32 results on '"Magalhães ME"'
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2. Capítulo 3 - Avaliação Clínica e Complementar
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Malachias, MVB, primary, Souza, WKSB, additional, Plavnik, FL, additional, Rodrigues, CIS, additional, Brandão, AA, additional, Neves, MFT, additional, Bortolotto, LA, additional, Franco, RJS, additional, Figueiredo, CEP, additional, Jardim, PCBV, additional, Amodeo, C, additional, Barbosa, ECD, additional, Koch, V, additional, Gomes, MAM, additional, Paula, RB, additional, Póvoa, RMS, additional, Colombo, FC, additional, Ferreira Filho, S, additional, Miranda, RD, additional, Machado, CA, additional, Nobre, F, additional, Nogueira, AR, additional, Mion Júnior, D, additional, Kaiser, S, additional, Forjaz, CLM, additional, Almeida, FA, additional, Martim, JFV, additional, Sass, N, additional, Drager, LF, additional, Muxfeldt, E, additional, Bodanese, LC, additional, Feitosa, AD, additional, Malta, D, additional, Fuchs, S, additional, Magalhães, ME, additional, Oigman, W, additional, Gomes, OM, additional, Pierin, AMG, additional, Feitosa, GS, additional, Bortolotto, MRFL, additional, Magalhães, LBNC, additional, Silva, ACS, additional, Ribeiro, JM, additional, Borelli, FAO, additional, Gus, M, additional, Passarelli Júnior, O, additional, Toledo, JY, additional, Salles, GF, additional, Martins, LC, additional, Jardim, TSV, additional, Guimarães, ICB, additional, Antonello, IC, additional, Lima Júnior, E, additional, Matsudo, V, additional, Silva, GV, additional, Costa, LS, additional, Alessi, A, additional, Scala, LCN, additional, Coelho, EB, additional, Souza, D, additional, Lopes, HF, additional, Gowdak, MMG, additional, Cordeiro Júnior, AC, additional, Torloni, MR, additional, Klein, MRST, additional, Nogueira, PK, additional, Lotaif, LAD, additional, Rosito, GBA, additional, and Moreno Júnior, H, additional
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- 2016
- Full Text
- View/download PDF
3. Capítulo 12 - Hipertensão Arterial Secundária
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Malachias, MVB, primary, Souza, WKSB, additional, Plavnik, FL, additional, Rodrigues, CIS, additional, Brandão, AA, additional, Neves, MFT, additional, Bortolotto, LA, additional, Franco, RJS, additional, Figueiredo, CEP, additional, Jardim, PCBV, additional, Amodeo, C, additional, Barbosa, ECD, additional, Koch, V, additional, Gomes, MAM, additional, Paula, RB, additional, Póvoa, RMS, additional, Colombo, FC, additional, Ferreira Filho, S, additional, Miranda, RD, additional, Machado, CA, additional, Nobre, F, additional, Nogueira, AR, additional, Mion Júnior, D, additional, Kaiser, S, additional, Forjaz, CLM, additional, Almeida, FA, additional, Martim, JFV, additional, Sass, N, additional, Drager, LF, additional, Muxfeldt, E, additional, Bodanese, LC, additional, Feitosa, AD, additional, Malta, D, additional, Fuchs, S, additional, Magalhães, ME, additional, Oigman, W, additional, Gomes, OM, additional, Pierin, AMG, additional, Feitosa, GS, additional, Bortolotto, MRFL, additional, Magalhães, LBNC, additional, Silva, ACS, additional, Ribeiro, JM, additional, Borelli, FAO, additional, Gus, M, additional, Passarelli Júnior, O, additional, Toledo, JY, additional, Salles, GF, additional, Martins, LC, additional, Jardim, TSV, additional, Guimarães, ICB, additional, Antonello, IC, additional, Lima Júnior, E, additional, Matsudo, V, additional, Silva, GV, additional, Costa, LS, additional, Alessi, A, additional, Scala, LCN, additional, Coelho, EB, additional, Souza, D, additional, Lopes, HF, additional, Gowdak, MMG, additional, Cordeiro Júnior, AC, additional, Torloni, MR, additional, Klein, MRST, additional, Nogueira, PK, additional, Lotaif, LAD, additional, Rosito, GBA, additional, and Moreno Júnior, H, additional
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- 2016
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4. Capítulo 4 - Estratificaçã o de Risco Cardiovascular
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Malachias, MVB, primary, Souza, WKSB, additional, Plavnik, FL, additional, Rodrigues, CIS, additional, Brandão, AA, additional, Neves, MFT, additional, Bortolotto, LA, additional, Franco, RJS, additional, Figueiredo, CEP, additional, Jardim, PCBV, additional, Amodeo, C, additional, Barbosa, ECD, additional, Koch, V, additional, Gomes, MAM, additional, Paula, RB, additional, Póvoa, RMS, additional, Colombo, FC, additional, Ferreira Filho, S, additional, Miranda, RD, additional, Machado, CA, additional, Nobre, F, additional, Nogueira, AR, additional, Mion Júnior, D, additional, Kaiser, S, additional, Forjaz, CLM, additional, Almeida, FA, additional, Martim, JFV, additional, Sass, N, additional, Drager, LF, additional, Muxfeldt, E, additional, Bodanese, LC, additional, Feitosa, AD, additional, Malta, D, additional, Fuchs, S, additional, Magalhães, ME, additional, Oigman, W, additional, Gomes, OM, additional, Pierin, AMG, additional, Feitosa, GS, additional, Bortolotto, MRFL, additional, Magalhães, LBNC, additional, Silva, ACS, additional, Ribeiro, JM, additional, Borelli, FAO, additional, Gus, M, additional, Passarelli Júnior, O, additional, Toledo, JY, additional, Salles, GF, additional, Martins, LC, additional, Jardim, TSV, additional, Guimarães, ICB, additional, Antonello, IC, additional, Lima Júnior, E, additional, Matsudo, V, additional, Silva, GV, additional, Costa, LS, additional, Alessi, A, additional, Scala, LCN, additional, Coelho, EB, additional, Souza, D, additional, Lopes, HF, additional, Gowdak, MMG, additional, Cordeiro Júnior, AC, additional, Torloni, MR, additional, Klein, MRST, additional, Nogueira, PK, additional, Lotaif, LAD, additional, Rosito, GBA, additional, and Moreno Júnior, H, additional
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- 2016
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5. Capítulo 13 - Hipertensão Arterial Resistente
