23 results on '"Clarke, David A."'
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2. Cystic fibrosis transmembrane conductance regulator has an altered structure when its maturation is inhibited
3. Extended circular dichroism measurements using synchrotron radiation show that the assembly of clathrin coats requires no change in secondary structure
4. The glycosylation and orientation in the membrane of the third cytoplasmic loop of human P-glycoprotein is affected by mutations and substrates
5. Disease-associated mutations in cytoplasmic loops 1 and 2 of cystic fibrosis transmembrane conductance regulator impede processing or opening of the channel
6. Mutations to amino acids located in predicted transmembrane segment 6 (TM6) modulate the activity and substrate specificity of human P-glycoprotein
7. Molecular cloning and tissue distribution of alternatively spliced mRNAs encoding possible mammalian homologues of the yeast secretory pathway calcium pump
8. The V510D suppressor mutation stabilizes -F508-CFTR at the cell surface
9. Subdivision of the bacterioferritin comigratory protein family of bacterial peroxiredoxins based on catalytic activity
10. Correctors enhance maturation of [Delta]F508 CFTR by promoting interactions between the two halves of the molecule
11. Interrogating the molecular details of the peroxiredoxin activity of the Escherichia coli bacterioferritin comigratory protein using high-resolution mass spectrometry
12. Nucleotide binding, ATP hydrolysis, and mutation of the catalytic carboxylates of human P-glycoprotein cause distinct conformational changes in the transmembrane segment
13. ATP hydrolysis promotes interactions between the extracellular ends for transmembrane segments 1 and 11 of human multidrug resistance P-glycoprotein
14. Drugs Modulate Interactions between the First Nucleotide-Binding Domain and the Fourth Cytoplasmic Loop of Human P-Glycoprotein
15. Drug Rescue Distinguishes between Different Structural Models of Human P-Glycoprotein
16. Bithiazole Correctors Rescue CFTR Mutants by Two Different Mechanisms
17. A Salt Bridge in Intracellular Loop 2 Is Essential for Folding of Human P-Glycoprotein
18. Benzbromarone Stabilizes ΔF508 CFTR at the Cell Surface
19. The W232R Suppressor Mutation Promotes Maturation of a Truncation Mutant Lacking both Nucleotide-Binding Domains and Restores Interdomain Assembly and Activity of P-glycoprotein Processing Mutants
20. The V510D Suppressor Mutation Stabilizes ΔF508-CFTR at the Cell Surface
21. Correctors Enhance Maturation of ΔF508 CFTR by Promoting Interactions between the Two Halves of the Molecule
22. Nucleotide Binding, ATP Hydrolysis, and Mutation of the Catalytic Carboxylates of Human P-Glycoprotein Cause Distinct Conformational Changes in the Transmembrane Segments
23. ATP Hydrolysis Promotes Interactions between the Extracellular Ends of Transmembrane Segments 1 and 11 of Human Multidrug Resistance P-Glycoprotein
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