35 results on '"Sancho-Bru, P"'
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2. Ductular reaction cells promote angiogenesis via SLIT2/ROBO1 pathway in chronic liver disease
3. Chronic liver damage causes a reduction in the number and a dysfunction of the activity of liver resident macrophages
4. Generation of a unique liver progenitor cell gene signature to identify LPCs and LPC activation in chronic liver diseases
5. Ductular reaction cells display an inflammatory profile and recruit neutrophils in alcoholic hepatitis
6. The expression of microRNA-155 in inflammatory cells determine liver injury
7. Whole transcriptome analysis of ductular reaction from patients with alcoholic hepatitis. Similarities to ductular reaction in DDC mouse model
8. CD5L modulates primary human hepatic stellate cells activation
9. Adipocyte fatty acid binding protein in cirrhosis: increased hepatic gene expression and plasma levels and correlation with clinical outcomes
10. Neutrophil gelatinase-associated lipocalin is a biomarker of acute-on-chronic liver failure and prognosis in cirrhosis
11. Directed Differentiation of IPS Cells to Hepatic Stellate Cells
12. In Silico Characterization of Liver Progenitor Cells
13. PTX3 is a Novel Marker of Disease Progression in Alcoholic Hepatitis and Attenuates LPS-Induced Liver Injury and Inflammation
14. Lipocalin-2 Gene and its Protein Neutrophil Gelatinase-Associated Lipocalin are Biomarkers of Acute-On-Chronic Liver Failure
15. Growth Differentiation Factor 15 (GDF-15) A Surrogate Marker of Endothelial Dysfunction Correlates with Severity and Predicts Short-Term Survival of Patients with Alcoholic Hepatitis
16. P0435 : Identification of miR-192 as a novel key regulator of quiescence maintenance in human hepatic stellate cells
17. P0436 : Pentraxin-3 induces hepatic stellate cell activation and attenuates the inflammatory response during hepatic injury
18. P0088 : Fermented milk containing Lactobacillus paracasei subsp. paracasei CNCM I -1518 reduces bacterial translocation in rats treated with CCL4
19. P605 GENE EXPRESSION AND DNA METHYLATION PROFILING OF DISTINCT ADULT HUMAN LIVER CELLS AND CULTURE-ACTIVATED HEPATIC STELLATE CELLS
20. O140 INTEGRATIVE MICRORNA PROFILING IDENTIFIES MIR-21 AND MIR-155 AS POTENTIAL REGULATORS IN ALCOHOLIC HEPATITIS
21. P112 LIVER DAMAGE DETERMINES THE GENE EXPRESSION PROFILE OF LIVER PROGENITOR CELLS AND THEIR CONTRIBUTION TO HEPATIC REGENERATION
22. P628 INTEGRATIVE MICRORNA AND GENOMIC ANALYSIS IDENTIFIES miR-21, miR-100 AND miR-192 AS NEW REGULATORY ELEMENTS OF HEPATIC STELLATE CELL ACTIVATION
23. P601 CCR6 DEFICIENCY ENHANCES LIVER INFLAMMATION AND FIBROSIS BY MODULATING HEPATIC MACROPHAGES M1/M2 BALANCE AND INFLAMMATORY CELLS RECRUITMENT
24. P343 IDENTIFICATION OF A NOVEL HEXOKINASE IN PATIENTS WITH ALCOHOLIC HEPATITIS
25. P344 TRANSLATIONAL STUDY IDENTIFIES NEUTROPHIL GELATINASE-ASSOCIATED LIPOCALIN (NGAL) AS A POTENTIAL MEDIATOR OF LIVER INJURY AND FIBROGENESIS IN ALCOHOLIC HEPATITIS
26. 37 THE BILIARY COMPARTMENT IS THE ORIGIN OF LIVER PROGENITOR CELLS AND CONTRIBUTES TO NEWLY GENERATED HEPATOCYTES IN HEPATIC REGENERATION
27. 513 CCL20 AS POTENTIAL MEDIATOR OF INFLAMMATION AND FIBROSIS IN PATIENTS WITH ALCOHOLIC HEPATITIS: EVIDENCES OF A TRANSLATIONAL STUDY
28. 1126 IDENTIFICATION OF p90RSK AS A NOVEL THERAPEUTIC TARGET FOR LIVER FIBROSIS
29. 19 LIVER PROGENITOR CELL MARKERS CORRELATE WITH LIVER DAMAGE AND PREDICT SHORT-TERM MORTALITY IN PATIENTS WITH ALCOHOLIC HEPATITIS
30. 485 IDENTIFICATION OF CCL20 AS A NOVEL MEDIATOR OF INFLAMMATION AND FIBROSIS IN ALCOHOLIC HEPATITIS
31. 858 UNIVERSAL HEPATOCYTE DIFFERENTIATION PROTOCOL FOR MULTIPOTENT ADULT PROGENITOR CELLS, EMBRYONIC AND INDUCED PLURIPOTENT STEM CELLS
32. Stem and progenitor cells for liver repopulation: can we standardise the process from bench to bedside?
33. 106 A role of resistin as a mediator of hepatic inflammation and oxidative stress
34. 18 Norepinephrine induces inflammatory actions and calcium mobilization in human hepatic stellate cells through alpha receptors
35. Human hepatic stellate cells express membrane proteins involved in antigen presentation and T-cell activation and internalize extracellular material
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