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Malachias, MVB, primary, Souza, WKSB, additional, Plavnik, FL, additional, Rodrigues, CIS, additional, Brandão, AA, additional, Neves, MFT, additional, Bortolotto, LA, additional, Franco, RJS, additional, Figueiredo, CEP, additional, Jardim, PCBV, additional, Amodeo, C, additional, Barbosa, ECD, additional, Koch, V, additional, Gomes, MAM, additional, Paula, RB, additional, Póvoa, RMS, additional, Colombo, FC, additional, Ferreira Filho, S, additional, Miranda, RD, additional, Machado, CA, additional, Nobre, F, additional, Nogueira, AR, additional, Mion Júnior, D, additional, Kaiser, S, additional, Forjaz, CLM, additional, Almeida, FA, additional, Martim, JFV, additional, Sass, N, additional, Drager, LF, additional, Muxfeldt, E, additional, Bodanese, LC, additional, Feitosa, AD, additional, Malta, D, additional, Fuchs, S, additional, Magalhães, ME, additional, Oigman, W, additional, Gomes, OM, additional, Pierin, AMG, additional, Feitosa, GS, additional, Bortolotto, MRFL, additional, Magalhães, LBNC, additional, Silva, ACS, additional, Ribeiro, JM, additional, Borelli, FAO, additional, Gus, M, additional, Passarelli Júnior, O, additional, Toledo, JY, additional, Salles, GF, additional, Martins, LC, additional, Jardim, TSV, additional, Guimarães, ICB, additional, Antonello, IC, additional, Lima Júnior, E, additional, Matsudo, V, additional, Silva, GV, additional, Costa, LS, additional, Alessi, A, additional, Scala, LCN, additional, Coelho, EB, additional, Souza, D, additional, Lopes, HF, additional, Gowdak, MMG, additional, Cordeiro Júnior, AC, additional, Torloni, MR, additional, Klein, MRST, additional, Nogueira, PK, additional, Lotaif, LAD, additional, Rosito, GBA, additional, and Moreno Júnior, H, additional
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- 2016
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6. Capítulo 5 - Decisão e Metas Terapêuticas
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Malachias, MVB, primary, Souza, WKSB, additional, Plavnik, FL, additional, Rodrigues, CIS, additional, Brandão, AA, additional, Neves, MFT, additional, Bortolotto, LA, additional, Franco, RJS, additional, Figueiredo, CEP, additional, Jardim, PCBV, additional, Amodeo, C, additional, Barbosa, ECD, additional, Koch, V, additional, Gomes, MAM, additional, Paula, RB, additional, Póvoa, RMS, additional, Colombo, FC, additional, Ferreira Filho, S, additional, Miranda, RD, additional, Machado, CA, additional, Nobre, F, additional, Nogueira, AR, additional, Mion Júnior, D, additional, Kaiser, S, additional, Forjaz, CLM, additional, Almeida, FA, additional, Martim, JFV, additional, Sass, N, additional, Drager, LF, additional, Muxfeldt, E, additional, Bodanese, LC, additional, Feitosa, AD, additional, Malta, D, additional, Fuchs, S, additional, Magalhães, ME, additional, Oigman, W, additional, Gomes, OM, additional, Pierin, AMG, additional, Feitosa, GS, additional, Bortolotto, MRFL, additional, Magalhães, LBNC, additional, Silva, ACS, additional, Ribeiro, JM, additional, Borelli, FAO, additional, Gus, M, additional, Passarelli Júnior, O, additional, Toledo, JY, additional, Salles, GF, additional, Martins, LC, additional, Jardim, TSV, additional, Guimarães, ICB, additional, Antonello, IC, additional, Lima Júnior, E, additional, Matsudo, V, additional, Silva, GV, additional, Costa, LS, additional, Alessi, A, additional, Scala, LCN, additional, Coelho, EB, additional, Souza, D, additional, Lopes, HF, additional, Gowdak, MMG, additional, Cordeiro Júnior, AC, additional, Torloni, MR, additional, Klein, MRST, additional, Nogueira, PK, additional, Lotaif, LAD, additional, Rosito, GBA, additional, and Moreno Júnior, H, additional
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- 2016
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7. Capítulo 1 - Conceituação, Epidemiologia e Prevenção Primária
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Malachias, MVB, primary, Souza, WKSB, additional, Plavnik, FL, additional, Rodrigues, CIS, additional, Brandão, AA, additional, Neves, MFT, additional, Bortolotto, LA, additional, Franco, RJS, additional, Figueiredo, CEP, additional, Jardim, PCBV, additional, Amodeo, C, additional, Barbosa, ECD, additional, Koch, V, additional, Gomes, MAM, additional, Paula, RB, additional, Póvoa, RMS, additional, Colombo, FC, additional, Ferreira Filho, S, additional, Miranda, RD, additional, Machado, CA, additional, Nobre, F, additional, Nogueira, AR, additional, Mion Júnior, D, additional, Kaiser, S, additional, Forjaz, CLM, additional, Almeida, FA, additional, Martim, JFV, additional, Sass, N, additional, Drager, LF, additional, Muxfeldt, E, additional, Bodanese, LC, additional, Feitosa, AD, additional, Malta, D, additional, Fuchs, S, additional, Magalhães, ME, additional, Oigman, W, additional, Gomes, OM, additional, Pierin, AMG, additional, Feitosa, GS, additional, Bortolotto, MRFL, additional, Magalhães, LBNC, additional, Silva, ACS, additional, Ribeiro, JM, additional, Borelli, FAO, additional, Gus, M, additional, Passarelli Júnior, O, additional, Toledo, JY, additional, Salles, GF, additional, Martins, LC, additional, Jardim, TSV, additional, Guimarães, ICB, additional, Antonello, IC, additional, Lima Júnior, E, additional, Matsudo, V, additional, Silva, GV, additional, Costa, LS, additional, Alessi, A, additional, Scala, LCN, additional, Coelho, EB, additional, Souza, D, additional, Lopes, HF, additional, Gowdak, MMG, additional, Cordeiro Júnior, AC, additional, Torloni, MR, additional, Klein, MRST, additional, Nogueira, PK, additional, Lotaif, LAD, additional, Rosito, GBA, additional, and Moreno Júnior, H, additional
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- 2016
- Full Text
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8. Capítulo 10 - Hipertensão na Criança e no Adolescente
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Malachias, MVB, primary, Souza, WKSB, additional, Plavnik, FL, additional, Rodrigues, CIS, additional, Brandão, AA, additional, Neves, MFT, additional, Bortolotto, LA, additional, Franco, RJS, additional, Figueiredo, CEP, additional, Jardim, PCBV, additional, Amodeo, C, additional, Barbosa, ECD, additional, Koch, V, additional, Gomes, MAM, additional, Paula, RB, additional, Póvoa, RMS, additional, Colombo, FC, additional, Ferreira Filho, S, additional, Miranda, RD, additional, Machado, CA, additional, Nobre, F, additional, Nogueira, AR, additional, Mion Júnior, D, additional, Kaiser, S, additional, Forjaz, CLM, additional, Almeida, FA, additional, Martim, JFV, additional, Sass, N, additional, Drager, LF, additional, Muxfeldt, E, additional, Bodanese, LC, additional, Feitosa, AD, additional, Malta, D, additional, Fuchs, S, additional, Magalhães, ME, additional, Oigman, W, additional, Gomes, OM, additional, Pierin, AMG, additional, Feitosa, GS, additional, Bortolotto, MRFL, additional, Magalhães, LBNC, additional, Silva, ACS, additional, Ribeiro, JM, additional, Borelli, FAO, additional, Gus, M, additional, Passarelli Júnior, O, additional, Toledo, JY, additional, Salles, GF, additional, Martins, LC, additional, Jardim, TSV, additional, Guimarães, ICB, additional, Antonello, IC, additional, Lima Júnior, E, additional, Matsudo, V, additional, Silva, GV, additional, Costa, LS, additional, Alessi, A, additional, Scala, LCN, additional, Coelho, EB, additional, Souza, D, additional, Lopes, HF, additional, Gowdak, MMG, additional, Cordeiro Júnior, AC, additional, Torloni, MR, additional, Klein, MRST, additional, Nogueira, PK, additional, Lotaif, LAD, additional, Rosito, GBA, additional, and Moreno Júnior, H, additional
