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6. Suppl.Mat., Suppl. Table, Suppl. Figs Legends from Phenformin-Induced Mitochondrial Dysfunction Sensitizes Hepatocellular Carcinoma for Dual Inhibition of mTOR

9. Suppl.Mat., Suppl. Table, Suppl. Figs Legends from Phenformin-Induced Mitochondrial Dysfunction Sensitizes Hepatocellular Carcinoma for Dual Inhibition of mTOR

15. A compound directed against S6K1 hampers fat mass expansion and mitigates diet-induced hepatosteatosis

16. The PI3K-mTOR Pathway

17. Impaired ribosome biogenesis checkpoint activation induces p53-dependent MCL-1 degradation and MYC-driven lymphoma death

19. Nucleotide depletion reveals the impaired ribosome biogenesis checkpoint as a barrier against DNA damage

20. S6K

24. Autogenous Control of 5′TOP mRNA Stability by 40S Ribosomes

25. A Phase Ib Study of the Dual PI3K/mTOR Inhibitor Dactolisib (BEZ235) Combined with Everolimus in Patients with Advanced Solid Malignancies

26. EPRS is a critical mTORC1–S6K1 effector that influences adiposity in mice

27. S6K1 Phosphorylation of H2B Mediates EZH2 Trimethylation of H3: A Determinant of Early Adipogenesis

28. S6K-STING interaction regulates cytosolic DNA–mediated activation of the transcription factor IRF3

33. S6K1 controls pancreatic β cell size independently of intrauterine growth restriction

36. Mechanistic Target of Rapamycin (Mtor) Is Essential for Murine Embryonic Heart Development and Growth

38. mTOR Inhibitors Synergize on Regression, Reversal of Gene Expression, and Autophagy in Hepatocellular Carcinoma

39. Metabolic Control by S6 Kinases Depends on Dietary Lipids

40. A guide to analysis of mouse energy metabolism

44. mTORC1-Mediated Cell Proliferation, But Not Cell Growth, Controlled by the 4E-BPs

45. The Nuclear Receptor DHR3 Modulates dS6 Kinase–Dependent Growth in Drosophila

46. Metformin, Independent of AMPK, Inhibits mTORC1 in a Rag GTPase-Dependent Manner

47. S6K1 Plays a Critical Role in Early Adipocyte Differentiation

48. Abstract 5349: Vesicular stomatitis virus oncolysis is potentiated by impairing mTORC1-dependent type-I IFN production

49. A novel type of cellular senescence that can be enhanced in mouse models and human tumor xenografts to suppress prostate tumorigenesis

50. Vesicular stomatitis virus oncolysis is potentiated by impairing mTORC1-dependent type I IFN production

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