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6 results on '"Tan, Kai"'

1. Transcriptomics of rhinovirus persistence reveals sustained expression of RIG‐I and interferon‐stimulated genes in nasal epithelial cells in vitro

Author

Ong, Hsiao Hui, primary, Andiappan, Anand Kumar, additional, Duan, Kaibo, additional, Lum, Josephine, additional, Liu, Jing, additional, Tan, Kai Sen, additional, Howland, Shanshan, additional, Lee, Bernett, additional, Ong, Yew Kwang, additional, Thong, Mark, additional, Chow, Vincent T., additional, and Wang, De‐Yun, additional

Published
2022
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2. Corrigendum: Role of yes‐associated protein in interleukin‐13 induced nasal remodeling of chronic rhinosinusitis with nasal polyps

Author

Yuan, Tian, primary, Zheng, Rui, additional, Liu, Jing, additional, Tan, Kai Sen, additional, Huang, Zhi‐qun, additional, Zhou, Xiang‐Min, additional, Zi, Xiao‐xue, additional, Qiu, Hui‐jun, additional, Wang, Xin‐yue, additional, Wang, Wei‐hao, additional, Deng, Hui‐yi, additional, Chen, Yu‐bin, additional, Kong, Wei‐feng, additional, Wu, Qing‐wu, additional, Huang, Ying, additional, Ong, Hsiao Hui, additional, Huang, Xue‐kun, additional, Chen, Zhuang‐gui, additional, Wang, De‐Yun, additional, and Yang, Qin‐Tai, additional

Published
2022
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3. Role of yes‐associated protein in interleukin‐13 induced nasal remodeling of chronic rhinosinusitis with nasal polyps

Author

Yuan, Tian, primary, Zheng, Rui, additional, Liu, Jing, additional, Tan, Kai Sen, additional, Huang, Zhi‐qun, additional, Zhou, Xiang‐Min, additional, Zi, Xiao‐xue, additional, Qiu, Hui‐jun, additional, Wang, Xin‐yue, additional, Wang, Wei‐hao, additional, Deng, Hui‐yi, additional, Chen, Yu‐bin, additional, Kong, Wei‐feng, additional, Wu, Qing‐wu, additional, Huang, Ying, additional, Ong, Hsiao Hui, additional, Huang, Xue‐kun, additional, Chen, Zhuang‐gui, additional, Wang, De‐Yun, additional, and Yang, Qin‐Tai, additional

Published
2021
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4. Hypoxia‐induced factor‐1α induces NLRP3 expression by M1 macrophages in noneosinophilic chronic rhinosinusitis with nasal polyps

Author

Zhong, Bing, primary, Du, Jintao, additional, Liu, Feng, additional, Liu, Yafeng, additional, Liu, Shixi, additional, Zhang, Jie, additional, Lin, Ping, additional, Zhou, Jiao, additional, Liu, Jing, additional, Ong, Hsiao Hui, additional, Tan, Kai Sen, additional, Wang, Deyun, additional, and Ba, Luo, additional

Published
2020
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5. Hypoxia‐induced factor‐1α induces NLRP3 expression by M1 macrophages in noneosinophilic chronic rhinosinusitis with nasal polyps.

Author

Zhong, Bing, Du, Jintao, Liu, Feng, Liu, Yafeng, Liu, Shixi, Zhang, Jie, Lin, Ping, Zhou, Jiao, Liu, Jing, Ong, Hsiao Hui, Tan, Kai Sen, Wang, Deyun, and Ba, Luo

Subjects
NASAL polyps, MACROPHAGES, SINUSITIS
Abstract

Hypoxia-induced factor-1 induces NLRP3 expression by M1 macrophages in noneosinophilic chronic rhinosinusitis with nasal polyps Keywords: chronic rhinosinusitis with nasal polyps; hypoxia-induced factor-1 ; inflammation; macrophage; NOD-like receptor family; pyrin domain containing 3 EN chronic rhinosinusitis with nasal polyps hypoxia-induced factor-1 inflammation macrophage NOD-like receptor family pyrin domain containing 3 582 586 5 02/20/21 20210201 NES 210201 To the Editor, Chronic rhinosinusitis with nasal polyps (CRSwNP) is a widespread chronic inflammatory disease of the nasal mucosa and can manifest in different phenotypes. HIF-1 , hypoxia-induced factor-1 ; NLRP3, NOD-like receptor family, pyrin domain containing 3; eosCRSwNP, eosinophilic chronic rhinosinusitis with nasal polyps gl Additionally, we found that Arg-1-positive type II macrophage was mainly observed in eosCRSwNP, while the INOS-positive type I macrophage was observed mostly in non-eosCRSwNP. [Extracted from the article]

Published
2021
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6. Type 2‐low asthma phenotypes by integration of sputum transcriptomics and serum proteomics.

Author

Zounemat Kermani, Nazanin, Saqi, Mansoor, Agapow, Paul, Pavlidis, Stelios, Kuo, Chihhsi, Tan, Kai Sen, Mumby, Sharon, Sun, Kai, Loza, Matthew, Baribaud, Frederic, Sousa, Ana R., Riley, John, Wheelock, Asa M., Wheelock, Craig E., De Meulder, Bertrand, Schofield, Jim, Sánchez‐Ovando, Stephany, Simpson, Jodie Louise, Baines, Katherine Joanne, and Wark, Peter A.

Subjects
PROTEOMICS, SPUTUM, ASTHMA, SERUM, TRANSCRIPTOMES, PHENOTYPES
Abstract

Keywords: asthma; bioinformatics; endotypes; precision medicine; systems biology EN asthma bioinformatics endotypes precision medicine systems biology 380 383 4 01/09/21 20210101 NES 210101 I To the Editor, i Asthma is a complex heterogeneous disease that presents with varying degrees of severity. We identified four optimal clusters (TAC*1, TAC*2, TAC*3a and TAC*3b) (Figure S3), in agreement with our previous clustering1 where TAC*3a and TAC*3b were combined. Asthma, bioinformatics, endotypes, precision medicine, systems biology. [Extracted from the article]

Published
2021
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