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- 2016
- Full Text
- View/download PDF
9. Capítulo 14 – Crise Hipertensiva
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Malachias, MVB, primary, Souza, WKSB, additional, Plavnik, FL, additional, Rodrigues, CIS, additional, Brandão, AA, additional, Neves, MFT, additional, Bortolotto, LA, additional, Franco, RJS, additional, Figueiredo, CEP, additional, Jardim, PCBV, additional, Amodeo, C, additional, Barbosa, ECD, additional, Koch, V, additional, Gomes, MAM, additional, Paula, RB, additional, Póvoa, RMS, additional, Colombo, FC, additional, Ferreira Filho, S, additional, Miranda, RD, additional, Machado, CA, additional, Nobre, F, additional, Nogueira, AR, additional, Mion Júnior, D, additional, Kaiser, S, additional, Forjaz, CLM, additional, Almeida, FA, additional, Martim, JFV, additional, Sass, N, additional, Drager, LF, additional, Muxfeldt, E, additional, Bodanese, LC, additional, Feitosa, AD, additional, Malta, D, additional, Fuchs, S, additional, Magalhães, ME, additional, Oigman, W, additional, Gomes, OM, additional, Pierin, AMG, additional, Feitosa, GS, additional, Bortolotto, MRFL, additional, Magalhães, LBNC, additional, Silva, ACS, additional, Ribeiro, JM, additional, Borelli, FAO, additional, Gus, M, additional, Passarelli Júnior, O, additional, Toledo, JY, additional, Salles, GF, additional, Martins, LC, additional, Jardim, TSV, additional, Guimarães, ICB, additional, Antonello, IC, additional, Lima Júnior, E, additional, Matsudo, V, additional, Silva, GV, additional, Costa, LS, additional, Alessi, A, additional, Scala, LCN, additional, Coelho, EB, additional, Souza, D, additional, Lopes, HF, additional, Gowdak, MMG, additional, Cordeiro Júnior, AC, additional, Torloni, MR, additional, Klein, MRST, additional, Nogueira, PK, additional, Lotaif, LAD, additional, Rosito, GBA, additional, and Moreno Júnior, H, additional
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- 2016
- Full Text
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10. Capítulo 8 - Hipertensão e Condições Clínicas Associadas
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Malachias, MVB, primary, Souza, WKSB, additional, Plavnik, FL, additional, Rodrigues, CIS, additional, Brandão, AA, additional, Neves, MFT, additional, Bortolotto, LA, additional, Franco, RJS, additional, Figueiredo, CEP, additional, Jardim, PCBV, additional, Amodeo, C, additional, Barbosa, ECD, additional, Koch, V, additional, Gomes, MAM, additional, Paula, RB, additional, Póvoa, RMS, additional, Colombo, FC, additional, Ferreira Filho, S, additional, Miranda, RD, additional, Machado, CA, additional, Nobre, F, additional, Nogueira, AR, additional, Mion Júnior, D, additional, Kaiser, S, additional, Forjaz, CLM, additional, Almeida, FA, additional, Martim, JFV, additional, Sass, N, additional, Drager, LF, additional, Muxfeldt, E, additional, Bodanese, LC, additional, Feitosa, AD, additional, Malta, D, additional, Fuchs, S, additional, Magalhães, ME, additional, Oigman, W, additional, Gomes, OM, additional, Pierin, AMG, additional, Feitosa, GS, additional, Bortolotto, MRFL, additional, Magalhães, LBNC, additional, Silva, ACS, additional, Ribeiro, JM, additional, Borelli, FAO, additional, Gus, M, additional, Passarelli Júnior, O, additional, Toledo, JY, additional, Salles, GF, additional, Martins, LC, additional, Jardim, TSV, additional, Guimarães, ICB, additional, Antonello, IC, additional, Lima Júnior, E, additional, Matsudo, V, additional, Silva, GV, additional, Costa, LS, additional, Alessi, A, additional, Scala, LCN, additional, Coelho, EB, additional, Souza, D, additional, Lopes, HF, additional, Gowdak, MMG, additional, Cordeiro Júnior, AC, additional, Torloni, MR, additional, Klein, MRST, additional, Nogueira, PK, additional, Lotaif, LAD, additional, Rosito, GBA, additional, and Moreno Júnior, H, additional
- Published
- 2016
- Full Text
- View/download PDF
11. Capítulo 11 - Hipertensão Arterial no Idoso
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Malachias, MVB, primary, Souza, WKSB, additional, Plavnik, FL, additional, Rodrigues, CIS, additional, Brandão, AA, additional, Neves, MFT, additional, Bortolotto, LA, additional, Franco, RJS, additional, Figueiredo, CEP, additional, Jardim, PCBV, additional, Amodeo, C, additional, Barbosa, ECD, additional, Koch, V, additional, Gomes, MAM, additional, Paula, RB, additional, Póvoa, RMS, additional, Colombo, FC, additional, Ferreira Filho, S, additional, Miranda, RD, additional, Machado, CA, additional, Nobre, F, additional, Nogueira, AR, additional, Mion Júnior, D, additional, Kaiser, S, additional, Forjaz, CLM, additional, Almeida, FA, additional, Martim, JFV, additional, Sass, N, additional, Drager, LF, additional, Muxfeldt, E, additional, Bodanese, LC, additional, Feitosa, AD, additional, Malta, D, additional, Fuchs, S, additional, Magalhães, ME, additional, Oigman, W, additional, Gomes, OM, additional, Pierin, AMG, additional, Feitosa, GS, additional, Bortolotto, MRFL, additional, Magalhães, LBNC, additional, Silva, ACS, additional, Ribeiro, JM, additional, Borelli, FAO, additional, Gus, M, additional, Passarelli Júnior, O, additional, Toledo, JY, additional, Salles, GF, additional, Martins, LC, additional, Jardim, TSV, additional, Guimarães, ICB, additional, Antonello, IC, additional, Lima Júnior, E, additional, Matsudo, V, additional, Silva, GV, additional, Costa, LS, additional, Alessi, A, additional, Scala, LCN, additional, Coelho, EB, additional, Souza, D, additional, Lopes, HF, additional, Gowdak, MMG, additional, Cordeiro Júnior, AC, additional, Torloni, MR, additional, Klein, MRST, additional, Nogueira, PK, additional, Lotaif, LAD, additional, Rosito, GBA, additional, and Moreno Júnior, H, additional
- Published
- 2016
- Full Text
- View/download PDF
12. Capítulo 6 - Tratamento não medicamentoso
