17 results on '"Sahlström, Peter"'
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2. The human bone marrow plasma cell compartment in rheumatoid arthritis - Clonal relationships and anti-citrulline autoantibody producing cells
3. Molecular signature of methotrexate response among rheumatoid arthritis patients
4. Integrated single cell and spatial transcriptomics reveal autoreactive differentiated B cells in joints of early rheumatoid arthritis
5. Integrated Single Cell and Spatial Transcriptomics Reveal Autoreactive Differentiated B Cells in Joints of Early Rheumatoid Arthritis
6. Early prediction of clinical response to anti-TNF treatment using multi-omics and machine learning in rheumatoid arthritis
7. Early Prediction of Clinical Response to Anti-TNF Treatment using Multi-omics and Machine Learning in Rheumatoid Arthritis
8. Different Hierarchies of Anti–Modified Protein Autoantibody Reactivities in Rheumatoid Arthritis
9. Molecular Biomarkers of Anti-TNF Response in Patients with Rheumatoid Arthritis
10. SAT0675B ANTI-MODIFIED PROTEIN AUTOANTIBODIES IN RA DISPLAY IMPORTANT PEPTIDE CROSS-REACTIVITY BUT YET PROTEIN RECOGNITION SELECTIVITY
11. Recognition of Amino Acid Motifs, Rather Than Specific Proteins, by Human Plasma Cell–Derived Monoclonal Antibodies to Posttranslationally Modified Proteins in Rheumatoid Arthritis
12. Differential ACPA Binding to Nuclear Antigens Reveals a PAD-Independent Pathway and a Distinct Subset of Acetylation Cross-Reactive Autoantibodies in Rheumatoid Arthritis
13. A Refined Protocol for Identifying Citrulline-specific Monoclonal Antibodies from Single Human B Cells from Rheumatoid Arthritis Patient Material
14. Pathogenic Citrulline‐Multispecific B Cell Receptor Clades in Rheumatoid Arthritis
15. 08.24 Monoclonal acpa antibodies recognising a common citrulline motif are mainly dependent on light chain hypermutations for antigen recognition
16. Contact-Dependent Interference with Invariant NKT Cell Activation by Herpes Simplex Virus-Infected Cells
17. NKG2D performs two functions in invariant NKT cells: Direct TCR-independent activation of NK-like cytolysis and co-stimulation of activation by CD1d
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