171 results on '"Paramio, Jesús M."'
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2. Contributors
3. Targeting histone modifications in cancer immunotherapy
4. Competitive Repopulation Assay of Long-Term Epidermal Stem Cell Regeneration Potential
5. Figure S1 from IKKβ-Mediated Resistance to Skin Cancer Development Is Ink4a/Arf-Dependent
6. Supplementary Table 1 from IKKβ-Mediated Resistance to Skin Cancer Development Is Ink4a/Arf-Dependent
7. Figure S4 from IKKβ-Mediated Resistance to Skin Cancer Development Is Ink4a/Arf-Dependent
8. Supplementary Information from IKKβ-Mediated Resistance to Skin Cancer Development Is Ink4a/Arf-Dependent
9. Data from IKKβ-Mediated Resistance to Skin Cancer Development Is Ink4a/Arf-Dependent
10. Figure S3 from IKKβ-Mediated Resistance to Skin Cancer Development Is Ink4a/Arf-Dependent
11. Supplementary Information from IKKβ-Mediated Resistance to Skin Cancer Development Is Ink4a/Arf-Dependent
12. Figure S5 from IKKβ-Mediated Resistance to Skin Cancer Development Is Ink4a/Arf-Dependent
13. Supplementary Table 1 from IKKβ-Mediated Resistance to Skin Cancer Development Is Ink4a/Arf-Dependent
14. Figure S2 from IKKβ-Mediated Resistance to Skin Cancer Development Is Ink4a/Arf-Dependent
15. Figure S2 from IKKβ-Mediated Resistance to Skin Cancer Development Is Ink4a/Arf-Dependent
16. Figure S5 from IKKβ-Mediated Resistance to Skin Cancer Development Is Ink4a/Arf-Dependent
17. Figure S4 from IKKβ-Mediated Resistance to Skin Cancer Development Is Ink4a/Arf-Dependent
18. Figure S3 from IKKβ-Mediated Resistance to Skin Cancer Development Is Ink4a/Arf-Dependent
19. Data from IKKβ-Mediated Resistance to Skin Cancer Development Is Ink4a/Arf-Dependent
20. Figure S1 from IKKβ-Mediated Resistance to Skin Cancer Development Is Ink4a/Arf-Dependent
21. Supplementary Methods and References from A Transposon-based Analysis Reveals RASA1 Is Involved in Triple-Negative Breast Cancer
22. Supplementary Tables S1-S7 from A Transposon-based Analysis Reveals RASA1 Is Involved in Triple-Negative Breast Cancer
23. Supplementary Data from BMP4 Induces M2 Macrophage Polarization and Favors Tumor Progression in Bladder Cancer
24. Data from A Transposon-based Analysis Reveals RASA1 Is Involved in Triple-Negative Breast Cancer
25. Supplementary Methods and References from A Transposon-based Analysis Reveals RASA1 Is Involved in Triple-Negative Breast Cancer
26. Data from A Transposon-based Analysis Reveals RASA1 Is Involved in Triple-Negative Breast Cancer
27. Supplementary Data from BMP4 Induces M2 Macrophage Polarization and Favors Tumor Progression in Bladder Cancer
28. Supplementary Tables S1-S7 from A Transposon-based Analysis Reveals RASA1 Is Involved in Triple-Negative Breast Cancer
29. Supp Table S3 from In Vivo Disruption of an Rb–E2F–Ezh2 Signaling Loop Causes Bladder Cancer
30. Supplementary Figures 1-4 from Spontaneous Squamous Cell Carcinoma Induced by the Somatic Inactivation of Retinoblastoma and Trp53 Tumor Suppressors
31. Supplementary Legends 1-2 from Deregulated Activity of Akt in Epithelial Basal Cells Induces Spontaneous Tumors and Heightened Sensitivity to Skin Carcinogenesis
32. Supp Table S2 from In Vivo Disruption of an Rb–E2F–Ezh2 Signaling Loop Causes Bladder Cancer
33. Supp Table S17 from In Vivo Disruption of an Rb–E2F–Ezh2 Signaling Loop Causes Bladder Cancer
34. Supp Table S16 from In Vivo Disruption of an Rb–E2F–Ezh2 Signaling Loop Causes Bladder Cancer
35. Data from Akt Activation Synergizes with Trp53 Loss in Oral Epithelium to Produce a Novel Mouse Model for Head and Neck Squamous Cell Carcinoma
36. Supp Table S5 from In Vivo Disruption of an Rb–E2F–Ezh2 Signaling Loop Causes Bladder Cancer
37. Supp Table S11 from In Vivo Disruption of an Rb–E2F–Ezh2 Signaling Loop Causes Bladder Cancer
38. Data from Akt Activation Synergizes with Trp53 Loss in Oral Epithelium to Produce a Novel Mouse Model for Head and Neck Squamous Cell Carcinoma
39. Supp Table S15 from In Vivo Disruption of an Rb–E2F–Ezh2 Signaling Loop Causes Bladder Cancer
40. Supp Table S15 from In Vivo Disruption of an Rb–E2F–Ezh2 Signaling Loop Causes Bladder Cancer
41. Data from Spontaneous Squamous Cell Carcinoma Induced by the Somatic Inactivation of Retinoblastoma and Trp53 Tumor Suppressors
42. Supp Table S5 from In Vivo Disruption of an Rb–E2F–Ezh2 Signaling Loop Causes Bladder Cancer
43. Supplementary Figure 1 from Deregulated Activity of Akt in Epithelial Basal Cells Induces Spontaneous Tumors and Heightened Sensitivity to Skin Carcinogenesis
44. Supplementary Figures 1-10 from Akt Activation Synergizes with Trp53 Loss in Oral Epithelium to Produce a Novel Mouse Model for Head and Neck Squamous Cell Carcinoma
45. Supp Table S10 from In Vivo Disruption of an Rb–E2F–Ezh2 Signaling Loop Causes Bladder Cancer
46. Supplementary Information from In Vivo Disruption of an Rb–E2F–Ezh2 Signaling Loop Causes Bladder Cancer
47. Data from In Vivo Disruption of an Rb–E2F–Ezh2 Signaling Loop Causes Bladder Cancer
48. Supplementary Legends 1-2 from Deregulated Activity of Akt in Epithelial Basal Cells Induces Spontaneous Tumors and Heightened Sensitivity to Skin Carcinogenesis
49. Supplementary Data - Gene List from Spontaneous Squamous Cell Carcinoma Induced by the Somatic Inactivation of Retinoblastoma and Trp53 Tumor Suppressors
50. Supplementary Figure 2 from Deregulated Activity of Akt in Epithelial Basal Cells Induces Spontaneous Tumors and Heightened Sensitivity to Skin Carcinogenesis
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