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Malachias, MVB, primary, Souza, WKSB, additional, Plavnik, FL, additional, Rodrigues, CIS, additional, Brandão, AA, additional, Neves, MFT, additional, Bortolotto, LA, additional, Franco, RJS, additional, Figueiredo, CEP, additional, Jardim, PCBV, additional, Amodeo, C, additional, Barbosa, ECD, additional, Koch, V, additional, Gomes, MAM, additional, Paula, RB, additional, Póvoa, RMS, additional, Colombo, FC, additional, Ferreira Filho, S, additional, Miranda, RD, additional, Machado, CA, additional, Nobre, F, additional, Nogueira, AR, additional, Mion Júnior, D, additional, Kaiser, S, additional, Forjaz, CLM, additional, Almeida, FA, additional, Martim, JFV, additional, Sass, N, additional, Drager, LF, additional, Muxfeldt, E, additional, Bodanese, LC, additional, Feitosa, AD, additional, Malta, D, additional, Fuchs, S, additional, Magalhães, ME, additional, Oigman, W, additional, Gomes, OM, additional, Pierin, AMG, additional, Feitosa, GS, additional, Bortolotto, MRFL, additional, Magalhães, LBNC, additional, Silva, ACS, additional, Ribeiro, JM, additional, Borelli, FAO, additional, Gus, M, additional, Passarelli Júnior, O, additional, Toledo, JY, additional, Salles, GF, additional, Martins, LC, additional, Jardim, TSV, additional, Guimarães, ICB, additional, Antonello, IC, additional, Lima Júnior, E, additional, Matsudo, V, additional, Silva, GV, additional, Costa, LS, additional, Alessi, A, additional, Scala, LCN, additional, Coelho, EB, additional, Souza, D, additional, Lopes, HF, additional, Gowdak, MMG, additional, Cordeiro Júnior, AC, additional, Torloni, MR, additional, Klein, MRST, additional, Nogueira, PK, additional, Lotaif, LAD, additional, Rosito, GBA, additional, and Moreno Júnior, H, additional
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- 2016
- Full Text
- View/download PDF
13. Capítulo 9 - Hipertensão Arterial na gestação
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Malachias, MVB, primary, Souza, WKSB, additional, Plavnik, FL, additional, Rodrigues, CIS, additional, Brandão, AA, additional, Neves, MFT, additional, Bortolotto, LA, additional, Franco, RJS, additional, Figueiredo, CEP, additional, Jardim, PCBV, additional, Amodeo, C, additional, Barbosa, ECD, additional, Koch, V, additional, Gomes, MAM, additional, Paula, RB, additional, Póvoa, RMS, additional, Colombo, FC, additional, Ferreira Filho, S, additional, Miranda, RD, additional, Machado, CA, additional, Nobre, F, additional, Nogueira, AR, additional, Mion Júnior, D, additional, Kaiser, S, additional, Forjaz, CLM, additional, Almeida, FA, additional, Martim, JFV, additional, Sass, N, additional, Drager, LF, additional, Muxfeldt, E, additional, Bodanese, LC, additional, Feitosa, AD, additional, Malta, D, additional, Fuchs, S, additional, Magalhães, ME, additional, Oigman, W, additional, Gomes, OM, additional, Pierin, AMG, additional, Feitosa, GS, additional, Bortolotto, MRFL, additional, Magalhães, LBNC, additional, Silva, ACS, additional, Ribeiro, JM, additional, Borelli, FAO, additional, Gus, M, additional, Passarelli Júnior, O, additional, Toledo, JY, additional, Salles, GF, additional, Martins, LC, additional, Jardim, TSV, additional, Guimarães, ICB, additional, Antonello, IC, additional, Lima Júnior, E, additional, Matsudo, V, additional, Silva, GV, additional, Costa, LS, additional, Alessi, A, additional, Scala, LCN, additional, Coelho, EB, additional, Souza, D, additional, Lopes, HF, additional, Gowdak, MMG, additional, Cordeiro Júnior, AC, additional, Torloni, MR, additional, Klein, MRST, additional, Nogueira, PK, additional, Lotaif, LAD, additional, Rosito, GBA, additional, and Moreno Júnior, H, additional
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- 2016
- Full Text
- View/download PDF
14. Capítulo 7 – Tratamento Medicamentoso
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Malachias, MVB, primary, Souza, WKSB, additional, Plavnik, FL, additional, Rodrigues, CIS, additional, Brandão, AA, additional, Neves, MFT, additional, Bortolotto, LA, additional, Franco, RJS, additional, Figueiredo, CEP, additional, Jardim, PCBV, additional, Amodeo, C, additional, Barbosa, ECD, additional, Koch, V, additional, Gomes, MAM, additional, Paula, RB, additional, Póvoa, RMS, additional, Colombo, FC, additional, Ferreira Filho, S, additional, Miranda, RD, additional, Machado, CA, additional, Nobre, F, additional, Nogueira, AR, additional, Mion Júnior, D, additional, Kaiser, S, additional, Forjaz, CLM, additional, Almeida, FA, additional, Martim, JFV, additional, Sass, N, additional, Drager, LF, additional, Muxfeldt, E, additional, Bodanese, LC, additional, Feitosa, AD, additional, Malta, D, additional, Fuchs, S, additional, Magalhães, ME, additional, Oigman, W, additional, Gomes, OM, additional, Pierin, AMG, additional, Feitosa, GS, additional, Bortolotto, MRFL, additional, Magalhães, LBNC, additional, Silva, ACS, additional, Ribeiro, JM, additional, Borelli, FAO, additional, Gus, M, additional, Passarelli Júnior, O, additional, Toledo, JY, additional, Salles, GF, additional, Martins, LC, additional, Jardim, TSV, additional, Guimarães, ICB, additional, Antonello, IC, additional, Lima Júnior, E, additional, Matsudo, V, additional, Silva, GV, additional, Costa, LS, additional, Alessi, A, additional, Scala, LCN, additional, Coelho, EB, additional, Souza, D, additional, Lopes, HF, additional, Gowdak, MMG, additional, Cordeiro Júnior, AC, additional, Torloni, MR, additional, Klein, MRST, additional, Nogueira, PK, additional, Lotaif, LAD, additional, Rosito, GBA, additional, and Moreno Júnior, H, additional
- Published
- 2016
- Full Text
- View/download PDF
15. Capítulo 2 - Diagnóstico e Classificação
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Malachias, MVB, primary, Souza, WKSB, additional, Plavnik, FL, additional, Rodrigues, CIS, additional, Brandão, AA, additional, Neves, MFT, additional, Bortolotto, LA, additional, Franco, RJS, additional, Figueiredo, CEP, additional, Jardim, PCBV, additional, Amodeo, C, additional, Barbosa, ECD, additional, Koch, V, additional, Gomes, MAM, additional, Paula, RB, additional, Póvoa, RMS, additional, Colombo, FC, additional, Ferreira Filho, S, additional, Miranda, RD, additional, Machado, CA, additional, Nobre, F, additional, Nogueira, AR, additional, Mion Júnior, D, additional, Kaiser, S, additional, Forjaz, CLM, additional, Almeida, FA, additional, Martim, JFV, additional, Sass, N, additional, Drager, LF, additional, Muxfeldt, E, additional, Bodanese, LC, additional, Feitosa, AD, additional, Malta, D, additional, Fuchs, S, additional, Magalhães, ME, additional, Oigman, W, additional, Gomes, OM, additional, Pierin, AMG, additional, Feitosa, GS, additional, Bortolotto, MRFL, additional, Magalhães, LBNC, additional, Silva, ACS, additional, Ribeiro, JM, additional, Borelli, FAO, additional, Gus, M, additional, Passarelli Júnior, O, additional, Toledo, JY, additional, Salles, GF, additional, Martins, LC, additional, Jardim, TSV, additional, Guimarães, ICB, additional, Antonello, IC, additional, Lima Júnior, E, additional, Matsudo, V, additional, Silva, GV, additional, Costa, LS, additional, Alessi, A, additional, Scala, LCN, additional, Coelho, EB, additional, Souza, D, additional, Lopes, HF, additional, Gowdak, MMG, additional, Cordeiro Júnior, AC, additional, Torloni, MR, additional, Klein, MRST, additional, Nogueira, PK, additional, Lotaif, LAD, additional, Rosito, GBA, additional, and Moreno Júnior, H, additional
- Published
- 2016
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16. APOE and LDLR Gene Polymorphisms and Dyslipidemia Tracking. Rio de Janeiro Study.
