157 results on '"Shi, Yi"'
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2. Gut Microbiota Alter Visceral Pain Sensation and Inflammation via Modulation of Synthesis of Resolvin D1 in Colonic Tuft Cells.
3. 659: CHRONIC EFFECT OF LINACLOTIDE ON THE COMPOSITION AND FUNCTION OF GUT MICROBIOTA: POTENTIAL MECHANISMS FOR INHIBITION OF VISCERAL PAIN
4. 241: GUT MICROBIOTA REGULATES BIOSYNTHESIS OF RESOLVIN D1 IN THE COLONIC MUCOSA BY ACTIVATING TLR4 IN INTESTINAL TUFT CELLS
5. 411: A LOW FODMAP DIET IMPROVES COLONIC BARRIER FUNCTION AND MAST CELL ACTIVATION IN IBS-D PATIENTS
6. 784 KYNURENIC ACID (KYNA) LEVELS ARE REDUCED IN IBS-D, RESULTING IN VISCERAL HYPERSENSITIVITY: ROLE OF MICROBIAL DYSBIOSIS
7. Gut Microbiota Alter Visceral Pain Sensation and Inflammation via Modulation of Synthesis of Resolvin D1 in Colonic Tuft Cells
8. Retraction notice to “Increased Activation of the TRESK K+ Mediates Vago-Vagal Reflex Malfunction in Diabetic Rats” Gastroenterology 2016;151:910–922.e7
9. 411: A LOW FODMAP DIET IMPROVES COLONIC BARRIER FUNCTION AND MAST CELL ACTIVATION IN IBS-D PATIENTS
10. 241: GUT MICROBIOTA REGULATES BIOSYNTHESIS OF RESOLVIN D1 IN THE COLONIC MUCOSA BY ACTIVATING TLR4 IN INTESTINAL TUFT CELLS
11. 659: CHRONIC EFFECT OF LINACLOTIDE ON THE COMPOSITION AND FUNCTION OF GUT MICROBIOTA: POTENTIAL MECHANISMS FOR INHIBITION OF VISCERAL PAIN
12. Fr429 TLR4 SIGNAL TRANSDUCTION PATHWAY MEDIATES UPREGULATION OF 2-PORE DOMAIN POTASSIUM TRESK CHANNEL AND IS RESPONSIBLE FOR HIGH-FAT DIET-INDUCED VAGAL AFFERENT DYSFUNCTION
13. 377 MAST-CELL ACTIVATION LEADING TO COLONIC BARRIER DYSFUNCTION FOLLOWING HIGH-FODMAP DIET IS MEDIATED VIA LIPOPOLYSACCHARIDE
14. Sa593 FERMENTATION PRODUCT PROPIONIC ACID PRODUCED BY SPECIFIC GUT BACTERIA INDUCES BARRIER DYSFUNCTION AND SUBCLINICAL MUCOSA INFLAMMATION
15. 658 GUT DYSBIOSIS PROMOTES COLONIC M1 MACROPHAGE EXPRESSION AND CONTRIBUTES TO MUCOSA INFLAMMATION: MEDIATION BY LPS ACTING ON TOLL-LIKE RECEPTOR 4
16. Su1732 OVERABUNDANCE OF HISTAMINE PRODUCING ENTEROBACTERIACEAE CONTRIBUTES TO GASTRIC PAIN IN PATIENTS WITH DIABETIC GASTROPARESIS
17. Sa1094 EVIDENCE OF DUODENAL EPITHELIAL BARRIER IMPAIRMENT (“LEAKY GUT”) AND INCREASED PYROPTOSIS IN PATIENTS WITH FUNCTIONAL DYSPEPSIA ON CONFOCAL LASER ENDOMICROSCOPY AND “EX-VIVO” MUCOSA ANALYSIS
18. 1011 IMPAIRED THERMOGENESIS IN BROWN ADIPOSE TISSUE INDUCED BY DIETARY EMULSIFIERS CARBOXYMETHYL CELLULOSE IS MEDIATED BY MUCOLYTIC BACTERIA AND LPS ENDOTOXEMIA
19. Su1229 PANETH CELL LOSS IS ASSOCIATED WITH MUCOSA DYSBIOSIS, INTESTINAL BARRIER DYSFUNCTION AND MUCOSA INFLAMMATION
20. 132 GUT DYSBIOSIS INDUCED BY CIRCADIAN RHYTHM DISRUPTION PROMOTES VISCERAL HYPERSENSITIVITY: MEDIATION BY DEGRADATION OF COLONIC MUCUS BARRIER AND LPS ENDOTOXEMIA.
