105 results on '"Agostinelli A"'
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2. An oestrogen receptor β-selective agonist exerts anti-neoplastic effects in experimental intrahepatic cholangiocarcinoma
3. Clinical implications of novel aspects of biliary pathophysiology
4. Human cholangiocarcinoma development is associated with dysregulation of opioidergic modulation of cholangiocyte growth
5. An immunohistochemical study on lymphoid T-cell subsets and activation state in hepatocellular carcinoma
6. An immunohistochemical study on lymphoid T-cell subsets and activation state in hepatocellular carcinoma
7. Lack of NLRP3-inflammasome leads to gut-liver axis derangement and increases hepatic injury in a mouse model of NAFLD
8. Lack of NLRP3-inflammasome leads to gut-liver axis derangement and increases hepatic injury in a mouse model of NAFLD
9. Nlrp3 inflammasome activation by microbial products leads to Il-18 synthesis and impaired epithelial barrier function in cholangiocytes
10. Nlrp3 inflammasome activation by microbial products leads to Il-18 synthesis and impaired epithelial barrier function in cholangiocytes
11. Selective LXRα intestinal activation reduces hepatic inflammation and fibrosis during the development of chronic liver injury
12. NLRP3 inflammasome increases hepatic fibrosis by inducing inflammatory signals in hepatic stellate cells
13. Gut–liver axis derangement due to lack of inflammasome activity leads to visceral obesity and NASH development
14. Selective LXRα intestinal activation reduces hepatic inflammation and fibrosis during the development of chronic liver injury
15. NLRP3 inflammasome increases hepatic fibrosis by inducing inflammatory signals in hepatic stellate cells
16. Gut–liver axis derangement due to lack of inflammasome activity leads to visceral obesity and NASH development
17. PC.01.2 PDX-1 MRNA EXPRESSION IN ENDOSCOPIC ULTRASOUND-GUIDED FINE NEEDLE CYTOASPIRATE AS A NEW MARKER FOR IMPROVING THE DIAGNOSIS OF PANCREATIC CANCER
18. Gut-stimulated inflammasome activation drives progression of liver injury in NASH
19. Activation of the developmental pathway Ngn-3/miR-7a regulates cholangiocytes proliferation in response to injury
20. Gut-stimulated inflammasome activation drives progression of liver injury in NASH
21. Activation of the developmental pathway Ngn-3/miR-7a regulates cholangiocytes proliferation in response to injury
22. OC2 An estrogen receptor β selective agonist exerts specific pro-apoptotic effects and promotes tumor regression in a rat model of intra-hepatic cholangiocarcinoma
23. P.138 FROM NAFLD TO HCC: THE ROLE OF FIBROSIS AS A COFACTOR IN A NEW MOUSE MODEL
24. T.N.44 SEMAPHORIN7A, A NEW PLAYER IN HEPATIC FIBROSIS: AN IN VITRO AND IN VIVO STUDY
25. SEMAPHORIN 7A IS EXPRESSED IN HEPATIC STELLLATE CELLS AND ENHANCES FIBROSIS IN A TGFβ-MEDIATED MECHANISM
26. c-ABL activity is required for the fibrogenetic behaviour of hepatic stellate cells
27. PC.01.2 PDX-1 MRNA EXPRESSION IN ENDOSCOPIC ULTRASOUND-GUIDED FINE NEEDLE CYTOASPIRATE AS A NEW MARKER FOR IMPROVING THE DIAGNOSIS OF PANCREATIC CANCER