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Freitas RG, Campana EM, Pozzan R, Brandão AA, Brandão AP, Magalhães ME, and Silva DA
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- Adolescent, Adult, Apolipoprotein E2 genetics, Apolipoprotein E3 genetics, Apolipoprotein E4 genetics, Blood Pressure, Body Mass Index, Brazil, Child, Female, Gene Frequency, Genotype, Humans, Longitudinal Studies, Male, Polymerase Chain Reaction, Risk Factors, Waist Circumference, Young Adult, Apolipoproteins E genetics, Dyslipidemias genetics, Genetic Association Studies, Polymorphism, Genetic genetics, Receptors, LDL genetics
- Abstract
Background: Studies show an association between changes in apolipoprotein E (ApoE) and LDLR receptor with the occurrence of dyslipidemia., Objectives: To investigate the association between polymorphisms of the APOE (ε2, ε3, ε4) and LDLR (A370T) genes with the persistence of abnormal serum lipid levels in young individuals followed up for 17 years in the Rio de Janeiro Study., Methods: The study included 56 individuals (35 males) who underwent three assessments at different ages: A1 (mean age 13.30 ± 1.53 years), A2 (22.09 ± 1.91 years) and A3 (31.23 ± 1.99 years). Clinical evaluation with measurement of blood pressure (BP) and body mass index (BMI) was conducted at all three assessments. Measurement of waist circumference (WC) and serum lipids, and analysis of genetic polymorphisms by PCR-RFLP were performed at A2 and A3. Based on dyslipidemia tracking, three groups were established: 0 (no abnormal lipid value at A2 and A3), 1 (up to one abnormal lipid value at A2 or A3) and 2 (one or more abnormal lipid values at A2 and A3)., Results: Compared with groups 0 and 1, group 2 presented higher mean values of BP, BMI, WC, LDL-c and TG (p < 0.01) and lower mean values of HDL-c (p = 0.001). Across the assessments, all individuals with APOE genotypes ε2/ε4 and ε4/ε4 maintained at least one abnormal lipid variable, whereas those with genotype ε2/ε3 did not show abnormal values (χ2 = 16.848, p = 0.032). For the LDLR genotypes, there was no significant difference among the groups., Conclusions: APOE gene polymorphisms were associated with dyslipidemia in young individuals followed up longitudinally from childhood.
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- 2015
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17. Blood pressure in adolescence, adipokines and inflammation in young adults. The rio de janeiro study.
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Campana EM, Brandão AA, Pozzan R, Magalhães ME, Fonseca FL, Pizzi OL, Freitas EV, and Brandão AP
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- Adolescent, Adult, Age Factors, Apolipoproteins blood, Brazil, Cardiovascular Diseases etiology, Cardiovascular Diseases metabolism, Child, Female, Follow-Up Studies, Humans, Hypertension metabolism, Inflammation Mediators blood, Male, Metabolic Syndrome blood, Reference Values, Risk Factors, Young Adult, Adipokines blood, Anthropometry, Blood Pressure physiology
- Abstract
Background: The impact of blood pressure (BP) during adolescence on other cardiovascular risk factors in young adults is important for the primary prevention., Objective: To evaluate BP, anthropometric indexes, metabolic and inflammatory profiles in young individuals stratified by their BP behavior recorded for 18 years., Methods: A total of 116 individuals, of whom 63 were males, from the Rio de Janeiro study (follow-up of 17.76 ± 1.63 years), were assessed at two moments: A1 (12.40 ± 1.49 years) and A2 (30.09 ± 2.01 years). The 116 individuals were divided into two groups: GN (n = 71), of participants with normal BP at A1; and GH (n = 45), of those with abnormal BP at A1. BP, weight, height and body mass index (BMI) were measured at A1 and A2. At A2, abdominal circumference (AC) and laboratory, metabolic and inflammatory variables were included., Results: 1) No difference was observed between the groups as regards age and gender; 2) At A2, GH showed higher mean weight, BMI, BP, insulin, HOMA-IR (p < 0.001), leptin (p < 0.02), apolipoprotein B100 and A1 (p < 0.02), apolipoprotein B100 / apolipoprotein A1 ratio (p < 0.010); and higher prevalences of overweight/obesity (p < 0.001), of increased AC (p < 0.001) and of hypertension (p < 0.02); 3) No difference was observed between the groups as regards the inflammatory variables; 4) There was a positive correlation of BP at A1 with BP, BMI, insulin, leptin and HOMA-IR at A2 (p < 0.05)., Conclusion: BP in adolescence was associated with higher values of BP, and anthropometric and metabolic variables in young adulthood, but not with inflammatory variables.
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- 2014
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18. [I Brazilian Guidelines for cardiovascular prevention].
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Simão AF, Precoma DB, Andrade JP, Correa FH, Saraiva JF, Oliveira GM, Murro AL, Campos A, Alessi A, Avezum A Jr, Achutti AC, Miguel AC, Sousa AC, Lotemberg AM, Lins AP, Falud AA, Brandão AA, Sanjuliani AF, Sbissa AS, Alencar FA, Herdy AH, Polanczyk CA, Lantieri CJ, Machado CA, Scherr C, Stoll C, Amodeo C, Araújo CG, Saraiva D, Moriguchi EH, Mesquita ET, Fonseca FA, Campos GP, Soares GP, Feitosa GS, Xavier HT, Castro I, Giuliano IC, Rivera IV, Guimaraes IC, Issa JS, Souza JR, Faria NJ, Cunha LB, Pellanda LC, Bortolotto LA, Bertolami MC, Miname MH, Gomes MA, Tambascia M, Malachias MV, Silva MA, Izar MC, Magalhães ME, Bacellar MS, Milani M, Wajngarten M, Ghorayeb N, Coelho OR, Villela PB, Jardim PC, Santos Filho RD, Stein R, Cassani RS, D'Avila RI, Ferreira RM, Barbosa RB, Povoa RM, Kaiser SE, Ismael SC, Carvalho T, Giraldez VZ, Coutinho W, and Souza WK
- Subjects
- Aspirin therapeutic use, Brazil, Evidence-Based Medicine, Female, Humans, Hypertension prevention & control, Meta-Analysis as Topic, Quality of Life, Randomized Controlled Trials as Topic, Risk Assessment, Risk Factors, Treatment Outcome, Cardiovascular Diseases prevention & control, Health Promotion
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- 2013
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19. Renal sympathetic denervation for resistant hypertension treatment : current perspectives.
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Brandão AA, Campana EM, Magalhães ME, and Ferreira E
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- Blood Pressure, Catheter Ablation methods, Humans, Sympathectomy trends, Sympathetic Nervous System physiopathology, Sympathetic Nervous System surgery, Time Factors, Treatment Outcome, Coronary Vasospasm surgery, Hypertension surgery, Kidney innervation, Sympathectomy methods
- Abstract
The authors review the concept of resistant hypertension and the involvement of the sympathetic nervous system in hypertension as a rational basis for the technique of renal sympathetic denervation (RSD) performed percutaneously. This revision is the result of an active search for scientific articles with the term "renal denervation" in the Medline and PubMed databases. The techniques and devices used in the procedure are presented, as well as clinical outcomes at six, 12 and 24 months after the intervention with the Symplicity catheter. Significant decreases and progressively higher reductions of systolic and diastolic blood pressure were observed after RSD. The complication rate was minimal. New devices for RSD and its ongoing clinical studies are cited. In conclusion, the RSD presents itself as an effective and safe approach to resistant hypertension. Results from ongoing studies and longer follow-up of these patients are expected to confirm the initial results and put into perspective the expansion of the procedure use in hypertension approach.
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- 2013
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20. Pulse wave velocity, blood pressure and adipocytokines in young adults: the Rio de Janeiro study.