21. Fr429 TLR4 SIGNAL TRANSDUCTION PATHWAY MEDIATES UPREGULATION OF 2-PORE DOMAIN POTASSIUM TRESK CHANNEL AND IS RESPONSIBLE FOR HIGH-FAT DIET-INDUCED VAGAL AFFERENT DYSFUNCTION
22. 377 MAST-CELL ACTIVATION LEADING TO COLONIC BARRIER DYSFUNCTION FOLLOWING HIGH-FODMAP DIET IS MEDIATED VIA LIPOPOLYSACCHARIDE
23. Sa593 FERMENTATION PRODUCT PROPIONIC ACID PRODUCED BY SPECIFIC GUT BACTERIA INDUCES BARRIER DYSFUNCTION AND SUBCLINICAL MUCOSA INFLAMMATION
24. Sa1094 EVIDENCE OF DUODENAL EPITHELIAL BARRIER IMPAIRMENT ('LEAKY GUT') AND INCREASED PYROPTOSIS IN PATIENTS WITH FUNCTIONAL DYSPEPSIA ON CONFOCAL LASER ENDOMICROSCOPY AND 'EX-VIVO' MUCOSA ANALYSIS
25. Su1732 OVERABUNDANCE OF HISTAMINE PRODUCING ENTEROBACTERIACEAE CONTRIBUTES TO GASTRIC PAIN IN PATIENTS WITH DIABETIC GASTROPARESIS
26. Su1229 PANETH CELL LOSS IS ASSOCIATED WITH MUCOSA DYSBIOSIS, INTESTINAL BARRIER DYSFUNCTION AND MUCOSA INFLAMMATION
27. 1011 IMPAIRED THERMOGENESIS IN BROWN ADIPOSE TISSUE INDUCED BY DIETARY EMULSIFIERS CARBOXYMETHYL CELLULOSE IS MEDIATED BY MUCOLYTIC BACTERIA AND LPS ENDOTOXEMIA
28. 658 GUT DYSBIOSIS PROMOTES COLONIC M1 MACROPHAGE EXPRESSION AND CONTRIBUTES TO MUCOSA INFLAMMATION: MEDIATION BY LPS ACTING ON TOLL-LIKE RECEPTOR 4
29. 132 GUT DYSBIOSIS INDUCED BY CIRCADIAN RHYTHM DISRUPTION PROMOTES VISCERAL HYPERSENSITIVITY: MEDIATION BY DEGRADATION OF COLONIC MUCUS BARRIER AND LPS ENDOTOXEMIA
30. 874 – Gut Dysbiosis Characterized by Overabundance of Enterobacteriaceae Contributes to Gastric Pain in Diabetic Gastroparesis
31. 264 – Dietary Emulsifiers Reduce Thermogenesis in Brown Adipose Tissue, Leading to Obesity and Metabolic Syndrome
32. 692 – Escherichia Coli Heat-Stable Toxin Impairs Colonic Epithelial Barrier Function Via Cgmp-Induced Oxidative Stress, Apoptosis, and Nf-ΚB Activation
33. Su2004 – Disruption of Circadian Rhythm Reduces Thermogenesis in Brown Adipose Tissue, Leading to Abnormal Weight Gain and Development of Metabolic Syndrome. Mediation by Gut Dysbiosis and Endotoxemia
34. RETRACTED: Increased Activation of the TRESK K+ Mediates Vago-Vagal Reflex Malfunction in Diabetic Rats
35. 874 – Gut Dysbiosis Characterized by Overabundance of Enterobacteriaceae Contributes to Gastric Pain in Diabetic Gastroparesis
36. Su2004 – Disruption of Circadian Rhythm Reduces Thermogenesis in Brown Adipose Tissue, Leading to Abnormal Weight Gain and Development of Metabolic Syndrome. Mediation by Gut Dysbiosis and Endotoxemia
37. 264 – Dietary Emulsifiers Reduce Thermogenesis in Brown Adipose Tissue, Leading to Obesity and Metabolic Syndrome
38. 692 – Escherichia Coli Heat-Stable Toxin Impairs Colonic Epithelial Barrier Function Via Cgmp-Induced Oxidative Stress, Apoptosis, and Nf-ΚB Activation
39. 288 - Mucosal Oxidative Stress and Intestinal Barrier Dysfunction Contribute to Diabetic Diarrhea which is Prevented by Resolvins
40. 830 - Secondary Bile Acids are Responsible for High Fat Diet (HFD)-Induced Upregulation of R-Spondin 3 Which in Turn Mediates HFD-Inducted Intestinal Epithelial Proliferation in Rodents
41. Sa1551 - Gut Dysbiosis Contributes Significantly to Gastric Pain in Diabetic Gastroparesis: Treatment Effects with Rifaximin
42. 265 - Resolvins Prevent Ros-Mediated Mucosa Injury and Protect Gut Barrier Function by Upregulating Hemoxygenase-1 (HO-1)
43. 1099 - Artificial Sweetener (Saccharin) Modulates Visceral Nociception by Lipopolysaccharide-Mediated Gut Barrier Dysfunction and Intestinal Inflammation
44. Increased Activation of the TRESK K
45. 1099 - Artificial Sweetener (Saccharin) Modulates Visceral Nociception by Lipopolysaccharide-Mediated Gut Barrier Dysfunction and Intestinal Inflammation
46. 830 - Secondary Bile Acids are Responsible for High Fat Diet (HFD)-Induced Upregulation of R-Spondin 3 Which in Turn Mediates HFD-Inducted Intestinal Epithelial Proliferation in Rodents
47. Sa1551 - Gut Dysbiosis Contributes Significantly to Gastric Pain in Diabetic Gastroparesis: Treatment Effects with Rifaximin
48. 265 - Resolvins Prevent Ros-Mediated Mucosa Injury and Protect Gut Barrier Function by Upregulating Hemoxygenase-1 (HO-1)
49. 288 - Mucosal Oxidative Stress and Intestinal Barrier Dysfunction Contribute to Diabetic Diarrhea which is Prevented by Resolvins
50. Bile Acids Regulate Intestinal Paneth Cell Functions and Contribute to Gut Dysbiosis Induced by High Fat Feeding
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