237 results on '"Van Remmen, Holly"'
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2. Lipid Peroxidation is a Critical Feedback Regulator of Mitochondria in Articular Chondrocytes
3. Elevated phospholipid hydroperoxide glutathione peroxidase (GPX4) expression modulates oxylipin formation and inhibits age-related skeletal muscle atrophy and weakness
4. Senolytics do not prevent muscle loss in Sod1 knockout mice
5. Long-term methamphetamine self-administration increases mesolimbic mitochondrial oxygen consumption and decreases striatal glutathione
6. Neuronal deletion of MnSOD in mice leads to demyelination, inflammation and progressive paralysis that mimics phenotypes associated with progressive multiple sclerosis
7. Acylated Ghrelin Receptor Agonist HM01 Decreases Lean Body and Muscle Mass, but Unacylated Ghrelin Protects against Redox-Dependent Sarcopenia
8. Scavenging lipid hydroperoxides by glutathione peroxidase 4 overexpression attenuates contractile and mitochondrial dysfunction in a murine model of accelerated sarcopenia.
9. Lipid hydroperoxides and oxylipins are mediators of denervation induced muscle atrophy
10. Decreased mitochondrial respiration in cardiac fibers isolated from a mouse model of Friedreich’s ataxia
11. The Role of Oxylipins in Muscle Atrophy
12. Impact of aging and oxidative stress on specific components of excitation contraction coupling in regulating force generation
13. Methamphetamine Self-Administration Differential Effects on Mesolimbic Glutathione Levels, Mitochondrial Respiration and Dopamine Neuron Firing Activity
14. Senolytic treatment reduces cell senescence and necroptosis in Sod1 knockout mice that is associated with reduced inflammation and hepatocellular carcinoma
15. Guidelines for measuring reactive oxygen species and oxidative damage in cells and in vivo
16. Selective Ablation of Sod2 in Astrocytes Induces Sex-Specific Effects on Cognitive Function, d-Serine Availability, and Astrogliosis
17. Senolytic Treatment Reduces Cell Senescence and Necroptosis in Sod1 Knockout Mice that is Associated with Reduced Inflammation and Hepatocellular Carcinoma
18. Age Related Changes in Muscle Mass and Force Generation in the Triple Transgenic (3xTgAD) Mouse Model of Alzheimer’s Disease
19. Scavenging mitochondrial hydrogen peroxide by peroxiredoxin 3 overexpression attenuates contractile dysfunction and muscle atrophy in a murine model of accelerated sarcopenia
20. Impact of elevated oxidative stress on mechanisms related to muscle force generation
21. Lipid hydroperoxides are key drivers of denervation-induced muscle atrophy
22. Deletion of Sod1 in motor neurons exacerbates age-related changes in axons and NMJs associated with premature muscle atrophy in aging mice
23. Removal of p16INK4 Expressing Cells in Late Life has Moderate Beneficial Effects on Skeletal Muscle Function in Male Mice
24. Pharmacologic treatment with OKN-007 reduces alpha-motor neuron loss in spinal cord of aging mice
25. The SarcoEndoplasmic Reticulum Calcium ATPase (SERCA) pump: a potential target for intervention in aging and skeletal muscle pathologies
26. Deletion of Neuronal CuZnSOD Accelerates Age-Associated Muscle Mitochondria and Calcium Handling Dysfunction That Is Independent of Denervation and Precedes Sarcopenia
27. Oklahoma Nathan Shock Aging Center — assessing the basic biology of aging from genetics to protein and function
28. Muscle mitochondrial catalase expression prevents neuromuscular junction disruption, atrophy, and weakness in a mouse model of accelerated sarcopenia
29. Determining the contributions of protein synthesis and breakdown to muscle atrophy requires non‐steady‐state equations
30. Tumor burden negatively impacts protein turnover as a proteostatic process in non-cancerous liver, heart, and muscle, but not brain
31. Unacylated ghrelin ameliorates muscle atrophy and contractile dysfunction in age‐associated loss of muscle mass and function
32. SOD1 regulates ribosome biogenesis in KRAS mutant non-small cell lung cancer
33. Transgenic expression of SOD1 specifically in neurons of Sod1 deficient mice prevents defects in muscle mitochondrial function and calcium handling
34. Neuron specific lack of SOD1 leads to accelerated age-related loss of NMJ structure
35. Sirt5 Deficiency Causes Posttranslational Protein Malonylation and Dysregulated Cellular Metabolism in Chondrocytes Under Obesity Conditions
36. A Novel Stable Isotope Approach Demonstrates Surprising Degree of Age-Related Decline in Skeletal Muscle Collagen Proteostasis
37. Reduced adenosine diphosphate sensitivity in skeletal muscle mitochondria increases reactive oxygen species production in mouse models of aging and oxidative stress but not denervation
38. Restoration of Sarcoplasmic Reticulum Ca2+ ATPase (SERCA) Activity Prevents Age-Related Muscle Atrophy and Weakness in Mice
39. Sirt5 deficiency causes post-translational protein malonylation and dysregulated cellular metabolism in chondrocytes under obesity conditions
40. The Effect of Skeletal Muscle Specific Mitochondrial Catalase (mmCAT) on Restoring Muscle Functions and Reducing Oxidative Stress in SOD1KO Mice
41. The lipid hydroperoxide scavenger liproxstatin-1 reduces denervation-induced muscle pathology
42. Unacylated Ghrelin Protects Against Neurogenic Atrophy and Contractile Dysfunction in a Redox-dependent Sarcopenia
43. Lack of SOD1 In Neuronal Tissue Affects Neuromuscular Junction Stability in Advanced Age
44. An Objective Appraisal of the Free Radical Theory of Aging
45. Contributors
46. Mitochondria, Oxidative Damage and Longevity: What Can Comparative Biology Teach Us?
47. The Anti-Ageing Action of Dietary Restriction
48. Cancer cachexia in a mouse model of oxidative stress
49. Neuron‐specific deletion of CuZnSOD leads to an advanced sarcopenic phenotype in older mice
50. Targeting cPLA2 derived lipid hydroperoxides as a potential intervention for sarcopenia
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