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Pizzi OL, Brandão AA, Pozzan R, Magalhães ME, Campana EM, Fonseca FL, Freitas EV, and Brandão AP
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- Adult, Anthropometry, Blood Pressure Determination, Brazil, Cardiovascular Diseases physiopathology, Female, Humans, Male, Multivariate Analysis, Risk Factors, Sex Distribution, Adipokines blood, Blood Pressure physiology, Pulse Wave Analysis
- Abstract
Background: Data on noninvasive vascular assessment and their association with cardiovascular risk variables are scarce in young individuals., Objective: To evaluate the association between pulse wave velocity and blood pressure, anthropometric and metabolic variables, including adipocytokines, in young adults., Methods: A total of 96 individuals aged 26 to 35 years (mean 30.09 ± 1.92; 51 males) were assessed in the Rio de Janeiro study. Pulse wave velocity (Complior method), blood pressure, body mass index, glucose, lipid profile, leptin, insulin, adiponectin and insulin resistance index (HOMA-IR) were analyzed. Subjects were stratified into three groups according to the PWV tertile for each gender., Results: The group with the highest pulse wave velocity (PWV) tertile showed higher mean systolic and diastolic blood pressure, mean blood pressure, body mass index, insulin, and HOMA-IR, as well as lower mean adiponectin; higher prevalence of diabetes mellitus/glucose intolerance and hyperinsulinemia. There was a significant positive correlation of PWV with systolic blood pressure, diastolic blood pressure, pulse pressure and mean blood pressure, body mass index, and LDL-cholesterol, and a negative correlation with HDL-cholesterol and adiponectin. In the multiple regression model, after adjustment of HDL-cholesterol, LDL-cholesterol and adiponectin for gender, age, body mass index and mean blood pressure, only the male gender and mean blood pressure remained significantly correlated with PWV., Conclusion: PWV in young adults showed a significant association with cardiovascular risk variables, especially in the male gender, and mean blood pressure as important determinant variables. The findings suggest that PWV measurement can be useful for the identification of vascular impairment in this age group.
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- 2013
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21. Pulse wave velocity in young adults: study of Rio de Janeiro.
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Pizzi OL, Brandão AA, Pozzan R, Magalhães ME, Freitas EV, and Brandão AP
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- Adult, Age Factors, Anthropometry, Blood Flow Velocity physiology, Brazil, Cardiovascular Diseases metabolism, Female, Hemodynamics physiology, Humans, Hypertension physiopathology, Longitudinal Studies, Male, Pulse, Risk Factors, Statistics, Nonparametric, Young Adult, Blood Pressure physiology, Cardiovascular Diseases physiopathology
- Abstract
Background: Pulse wave velocity (PWV) can be a marker of cardiovascular impairment, but there are few studies in young adults., Objective: To evaluate the association between blood pressure (BP), current anthropometric and metabolic variables and those obtained 13 years earlier, in childhood and adolescence, with PWV., Methods: Sixty individuals were followed longitudinally and split into two groups according to the percentile of blood pressure (BP) obtained 13 years earlier: Group 1 (G1): BP percentile < 50 (n = 25, 11M, 26.4 years old) and Group 2 (G2): BP > 95 percentile (n = 35, 19M, 25.4 years old). The individuals underwent clinical evaluation, laboratory analysis and measurements of PWV through the Complior method., Results: G1 showed higher mean age; G2 showed greater mean weight, systolic blood pressure (SBP), diastolic blood pressure (DBP), mean arterial pressure (MAP), PWV and blood glucose, and lower mean HDL-cholesterol. SBP, MAP and heart rate (HR) obtained during childhood and adolescence significantly correlated with PWV. Current weight, height, waist-hip ratio, SBP, DBP, pulse pressure (PP), MAP and creatinine presented a positive and significant correlation with PWV. A comparison of the average PWV adjusted for SBP, DBP, SBP and DBP, MAP and PP showed no statistically significant difference between groups., Conclusion: The percentile of BP in childhood/adolescence related to arterial distensibility assessed by PWV 13 years later. Changes in PWV can be identified in young individuals suggesting that early vascular impairment may be present in this age group, also related to blood pressure, anthropometric and metabolic variables.
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- 2011
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22. Association between uric acid and cardiovascular risk variables in a non-hospitalized population.
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Barbosa MC, Brandão AA, Pozzan R, Magalhães ME, Campana EM, Fonseca FL, Pizzi OL, Freitas EV, and Brandão AP
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- Adult, Age Factors, Biomarkers analysis, Blood Pressure, Body Mass Index, Brazil, Cardiovascular Diseases metabolism, Cross-Sectional Studies, Epidemiologic Methods, Female, Humans, Insulin blood, Kidney physiology, Male, Middle Aged, Risk Factors, Waist Circumference, Young Adult, Cardiovascular Diseases etiology, Metabolic Syndrome complications, Uric Acid analysis
- Abstract
Background: The association between uric acid (UA) and cardiovascular risk variables remains a controversial issue in epidemiological studies., Objective: To evaluate the association between UA, blood pressure (BP), anthropometric indices and metabolic variables in a non-hospitalized population stratified by UA quintiles., Methods: A cross-sectional observational study evaluated 756 individuals (369 males), aged 50.3 ± 16.12 years, divided in UA quintiles. BP, body mass index (BMI), abdominal circumference (AC), UA, glucose, insulin, HOMA-IR, total cholesterol (TC), LDL-c, HDL-c, triglycerides (TG) and creatinine (C) levels were obtained. The estimated glomerular filtration rate (eGFR) was calculated and arterial hypertension (AH) was considered when BP > 140x90 mmHg, overweight/obesity (OW/O) was considered when BMI > 25 kg/m² and metabolic syndrome (MS) was established according to the I Brazilian Guideline of MS., Results: 1) There was no difference between the groups regarding the distribution by sex and age range; 2) The highest UA quintiles presented higher mean age (p < 0.01), BMI, AC (p < 0.01), SBP, DBP (p < 0.001), TC, LDL-c, TG (p < 0.01), C and eGFR (p < 0.001) and lower mean HDL-c (p < 0.001); 3) The group with the highest UA quintile showed higher prevalence of AH, OW/O and MS (p < 0.001); 4) Higher percentages of the lowest quintiles of insulin (p < 0.02) and HOMA-IR (p < 0.01) were observed with the lowest quintiles of UA; 5) A logistic regression analysis showed that UA and the variables that compose MS were associated with the occurrence of MS (p < 0.01)., Conclusion: Higher quintiles of uric acid were associated with a worse cardiovascular risk profile and a worse kidney function profile in the non-hospitalized population sample studied.
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- 2011
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23. [Overweight and cardiovascular risk among young adults followed-up for 17 years: the Rio de Janeiro study, Brazil].
