18 results on '"Lines, Kate E."'
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2. JQ1 treatment significantly reduces POMC expression and ACTH secretion from the corticotrophinoma cell line, AtT20
3. Whole genome sequence analysis establishes correct diagnosis for a syndromic form of hyperuricaemia
4. Identification of a frame-shifting c.348dupC GNAS mutation in a family with Pseudohypoparathyroidism type 1a (PHP1a) by Whole Genome Sequencing
5. Epigenetic inhibitor treatment reduces proliferation via induction of apoptosis in a human typical bronchial carcinoid cell line
6. Utility of Whole Genome Sequencing in diagnosing complex disorders: lesson from renal tubular disorders
7. An epigenetic modifier reduces proliferation in pituitary cells and suppresses calcium-sensing receptor signalling
8. A case of vitamin D-dependent rickets type 2A (VDDR2A), caused by compound-heterozygous mutations in the vitamin D receptor (VDR)
9. Multiple endocrine neoplasia type 1 (MEN1) phenocopy due to a P.Leu380Phe cell division cycle 23 (CDC73) mutation
10. Combination of JQ1, an inhibitor of epigenetic pathways, and everolimus for treatment of pancreatic and bronchial neuroendocrine tumours
11. A time controlled [beta]-cell specific mouse model Men1L/L/RIP2-CreER for pancreatic neuroendocrine tumours (NETs)
12. MicroRNA miR-3156-5p is down-regulated in serum of Multiple Endocrine neoplasia type 1 patients, and regulates expression of mortality factor 4-like protein 2 (MORF4L2)
13. The epigenetic modifying compound, JQ1+, increases apoptosis in pituitary tumours
14. Treatment with the epigenetic modifying compound JQ1+ can significantly reduce the proliferation of pancreatic neuroendocrine tumours in a mouse model of multiple endocrine neoplasia type 1
15. Menin regulates the expression of miR-15a, which is downregulated and inversely correlates with cyclin D1 expression in mouse Men1-associated pituitary tumours
16. Bromodomain inhibitors reduce proliferation and increase apoptosis of human neuroendocrine tumour cells
17. Hypoxic incubation favours the development and growth of primary human parathyroid adenoma cells
18. Genetic background influences tumour phenotype in heterozygous Men1 knockout mice
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