109 results on '"Peng, DunFa"'
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2. Helicobacter pylori–induced RASAL2 Through Activation of Nuclear Factor-κB Promotes Gastric Tumorigenesis via β-catenin Signaling Axis
3. Induction of Fibroblast Growth Factor Receptor 4 by Helicobacter pylori via Signal Transducer and Activator of Transcription 3 With a Feedforward Activation Loop Involving Steroid Receptor Coactivator Signaling in Gastric Cancer
4. Aurora Kinase A Promotes Inflammation and Tumorigenesis in Mice and Human Gastric Neoplasia
5. Tu1164: AURORA KINASE A PROMOTES CANCER CELL SURVIVAL BY ACTIVATING UNFOLDED PROTEIN RESPONSE UNDER ONCOGENIC STRESS IN ESOPHAGEAL ADENOCARCINOMA
6. Tu1121: INDUCTION OF FGFR4 BY H. PYLORI MEDIATES ACTIVATION OF STAT3 THROUGH SRC SIGNALING IN GASTRIC CANCER
7. Sa1173: APE1 REDOX-DEPENDENT ACTIVATION OF YAP IN RESPONSE TO REFLUX CONDITIONS IN ESOPHAGEAL CARCINOGENESIS
8. Su1248 FREQUENT DYSREGULATION OF NEK FAMILY MEMBERS IN ESOPHAGEAL ADENOCARCINOMA
9. Su1244 REFLUX CONDITIONS PROMOTE CHEMORESISTANCE OF ESOPHAGEAL ADENOCARCINOMA BY ACTIVATING APE1-REDOX-SENSITIVE SOX9 SIGNALING
10. Su1188 SMAD3 INHIBITION SENSITIZES ESOPHAGEAL ADENOCARCINOMA CELLS TO OXALIPLATIN
11. 1291 CHRONIC GASTROESOPHAGEAL REFLUX PROMOTES CHEMORESISTANCE IN ESOPHAGEAL ADENOCARCINOMA THROUGH APE1-REDOX-DEPENDENT PRDX2 ACTIVATION AND FERROPTOSIS INHIBITION
12. 1290 FIBROBLAST GROWTH FACTOR RECEPTOR-4 PROMOTES ANTIOXIDANT RESPONSE VIA ACTIVATION OF NUCLEAR FACTOR ERYTHROID 2-RELATED FACTOR-2 IN GASTRIC CANCER
13. 1258 THE ROLE OF WEE1 IN GASTRIC CANCER PROGRESSION AND IMMUNE EVASION: INSIGHTS FROM H. PYLORI INFECTION
14. 333 H. PYLORI INFECTION INDUCES SOX9 THROUGH CAP-DEPENDENT TRANSLATION MEDIATED BY AURKA-EIF4E AXIS
15. Fr154 SMOKING INDUCES WEE1 EXPRESSION PROMOTING CANCER CELL SURVIVAL IN ESOPHAGEAL ADENOCARCINOMA
16. Fr156 APE1 REDOX FUNCTIONS MEDIATE E-CADHERIN CLEAVAGE AND EMT IN RESPONSE TO EXPOSURE TO ACIDIC BILE SALTS IN ESOPHAGEAL ADENOCARCINOMA
17. Tu1287 APE1 INHIBITS SMAD3 DEGRADATION BY BLOCKING ITS INTERACTION WITH ROC1 UBIQUITIN LIGASE COMPLEX IN ESOPHAGEAL ADENOCARCINOMA CELLS
18. Tu1268 REFLUX CONDITIONS-INDUCED E-CADHERIN CLEAVAGE AND EPITHELIAL-TO-MESENCHYMAL TRANSITION IS MEDIATED BY APE1 REDOX FUNCTION IN ESOPHAGEAL ADENOCARCINOMA
19. Mo1262 MULTIVALENT TYROSINE KINASE INHIBITION PROMOTES T CELL RECRUITMENT TO IMMUNE-DESERT GASTRIC CANCERS BY RESTRICTING EPITHELIAL-MESENCHYMAL TRANSITION VIA TUMORINTRINSIC IFN- γ SIGNALING
20. Su1219 TARGETING OVEREXPRESSED NRF2 IN ESOPHAGEAL ADENOCARCINOMA SENSITIZES TUMOR CELLS TO CISPLATIN THROUGH INDUCING FERROPTOSIS AND APOPTOSIS
21. 1268 SMOKING-WEE1-JAK2 AXIS PROMOTES DRUG RESISTANCE IN ESOPHAGEAL ADENOCARCINOMA
22. 933 ACTIVATION OF NOTCH SIGNALING VIA DLL1 IS MEDIATED BY APE1-REDOX-DEPENDENT NF-KB ACTIVATION IN ESOPHAGEAL ADENOCARCINOMA
23. 583 CDK1 BRIDGES NF-KB AND β-CATENIN SIGNALING IN RESPONSE TO H. PYLORI INFECTION IN GASTRIC TUMORIGENESIS
24. 32 HELICOBACTER PYLORI-MEDIATED ACTIVATION OF NF-κB-STAT3 NETWROK IS SUPPRESSED BY TFF1
25. 153 EXPOSURE OF BARRETT'S AND ESOPHAGEAL ADENOCARCINOMA CELLS TO BILE ACIDS PROMOTES E-CADHERIN CLEAVAGE VIA INDUCTION OF APE1-REDOX-MMP14 SIGNALING AXIS
26. Su1165 TARGETING NRF2 USING SPECIFIC INHIBITOR IN ESOPHAGEAL ADENOCARCINOMA
27. Sa1218 ACIDIC BILE SALT MEDIATED INDUCTION AND REGULATION OF NRF2 IS APE1 DEPENDENT IN BARRET AND ESOPHAGEAL ADENOCARCINOMA CELLS.