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Fonseca FL, Brandão AA, Pozzan R, Campana EM, Pizzi OL, Magalhães ME, Freitas EV, and Brandão AP
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- Adolescent, Adult, Age Factors, Brazil epidemiology, Child, Female, Follow-Up Studies, Humans, Male, Metabolic Syndrome epidemiology, Metabolic Syndrome pathology, Overweight epidemiology, Risk Factors, Young Adult, Blood Pressure physiology, Body Mass Index, Cardiovascular Diseases etiology, Overweight pathology
- Abstract
Background: The adoption of primary prevention measures among young people has a favorable impact on the context of cardiovascular diseases., Objective: To assess blood pressure (BP) and cardiovascular risk variables among young adults stratified according to the body mass index (BMI) behavior obtained along 17 years, since childhood/adolescence (C/A)., Methods: Three assessments were carried out in 115 individuals pertaining to the study cohort of Rio de Janeiro, Brazil. A1: 12.97 +/- 1.48 years old; A2: 21.90 +/- 1.71 years old; A3: 30.65 +/- 2.00 years olds and divided into three groups according to BMI in the three assessments: Group N (always normal BMI; n=46), Group L (varying BMI; n=49) and Group O/O (always increased BMI; n=20). In A1, A2 and A3, BP and BMI were obtained. In A2 and A3, glucose (G) and lipidic profile were dosed. Also in A2, insulin (INS) was dosed and HOMA-IR was calculated. In A3, the measurement of waist circumference (WC), abdomen/hip relation (AHR) and body fat percentage (%BF) were added., Results: 1) Group O/O presented higher mean values of increased BP (p<0.0001) at the three assessments; 2) In A3, Group O/O showed higher mean values for WC, AHR and %BF and prevalence of increased WC and metabolic syndrome (MS) (p<0.0001); 3) higher mean values were observed for INS, HOMA-IR, LDL-c in A2, and G, cholesterol, LDL-c and triglycerides in A3 for Group O/O (p<0.05); 4) masculine sex and O/O at A1 determined higher risk for MS occurrence in adult age., Conclusion: The presence of O/O since C/A was associated with higher BP values, anthropometric indexes and higher prevalence of MS in the young adult phase.
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- 2010
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24. Blood pressure in young individuals as a cardiovascular risk marker. The Rio de Janeiro study.
- Author
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Campana EM, Brandão AA, Pozzan R, França Mde F, Fonseca FL, Pizzi OL, Magalhães ME, Freitas EV, and Brandão AP
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- Adolescent, Adult, Biomarkers analysis, Body Mass Index, Brazil epidemiology, Child, Epidemiologic Methods, Female, Humans, Hypertension epidemiology, Male, Metabolic Syndrome epidemiology, Obesity epidemiology, Risk Factors, Young Adult, Blood Pressure physiology, Cardiovascular Diseases etiology, Waist Circumference physiology
- Abstract
Background: The study of the cardiovascular risk variables in young populations is fundamental to establish primary prevention strategies., Objective: To evaluate the blood pressure (BP), anthropometric and metabolic profile in young individuals from The Rio de Janeiro Study, followed by 17 years., Methods: A total of 115 individuals (64 males) were evaluated at three different moments (follow-up: 212.23+/-16.0 months): A1 (12.97+/-1.48 years), A2 (21.90+/-1.71 years) and A3 (30.65+/-2.00 years) and divided in two groups: NG (n=84) with at least two normal BP measurements at the three assessments; HG (n=31) with at least two abnormal BP measurements at the three assessments. BP and body mass index (BMI) were obtained at the three assessments. Levels of glucose, triglycerides, total cholesterol and fractions were obtained at A2 and A3. Abdominal circumference (AC) was obtained only at A3., Results: 1) The means of BP, BMI and AC (p<0.0001) as well as the prevalence of systemic arterial hypertension (SAH) and overweight/obesity (O/O) (p<0.003) were higher in the HG at the three assessments; 2) The means of LDL-c and glycemia (p<0.05) at A2 and the prevalence of metabolic syndrome (MS) at A3 were higher in the HG; 3) the association SAH+O/O was more prevalent in the HG, whereas the association NBP+NBMI was more prevalent in the NG (p<0.0001) at the three assessments; 4) SAH at A1 (RR=5.20 = 5.20; p<0.0007), male gender (RR=5.26 = 5.26; p<0.0019) and OO at A1 (RR=3.40 = 3.40; p<0.0278) determined an increased risk for AH at the young adult life (A3)., Conclusion: After 17 years of follow-up, the BP of young individuals showed a significant association with the cardiovascular risk variables and the occurrence of MS at the young adult life.
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- 2009
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25. Importance of HDL-c for the occurrence of cardiovascular disease in the elderly.
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Freitas EV, Brandão AA, Pozzan R, Magalhães ME, Fonseca F, Pizzi O, Campana E, and Brandão AP
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- Age Factors, Aged, Biomarkers blood, Brazil epidemiology, Cardiovascular Diseases blood, Cardiovascular Diseases etiology, Female, Follow-Up Studies, Humans, Logistic Models, Male, Reference Values, Risk Factors, Cardiovascular Diseases epidemiology, Cholesterol, HDL blood
- Abstract
Background: Studies on the impact of HDL-c and the occurrence of cardiovascular disease (CV) in the elderly are scarce., Objective: To evaluate the clinical and laboratory variables and the occurrence of CV events in elderly patients stratified according to the behavior of HDL-c during an eight-year follow up., Methods: We evaluated 81 elderly patients, mean age of 68.51 +/- 6.32 years (38.2% male), in two stages (A1 and A2), with a minimum interval of five years. The subjects were divided into 3 groups according to HDL-c levels: normal HDL-c in both assessments (NG) (n = 31), low HDL-c in both assessments (LG) (n = 21) and variable HDL-c in A1 and A2 (VG) (n = 29). Main CV events were recorded: coronary heart disease (angina, myocardial infarction, percutaneous / surgical myocardial revascularization), stroke, transient ischemic attack, carotid disease, dementia and heart failure., Results: The groups did not differ in gender and age in A1 and A2. Mean triglyceride levels were lower in the NG in A1 (p = 0.027) and A2 (p = 0.016) than in the LG. The distribution of CV events was as follows: 13 events in the NG (41.9%), 16 (76.2%) in the LG, and 12 (41.4%) in the VG (chi2 = 7.149, p = 0.024). The logistic regression analysis showed that the older the patient (OR = 1.187, p = 0.0230) and the lower the HDL-c (OR = 0.9372, p = 0.0102), the greater the occurrence of events CV., Conclusion: Permanently low HDL-c during eight years of monitoring is a risk factor for the development of CV events in the elderly.
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- 2009
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26. [IV Brazilian Guideline for Dyslipidemia and Atherosclerosis prevention: Department of Atherosclerosis of Brazilian Society of Cardiology].
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Sposito AC, Caramelli B, Fonseca FA, Bertolami MC, Afiune Neto A, Souza AD, Lottenberg AM, Chacra AP, Faludi AA, Loures-Vale AA, Carvalho AC, Duncan B, Gelonese B, Polanczyk C, Rodrigues Sobrinho CR, Scherr C, Karla C, Armaganijan D, Moriguchi E, Saraiva F, Pichetti G, Xavier HT, Chaves H, Borges JL, Diament J, Guimarães JI, Nicolau JC, dos Santos JE, de Lima JJ, Vieira JL, Novazzi JP, Faria Neto JR, Torres KP, Pinto Lde A, Bricarello L, Bodanese LC, Introcaso L, Malachias MV, Izar MC, Magalhães ME, Schmidt MI, Scartezini M, Nobre M, Foppa M, Forti NA, Berwanger O, Gebara OC, Coelho OR, Maranhão RC, dos Santos Filho RD, Costa RP, Barreto S, Kaiser S, Ihara S, Carvalho Td, Martinez TL, Relvas WG, and Salgado W
- Subjects
- Adult, Age Distribution, Aged, Cholesterol blood, Clofibric Acid therapeutic use, Coronary Artery Disease etiology, Coronary Artery Disease physiopathology, Diet, Female, Humans, Hyperlipidemias complications, Hyperlipidemias physiopathology, Hypolipidemic Agents therapeutic use, Lipid Metabolism drug effects, Male, Metabolic Syndrome complications, Middle Aged, Naphthalenes therapeutic use, Risk Factors, Sex Distribution, Smoking adverse effects, Triglycerides blood, Coronary Artery Disease prevention & control, Hyperlipidemias therapy, Lipid Metabolism physiology
- Published
- 2007
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27. [Mechanisms of rhabdomyolysis with statins].