28. OC.03.4 DYSBIOSIS CONTRIBUTES TO LIVER FIBROSIS THROUGH BACTERIAL TRANSLOCATION AND ELEVATED ENDOTOXEMIA BY INCREASING GRAM– ABUNDANCE
29. OC-08 Dysbiosis contributes to liver fibrosis through bacterial translocation and elevated endotoxemia by increasing Gram– abundance
30. OC.03.4 DYSBIOSIS CONTRIBUTES TO LIVER FIBROSIS THROUGH BACTERIAL TRANSLOCATION AND ELEVATED ENDOTOXEMIA BY INCREASING GRAM– ABUNDANCE
31. OC-08 Dysbiosis contributes to liver fibrosis through bacterial translocation and elevated endotoxemia by increasing Gram– abundance
32. O2 DIETARY HABITS-MODIFIED MICROBIOTA CONTRIBUTES TO LIVER FIBROSIS THROUGH BACTERIAL TRANSLOCATION BY INCREASING GRAM-ABUNDANCE
33. OC.07.2 ROLE OF BACTERIAL TRANSLOCATION IN LIVER FIBROSIS AND HCC DEVELOPMENT
34. OC.05.5 PDX-1/HES-1 INTERACTIONS DETERMINE CHOLANGIOCYTE PROLIFERATIVE RESPONSE TO INJURY: THE SIGNIFICANCE FOR SCLEROSING CHOLANGITIS
35. OC.05.4 IMPAIRED PANCREATIC DUODENAL HOMEOBOX PROTEIN 1 (PDX-1) EXPRESSION PROMOTES DIABETES AND HEPATOCELLULAR CARCINOMA (HCC) DEVELOPMENT IN MICE
36. T-36 Role of bacterial translocation in liver fibrosis and HCC development
37. T-37 Impaired pancreatic duodenal homeobox protein 1 (PDX-1) expression promotes diabetes and hepatocellular carcinoma (HCC) development in mice
38. OC-1 PDX-1/Hes-1 interactions determine cholangiocyte proliferative response to injury: the significance for sclerosing cholangitis
39. P.1.4: AN ESTROGEN RECEPTOR-β SELECTIVE AGONIST EXERTS SPECIFIC PRO-APOPTOTIC EFFECTS AND PROMOTES TUMOR REGRESSION IN A RAT MODEL OF INTRA-HEPATIC CHOLANGIOCARCINOMA
40. P.1.16: THE ANTIFIBROTIC EFFECT OF SORAFENIB IS DUE TO DECREASED HSCS ACTIVATION MEDIATED BY ANGIOGENESIS BLOCKAGE
41. T-27 The antifibrotic effect of sorafenib is due to decreased HSCs activation mediated by angiogenesis blockage
42. O2 DIETARY HABITS-MODIFIED MICROBIOTA CONTRIBUTES TO LIVER FIBROSIS THROUGH BACTERIAL TRANSLOCATION BY INCREASING GRAM-ABUNDANCE
43. OC.05.4 IMPAIRED PANCREATIC DUODENAL HOMEOBOX PROTEIN 1 (PDX-1) EXPRESSION PROMOTES DIABETES AND HEPATOCELLULAR CARCINOMA (HCC) DEVELOPMENT IN MICE
44. OC.07.2 ROLE OF BACTERIAL TRANSLOCATION IN LIVER FIBROSIS AND HCC DEVELOPMENT
45. OC.05.5 PDX-1/HES-1 INTERACTIONS DETERMINE CHOLANGIOCYTE PROLIFERATIVE RESPONSE TO INJURY: THE SIGNIFICANCE FOR SCLEROSING CHOLANGITIS
46. OC-1 PDX-1/Hes-1 interactions determine cholangiocyte proliferative response to injury: the significance for sclerosing cholangitis
47. T-37 Impaired pancreatic duodenal homeobox protein 1 (PDX-1) expression promotes diabetes and hepatocellular carcinoma (HCC) development in mice
48. OC2 An estrogen receptor β selective agonist exerts specific pro-apoptotic effects and promotes tumor regression in a rat model of intra-hepatic cholangiocarcinoma
49. CS.1.5 SEMAPHORIN7A, A NEW PLAYER IN HEPATIC FIBROSIS: AN IN VITRO AND IN VIVO STUDY
50. P.138 FROM NAFLD TO HCC: THE ROLE OF FIBROSIS AS A COFACTOR IN A NEW MOUSE MODEL
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