28. Mo1295 SMOKING PROMOTES CHEMO-RESISTANCE THROUGH INDUCING WEE1 EXPRESSION IN ESOPHAGEAL ADENOCARCINOMA
29. Mo1783 – H. Pylori-Induced Prdx2 Protects Against Oxidative Stress and Promotes Resistance to Cisplatin
30. Su1115 – Activation of Egfr-Dna-Pk Pathway by Igfbp2 Protects Esophageal Adenocarcinoma Cells from Acidic Bile Saltsinduced Dna Damage and Apoptosis
31. Su1063 – Mir-4715-3P Modulates Aurka and Induces Ferroptosis in Upper Gastrointestinal Cancers
32. 282 – Exposure of Barrett's and Esophageal Adenocarcinoma Cells to Bile Acids Promotes Epithelial-To-Mesenchymal Transition Via Induction of Ape1
33. 334 - APE1 Upregulates MMP14 Expression to Promote Invasion of Barrett's Esophagus Cells and Esophageal Adenocarcinoma Cells Through Novel Redox-Sensitive ARF6-Mediated Exocytosis
34. 64 - TFF1 Suppresses IL-6 Mediated STAT3 Activation through Interfering with IL6Rα/GP130 Complex Formation
35. Sa1652 - Role of Nrf2 in Esophageal Premalignant Cells and Malignant Adenocarcinoma Cells: Protects Cells from Bile Salts-Induced Dna Damage
36. NRF2 Protects Barrett’s Esophageal Cells from Bile Salts-Induced Oxidative DNA Damage and Double Strand Breaks
37. Loss of TFF1 Promotes Cell Proliferation and Invasion Through Regulating of MIR-196B-5P in Mouse and Human Gastric Neoplasm
38. Bile Acid-Induced APE-1 Mediates Stat3 Activation in Barrett's and Esophageal Adenocarcinoma Cells
39. A New Function of APE1 in Barrett’s Esophagus and Esophageal Adenocarcinoma: APE1 Upregulates MMP2 and MMP14 to Promote Invasion
40. Tu2064 Glutathione Peroxidase 7 Suppresses Gastric Cancer Cell Growth and Invasion
41. 866 N-MYC Downregulated Gene 4 (NDRG4) Is a Potential Tumor Suppressor Gene in Esophageal Adenocarcinoma
42. Tu1126 Constitutive Overexpression and Activation of NRF2 in Esophageal Adenocarcinomas Counteracts Bile-Induced Oxidative Stress and Promotes Cancer Cell Survival
43. 13 DARPP32: A Bridge Between Pro-Inflammatory Signaling and Angiogenesis in Gastric Cancer
44. Mo1029 - Bile Acid-Induced APE-1 Mediates Stat3 Activation in Barrett's and Esophageal Adenocarcinoma Cells
45. Sa1145 - NRF2 Protects Barrett’s Esophageal Cells from Bile Salts-Induced Oxidative DNA Damage and Double Strand Breaks
46. Sa1153 - A New Function of APE1 in Barrett’s Esophagus and Esophageal Adenocarcinoma: APE1 Upregulates MMP2 and MMP14 to Promote Invasion
47. 192 - Loss of TFF1 Promotes Cell Proliferation and Invasion Through Regulating of MIR-196B-5P in Mouse and Human Gastric Neoplasm
48. 52 TFF1 Suppresses Cell Proliferation Through Regulation of PP2A-AKT-β-Catenin Signaling in Gastric Adenocarcinoma
49. 932 Loss of Glutathione Peroxidase 7 Promotes TNF-α-Induced NF-kB Activation in Barrett's Carcinogenesis
50. Mo1651 TFF1 Acquires Its Tumor Suppressor Functions Through Regulation of P53
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