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Magalhães ME
- Subjects
- Apoptosis drug effects, Cardiovascular Diseases prevention & control, Creatine Kinase analysis, Cyclosporine adverse effects, Cyclosporine metabolism, Drug Interactions, Humans, Hydroxymethylglutaryl-CoA Reductase Inhibitors metabolism, Rhabdomyolysis metabolism, Hydroxymethylglutaryl-CoA Reductase Inhibitors adverse effects, Rhabdomyolysis chemically induced
- Abstract
Rhabdomyolysis is uncommon, but is a serious side-effect observed following statin use. The occurrence of fatal rhabdomyolysis reported in the USA since the introduction of statins in the market in the late eighties was very rare (0.15 cases per million patients treated per year). However, myopathy, defined as muscle symptoms associated with CK elevation is much more common (1-5%). Mechanism of statin-induced myopathy is not fully understood. Several hypotheses have been proposed: decreased cellular levels of isoprenoids and ubiquinone, increased rates of apoptosis, changes in chloride channels impairing cell-membrane hyperpolarization, and changes in the cell-membrane permeability. Interactions with other drugs, and preexisting metabolic alterations may predispose to myopathy.
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- 2005
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28. Marfan's syndrome: early and severe form in siblings.
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Leite Mde F, Aoun NB, Borges MS, Magalhães ME, and Christiani LA
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- Aortic Aneurysm drug therapy, Aortic Aneurysm surgery, Child, Fatal Outcome, Female, Humans, Male, Marfan Syndrome drug therapy, Marfan Syndrome surgery, Aortic Aneurysm diagnosis, Marfan Syndrome diagnosis, Siblings
- Abstract
Marfan's syndrome is an inherited disorder of the connective tissue. Cardiologic manifestations, especially aortic dilation, are important causes of morbidity and mortality in the clinical course of the disease in adults and teenagers. In children, the presence of aortic aneurysm and its dissection or rupture is rare, occurring in patients with genetic mutation of the fibrillin gene but not in those who have the familial form of the disease. We describe here 2 patients, from the same family (siblings), diagnosed with gigantic aortic aneurysm early in infancy, one of them successfully undergoing surgery.
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- 2003
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29. National alert campaign about increased cholesterol: determination of cholesterol levels in 81,262 Brazilians.
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Martinez TL, Santos RD, Armaganijan D, Torres KP, Loures-Vale A, Magalhães ME, Lima JC, Moriguchi E, Amodeo C, and Ortiz J
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- Adolescent, Adult, Age Distribution, Brazil epidemiology, Coronary Artery Disease epidemiology, Female, Humans, Male, Mass Screening, Middle Aged, Prevalence, Risk Factors, Sex Distribution, Cholesterol blood, Coronary Artery Disease prevention & control, Health Promotion
- Abstract
Objective: To determine the levels of total cholesterol in a significant sample of the Brazilian population., Methods: Blood cholesterol was determined in 81.262 individuals > 18 years old (51% male, 44.7 +/- 15.7 years), using Accutrend equipment, in the cities São Paulo, Campinas, Campos do Jordão, São José dos Campos, Santos, Santo André , Ribeirão Preto, Porto Alegre, Rio de Janeiro, Belo Horizonte, Curitiba, Brasília, Salvador and documented in the presence of other risk factors (RF) for coronary artery disease (CAD) (systemic hypertension, CAD in the family, smoking, and diabetes). Participants were classified according to sex, age, and the presence or absence of RF, respectively, as 0 RF, 1 RF and > 2 RF. The percentage of individuals with cholesterol > 200 mg/dL and > 240 mg/dL was evaluated., Results: The prevalence of individuals with 0, 1, and > 2 risk factors was 30% (n = 24,589), 36% (n =29,324), and 34% (n = 27,349) respectively, (P=0.657), and the mean total cholesterol of the population was 199.0 +/- 35.0 mg/dL. Cholesterol levels above 200 and 240 mg/dL were found, respectively, in 40% (n = 32,515) and 13% (10.942) of individuals. The greater the number of risk factors the higher the levels of cholesterol (P<0.0001) and the greater the proportion of individuals with cholesterol > 200 mg/dL (P=0.032). No difference existed in the proportion of individuals with cholesterol > 240 mg/dL (P=0.11)., Conclusion: A great percentage of individuals with cholesterol levels above those recommended to prevent coronary artery disease was found.
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- 2003
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30. [Coronary fistula associated with severe pulmonary stenosis and cianosis].
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Aoun NB, Christiani LA, Magalhães ME, Succi E, Meier MA, and Albanesi Filho FM
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- Adolescent, Aortography, Coronary Disease diagnostic imaging, Coronary Disease surgery, Cyanosis diagnostic imaging, Cyanosis surgery, Echocardiography, Electrocardiography, Fistula diagnostic imaging, Fistula surgery, Humans, Male, Prognosis, Pulmonary Valve Stenosis diagnostic imaging, Pulmonary Valve Stenosis surgery, Coronary Disease complications, Cyanosis complications, Fistula complications, Pulmonary Valve Stenosis complications
- Abstract
A 17 year old male adolescent was admitted to the hospital due to dyspnea and cyanosis, since the age of 6 years. On physical examination, it was found a continuous murmur over the left esternal border at the 4th and 5th intercostal spaces suggesting a coronary fistula. A color-echocardiogram associated with an angiography confirmed the diagnosis of coronary fistula and severe pulmonary stenosis. The functional consequence was a right to left shunt with cyanosis. A surgical repair was performed with closure of the fistula at the point where it drained into the right ventricle plus a pulmonary commissurotomy. The patient had an uneventful recovery.
- Published
- 1996
31. [Acute myocardial infarct in a patient with dextrorotation].
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Albanesi Filho FM, Ginefra P, Magalhães ME, and de Albuquerque DC
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- Aged, Clinical Enzyme Tests, Dextrocardia complications, Electrocardiography, Female, Humans, Lung diagnostic imaging, Myocardial Infarction etiology, Pulmonary Edema diagnosis, Pulmonary Edema etiology, Radiography, Dextrocardia diagnosis, Myocardial Infarction diagnosis
- Abstract
A 79-year-old woman developed myocardial infarction associated with acute pulmonary edema. The electrocardiogram recorded QRS complex of QS type in V1 with upper ST extending from V3R to V6R. The polarity of the P wave in bipolar leads was positive, so compatible with dextrorotation complicated by myocardial infarction. The dextrorotation was confirmed with chest roentgenogram and echocardiogram.
- Published
- 1992
32. [Prolonged administration of propafenon in the treatment of premature ventricular beats. Evaluation by dynamic (Holter) electrocardiography].
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Rocha PJ, Albuquerque DC, Benchimol CB, Albanesi Filho FM, Magalhães ME, Schlesinger P, and Benchimol AB
- Subjects
- Blood Pressure drug effects, Heart Rate drug effects, Humans, Propafenone, Propiophenones administration & dosage, Arrhythmias, Cardiac drug therapy, Electrocardiography methods, Propiophenones therapeutic use
- Published
- 1980
Catalog
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