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1. Colitis-Induced Small Intestinal Hypomotility Is Dependent on Enteroendocrine Cell Loss in Mice

3. Human milk oligosaccharides reduce necrotizing enterocolitis induced-neuroinflammation and cognitive impairment in mice

9. The administration of amnion-derived multipotent cell secretome ST266 protects against necrotizing enterocolitis in mice and piglets

10. 460: TOLL-LIKE RECEPTOR 4 (TLR4) REGULATES MUCOSAL REGENERATION DURING ACUTE COLITIS.

11. Mo1670: BIOACTIVE LIPID DIET PREVENTS INJURY AND ENHANCES DEVELOPMENT IN NECROTIZING ENTEROCOLITIS

12. EP1373: SINGLE CELL ANALYSIS OF HUMAN INTESTINAL ORGANOIDS REVEALS GENE NETWORKS DRIVING ADAPTATION IN SBS

15. The administration of a pre-digested fat-enriched formula prevents necrotising enterocolitis-induced lung injury in mice

17. Toll-like receptor 4–mediated enteric glia loss is critical for the development of necrotizing enterocolitis

22. 573 THE HUMAN MILK OLIGOSACCHARIDES 2’-FUCOSYLLACTOSE AND 6’-SIAYLLACTOSE ATTENUATE NEC-ASSOCIATED BRAIN INJURY

24. Maternal aryl hydrocarbon receptor activation protects newborns against necrotizing enterocolitis

25. Necrotizing enterocolitis induces T lymphocyte–mediated injury in the developing mammalian brain

26. Insights image for “The human milk oligosaccharides 2’-fucosyllactose and 6’-sialyllactose protect against the development of necrotizing enterocolitis by inhibiting toll-like receptor 4 signaling.”

29. The human milk oligosaccharides 2’-fucosyllactose and 6’-sialyllactose protect against the development of necrotizing enterocolitis by inhibiting toll-like receptor 4 signaling

30. A Novel Role for Necroptosis in the Pathogenesis of Necrotizing Enterocolitis

31. A Dynamic Variation of Pulmonary ACE2 Is Required to Modulate Neutrophilic Inflammation in Response to Pseudomonas aeruginosa Lung Infection in Mice

33. Cognitive impairments induced by necrotizing enterocolitis can be prevented by inhibiting microglial activation in mouse brain

34. Attenuation of pulmonary ACE2 activity impairs inactivation of des-Arg9 bradykinin/BKB1R axis and facilitates LPS-induced neutrophil infiltration

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