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78 results on '"Perez-Mancera, Pedro A."'

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1. Figure S7 from Oncogenic KRAS Induces NIX-Mediated Mitophagy to Promote Pancreatic Cancer

2. Figure S6 from Oncogenic KRAS Induces NIX-Mediated Mitophagy to Promote Pancreatic Cancer

3. Supplementary Table S12 from Tissue-Specific Oncogenic Activity of KRASA146T

4. Supplementary Tables S14-S17 from Tissue-Specific Oncogenic Activity of KRASA146T

5. Data from Oncogenic KRAS Induces NIX-Mediated Mitophagy to Promote Pancreatic Cancer

6. Figure S1 from Oncogenic KRAS Induces NIX-Mediated Mitophagy to Promote Pancreatic Cancer

7. Figure S7 from Oncogenic KRAS Induces NIX-Mediated Mitophagy to Promote Pancreatic Cancer

8. Data from Oncogenic KRAS Induces NIX-Mediated Mitophagy to Promote Pancreatic Cancer

9. Data from Tissue-Specific Oncogenic Activity of KRASA146T

10. Supplementary Tables S14-S17 from Tissue-Specific Oncogenic Activity of KRASA146T

11. Figure S8 from Oncogenic KRAS Induces NIX-Mediated Mitophagy to Promote Pancreatic Cancer

12. Supplementary Table S13 from Tissue-Specific Oncogenic Activity of KRASA146T

13. Figure S4 from Oncogenic KRAS Induces NIX-Mediated Mitophagy to Promote Pancreatic Cancer

14. Data from Tissue-Specific Oncogenic Activity of KRASA146T

15. Supplementary Tables S2-S10 from Tissue-Specific Oncogenic Activity of KRASA146T

16. Supplementary Table S11 from Tissue-Specific Oncogenic Activity of KRASA146T

17. Figure S5 from Oncogenic KRAS Induces NIX-Mediated Mitophagy to Promote Pancreatic Cancer

18. Supplementary Table S1 from Tissue-Specific Oncogenic Activity of KRASA146T

19. Supplementary Table S12 from Tissue-Specific Oncogenic Activity of KRASA146T

20. Figure S2 from Oncogenic KRAS Induces NIX-Mediated Mitophagy to Promote Pancreatic Cancer

21. Figure S3 from Oncogenic KRAS Induces NIX-Mediated Mitophagy to Promote Pancreatic Cancer

22. Figure S2 from Oncogenic KRAS Induces NIX-Mediated Mitophagy to Promote Pancreatic Cancer

23. Figure S4 from Oncogenic KRAS Induces NIX-Mediated Mitophagy to Promote Pancreatic Cancer

24. Supplementary Figures S1-S18 from Tissue-Specific Oncogenic Activity of KRASA146T

25. Supplementary Tables S2-S10 from Tissue-Specific Oncogenic Activity of KRASA146T

26. Figure S6 from Oncogenic KRAS Induces NIX-Mediated Mitophagy to Promote Pancreatic Cancer

27. Supplementary Table S11 from Tissue-Specific Oncogenic Activity of KRASA146T

28. Supplementary Table S1 from Tissue-Specific Oncogenic Activity of KRASA146T

29. Figure S1 from Oncogenic KRAS Induces NIX-Mediated Mitophagy to Promote Pancreatic Cancer

30. Figure S5 from Oncogenic KRAS Induces NIX-Mediated Mitophagy to Promote Pancreatic Cancer

31. Supplementary Figures S1-S18 from Tissue-Specific Oncogenic Activity of KRASA146T

32. Figure S8 from Oncogenic KRAS Induces NIX-Mediated Mitophagy to Promote Pancreatic Cancer

33. Supplementary Table S13 from Tissue-Specific Oncogenic Activity of KRASA146T

34. Supplementary Table 3 from Fibroblasts from Distinct Pancreatic Pathologies Exhibit Disease-Specific Properties

35. Supplementary Table 1 from Fibroblasts from Distinct Pancreatic Pathologies Exhibit Disease-Specific Properties

36. Supplementary Table 4 from Fibroblasts from Distinct Pancreatic Pathologies Exhibit Disease-Specific Properties

37. Data from Chemoresistance in Pancreatic Cancer Is Driven by Stroma-Derived Insulin-Like Growth Factors

38. Supplemental Methods from Fibroblasts from Distinct Pancreatic Pathologies Exhibit Disease-Specific Properties

39. Supplementary Figure Legends, Supplementary Tables 1 and 2 and Supplementary Materials and Methods from Chemoresistance in Pancreatic Cancer Is Driven by Stroma-Derived Insulin-Like Growth Factors

40. Supplementary Table 3 from Fibroblasts from Distinct Pancreatic Pathologies Exhibit Disease-Specific Properties

41. Supplementary Figures 1 through 9 from Chemoresistance in Pancreatic Cancer Is Driven by Stroma-Derived Insulin-Like Growth Factors

42. Data from Fibroblasts from Distinct Pancreatic Pathologies Exhibit Disease-Specific Properties

43. Data from Chemoresistance in Pancreatic Cancer Is Driven by Stroma-Derived Insulin-Like Growth Factors

44. Supplementary Table 2 from Fibroblasts from Distinct Pancreatic Pathologies Exhibit Disease-Specific Properties

45. Supplementary Figure 1 from Fibroblasts from Distinct Pancreatic Pathologies Exhibit Disease-Specific Properties

46. Supplementary Figure Legends, Supplementary Tables 1 and 2 and Supplementary Materials and Methods from Chemoresistance in Pancreatic Cancer Is Driven by Stroma-Derived Insulin-Like Growth Factors

47. Supplementary Figure 2 from Fibroblasts from Distinct Pancreatic Pathologies Exhibit Disease-Specific Properties

48. Supplemental Methods from Fibroblasts from Distinct Pancreatic Pathologies Exhibit Disease-Specific Properties

49. Supplementary Table 1 from Fibroblasts from Distinct Pancreatic Pathologies Exhibit Disease-Specific Properties

50. Supplementary Figure 1 from Fibroblasts from Distinct Pancreatic Pathologies Exhibit Disease-Specific Properties

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