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Your search keyword '"Teixeira, Mauro M."' showing total 8 results
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8 results on '"Teixeira, Mauro M."'

1. Plasmin and plasminogen induce macrophage reprogramming and regulate key steps of inflammation resolution via annexin A1

Author

Sugimoto, Michelle A., Ribeiro, Ana Luíza C., Costa, Bruno R.C., Vago, Juliana P., Lima, Kátia M., Carneiro, Fernanda S., Ortiz, Mylena Maira O., Lima, Graziele Letícia N., Carmo, Aline A.F., Rocha, Renata M., Perez, Denise A., Reis, Alessandra C., Pinho, Vanessa, Miles, Lindsey A., Garcia, Cristiana C., Teixeira, Mauro M., and Sousa, Lirlândia P.

Published
2017
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4. The Role of Plasmin/Plasminogen in Experimental Polymicrobial Sepsis

Author

Vago, Juliana, primary, Brito, Larissa Froede, additional, Teixeira, Lívia C.R., additional, Moreira, Izabela Z., additional, Miranda, Thaís C., additional, Melo, Eliza M., additional, Bruno, Alexandre S., additional, Sugimoto, Michelle A., additional, Ferreira, Raphael G., additional, Alves-Filho, Jose C., additional, Teixeira, Mauro M., additional, Parmer, Robert J, additional, Miles, Lindsey A, additional, and Sousa, Lirlandia P., additional

Published
2020
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7. Control of murine Ly6Chighmonocyte traffic and immunosuppressive activities by atypical chemokine receptor D6

Author

Savino, Benedetta, Castor, Marina G., Caronni, Nicoletta, Sarukhan, Adelaida, Anselmo, Achille, Buracchi, Chiara, Benvenuti, Federica, Pinho, Vanessa, Teixeira, Mauro M., Mantovani, Alberto, Locati, Massimo, and Bonecchi, Raffaella

Abstract

The atypical chemokine receptor D6 is a decoy and scavenger receptor for most inflammatory CC chemokines and prevents the development of exacerbated inflammatory reactions. Here we report that mice lacking D6 expression in the nonhematopoietic compartment have a selective increase in the number of Ly6Chighmonocytes in the circulation and in secondary lymphoid tissues. Under inflammatory conditions, Ly6Chighmonocytes accumulate in increased number in secondary lymphoid organs of D6−/−mice in a CCR2-dependent manner. Ly6Chighmonocytes derived from D6−/−mice have enhanced immunosuppressive activity, inhibit the development of adaptive immune responses, and partially protect mice from the development of GVHD. Thus, control of CCR2 ligands by D6 regulates the traffic of Ly6Chighmonocytes and controls their immunosuppressive potential.

Published
2012
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8. Control of murine Ly6Chigh monocyte traffic and immunosuppressive activities by atypical chemokine receptor D6.

Author

Savino, Benedetta, Castor, Marina G., Caronni, Nicoletta, Sarukhan, Adelaida, Anselmo, Achille, Buracchi, Chiara, Benvenuti, Federica, Pinho, Vanessa, Teixeira, Mauro M., Manfovani, Alberto, Locati, Massimo, and Bonecchi, Raffaella

Subjects
*MONOCYTES, *IMMUNOSUPPRESSIVE agents, *CHEMOKINE receptors, *INFLAMMATION, *LABORATORY mice, *LYMPHOID tissue, *IMMUNE response
Abstract

The atypical chemokine receptor D6 is a decoy and scavenger receptor for most inflammatory CC chemokines and prevents the development of exacerbated inflammatory reactions. Here we report that mice lacking D6 expression in the nonhematopoietic compartment have a selective increase in the number of Ly6Chigh monocytes in the circulation and in secondary lymphoid tissues. Under inflammatory conditions, Ly6Chish monocytes accumulate in increased number in secondary lymphoid organs of D6-/- mice in a CCR2-dependent manner. Ly6Chigh monocytes derived from D6-/- mice have enhanced immunosuppressive activity, inhibit the development of adaptive immune responses, and partially protect mice from the development of GVHD. Thus, control of CCR2 ligands by D6 regulates the traffic of Ly6Chigh monocytes and controls their immunosuppressive potential. [ABSTRACT FROM AUTHOR]

Published
2012
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