33 results on '"Kallenberg, Cees G. M."'
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2. Granulomatosis with Polyangiitis (Wegenerʼs): An alternative name for Wegenerʼs Granulomatosis
3. Humoral responses after influenza vaccination are severely reduced in patients with rheumatoid arthritis treated with rituximab
4. Urinary CD4+ effector memory T cells reflect renal disease activity in antineutrophil cytoplasmic antibody-associated vasculitis
5. Studies of Cell-Mediated Immune Responses to Influenza Vaccination in Systemic Lupus Erythematosus
6. Cellular immunity in Wegenerʼs granulomatosis: Characterizing T lymphocytes
7. Systems Biology Analysis of Sjögrenʼs Syndrome and Mucosa-Associated Lymphoid Tissue Lymphoma in Parotid Glands
8. Skewed distribution of Th17 lymphocytes in patients with Wegenerʼs granulomatosis in remission
9. Opsonization of Late Apoptotic Cells by Systemic Lupus Erythematosus Autoantibodies Inhibits Their Uptake Via an Fcγ Receptor–Dependent Mechanism
10. Functional Defect of Circulating Regulatory CD4+ T Cells in Patients With Wegenerʼs Granulomatosis in Remission
11. Do apoptotic cells accumulate in the epidermis of patients with cutaneous lupus erythematosus after ultraviolet irradiation? Comment on the article by Kuhn et al
12. Abundance of the Long Pentraxin PTX3 at Sites of Leukocytoclastic Lesions in Patients With Small-Vessel Vasculitis
13. Churg-Strauss Syndrome: Just One Disease Entity?
14. Smoking and increased apoptosis in patients with systemic lupus erythematosus: comment on the article by Costenbader et al
15. The Prototypic Tissue Pentraxin PTX3, in Contrast to the Short Pentraxin Serum Amyloid P, Inhibits Phagocytosis of Late Apoptotic Neutrophils by Macrophages
16. Role of the Fcγ Receptor IIA Polymorphism in the Antiphospholipid Syndrome: An International Meta-Analysis
17. Serum Amyloid P Component Binds to Late Apoptotic Cells and Mediates Their Uptake by Monocyte-Derived Macrophages
18. Fcγ Receptor Gene Polymorphisms in Japanese Patients With Systemic Lupus Erythematosus: Contribution of FCGR2B to Genetic Susceptibility
19. The R-H polymorphism of Fcγ receptor IIa as a risk factor for systemic lupus erythematosus is independent of single-nucleotide polymorphisms in the interleukin-10 gene promoter
20. Fcγ RECEPTOR POLYMORPHISMS IN SYSTEMIC LUPUS ERYTHEMATOSUS: Association with Disease and In Vivo Clearance of Immune Complexes
21. Fcγ RECEPTOR POLYMORPHISMS IN WEGENER'S GRANULOMATOSIS: Risk Factors for Disease Relapse
22. THE FcγRIIIA-158F ALLELE IS A RISK FACTOR FOR SYSTEMIC LUPUS ERYTHEMATOSUS
23. Skewed Distribution of IgG Fc Receptor IIa (CD32) Polymorphism is Associated with Renal Disease in Systemic Lupus Erythematosus Patients
24. Reply
25. Patients' perceptions of the effects of systemic lupus erythematosus on health, function, income, and interpersonal relationships: A comparison with Wegener's granulomatosis
26. TFc?RIIIA-158F allele is a risk factor for systemic lupus erythematosus
27. T FcRIIIA158F allele is a risk factor for systemic lupus erythematosus
28. Serum levels of soluble adhesion molecules intercellular adhesion molecule 1, vascular cell adhesion molecule 1, and E‐selectin in patients with wegener's granulomatosis. Relationship to disease activity and relevance during followup
29. Relationship between antineutrophil cytoplasmic antibody levels and disease activity in wegener's granulomatosis: comment on article by kerr et al
30. Nomenclature of Systemic Vasculitides
31. Systems Biology Analysis of Sjögren's Syndrome and Mucosa-Associated Lymphoid Tissue Lymphoma in Parotid Glands.
32. Fcγ receptor gene polymorphisms in Japanese patients with systemic lupus erythematosus: Contribution of <TOGGLE>FCGR2B</TOGGLE> to genetic susceptibility
33. autoantibodies during captopril treatment
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