kathleen"'
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2. Supplementary Figure 3 from PP2A-B55β Antagonizes Cyclin E1 Proteolysis and Promotes Its Dysregulation in Cancer
3. Data from PP2A-B55β Antagonizes Cyclin E1 Proteolysis and Promotes Its Dysregulation in Cancer
4. Supplementary Figure 6 from PP2A-B55β Antagonizes Cyclin E1 Proteolysis and Promotes Its Dysregulation in Cancer
5. Supplementary Materials and Methods, Figure Legends from PP2A-B55β Antagonizes Cyclin E1 Proteolysis and Promotes Its Dysregulation in Cancer
6. Supplementary Table 1 from PP2A-B55β Antagonizes Cyclin E1 Proteolysis and Promotes Its Dysregulation in Cancer
7. Data from PP2A-B55β Antagonizes Cyclin E1 Proteolysis and Promotes Its Dysregulation in Cancer
8. Supplementary Figure 2 from PP2A-B55β Antagonizes Cyclin E1 Proteolysis and Promotes Its Dysregulation in Cancer
9. Supplementary Figure 1 from PP2A-B55β Antagonizes Cyclin E1 Proteolysis and Promotes Its Dysregulation in Cancer
10. Supplementary Figure 4 from PP2A-B55β Antagonizes Cyclin E1 Proteolysis and Promotes Its Dysregulation in Cancer
11. Mitogen‐activated protein kinase activity drives cell trajectories in colorectal cancer
12. Mutation-specific effects of NRAS oncogenes in colorectal cancer cells
13. SFPQ Depletion Is Synthetically Lethal with BRAFV600E in Colorectal Cancer Cells
14. Reduced replication origin licensing selectively kills KRAS-mutant colorectal cancer cells via mitotic catastrophe
15. Low-Dose Vertical Inhibition of the RAF-MEK-ERK Cascade Causes Apoptotic Death of KRAS Mutant Cancers
16. Mitogen-activated protein kinase activity drives cell trajectories in colorectal cancer
17. Abstract A20: Depletion of replication factor MCM7 is synthetically lethal to oncogenic KRAS expression
18. Abstract 5170: HDAC inhibitors and the mechanism of resistance in colorectal cancer: RAS and MYC - the partners in crime
19. PP2A-B55β Antagonizes Cyclin E1 Proteolysis and Promotes Its Dysregulation in Cancer
20. Re-replication induced by geminin depletion occurs from G2 and is enhanced by checkpoint activation
21. A Role for Dormant Origins in Tumor Suppression
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