26 results on '"Zhu, Ruixin"'
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2. 1094: ARTIFICIAL INTELLIGENCE NON-INVASIVE DIAGNOSTIC TOOLS BASED ON MICROBIAL GENES ACHIEVE SUPERIOR DIAGNOSTIC ACCURACY FOR CROHN'S DISEASE
3. Mo1775 MICROBIAL MULTI-DIMENSIONAL ANALYSES REVEAL BOTH BACTERIAL ABUNDANCE AND INDELS AS PROMISING NON-INVASIVE BIOMARKERS FOR PEDIATRIC CROHN'S DISEASE
4. Sa1866 ENHANCED MICROBIOTA PROFILING IN PATIENTS WITH QUIESCENT CROHN'S DISEASE THROUGH COMPARISON WITH PAIRED HEALTHY FIRST-DEGREE RELATIVES USING FECAL METAGENOMICS AND METABOLOMICS
5. Sa1897 GBP5 INDUCES PRO-INFLAMMATORY INNATE LYMPHOID CELLS BY STIMULATING STAT1 ACTIVITY AND EXPRESSION
6. Mo1905 INTRATUMORAL MICROBIOME AS NOVEL INDICATORS AND THERAPEUTIC TARGETS IN GASTROINTESTINAL CANCER METASTASIS
7. Mo1776 FECAL METAPROTEOMICS-DERIVED HOST AND MICROBIAL PROTEINS SERVE AS SUPERIOR DIAGNOSTIC MARKERS FOR CROHN'S DISEASE
8. EP114 DISTINCT PATTERNS OF HOST-MICROBE CROSSTALK IN THE PROGRESSION OF NONALCOHOLIC FATTY LIVER DISEASE
9. EP166 THE PREDICTIVE ABILITY OF INTRATUMORAL MICROBIOME FOR COLORECTAL CANCER METASTASIS AND ITS ROLE IN TUMOR MICROENVIRONMENT
10. EP165 UNIQUENESS OF HOST-MICROBIOTA MEDIATED HETEROGENEOUS TUMOR MICROENVIRONMENT AMONG CONSENSUS MOLECULAR SUBTYPES IN COLORECTAL CANCER
11. EP22 UNIQUE MICROBIAL ALTERATIONS AND SPECIFIC MULTI-KINGDOM BIOMARKERS OF OVERWEIGHT COLORECTAL CANCER
12. Tu1257 THE IMPACT OF FUT2 DEFICIENCY ON THE GUT MICROBIOME IN INFLAMMATORY BOWEL DISEASES
13. Mo1463 ANALYSIS AND APPLICATION OF KEYSTONE SPECIES IN NONALCOHOLIC FATTY LIVER DISEASE BASED ON CAUSAL INFERENCE AND DYNAMIC INTERVENTION MODELING
14. Tu1252 DAMP MOLECULES NMI AND IFP35 MAY MEDIATE EARLY INFLAMMATORY EVENTS IN THE PATHOGENESIS OF INFLAMMATORY BOWEL DISEASES
15. Su1092 CAUSAL INFERENCE ANALYSIS OF THE TRANSCRIPTOME IDENTIFIED DIFFERENTIAL INNATE IMMUNITY AND INFLAMMATORY PATHWAY BETWEEN CROHN'S DISEASE AND ULCERATIVE COLITIS
16. Su1542 – Keystone Species in the Pathogenesis of Non-Alcoholic Fatty Liver Disease
17. Su1554 – Causal Inference Reveals that Clostridiales Species are Keystone Species in Nafld Pathogenesis
18. 317 – Gut Microbiota Contributes to Promoting Secondary Bile Acid Biosynthesis in Nafld Pathogenesis
19. Suppressed Hepatic Bile Acid Signaling Despite Elevated Production of Primary and Secondary Bile Acids in NAFLD
20. High-Fat Diet Increases Clostridium Clusters XIVa in Obese Rodents: A Meta-Analysis
21. Su1857 Pathological Similarities and Differences Between Crohn's Disease and Ulcerative Colitis Revealed by Systematic Transcriptome Analysis
22. Su1800 Systematic Analysis of NAFLD Transcriptome Revealed Elevated Expression of Alcohol Metabolizing Genes in Mild and Severe NAFLD Livers
23. 745 - Suppressed Hepatic Bile Acid Signaling Despite Elevated Production of Primary and Secondary Bile Acids in NAFLD
24. Tu1935 - High-Fat Diet Increases Clostridium Clusters XIVa in Obese Rodents: A Meta-Analysis
25. Toward the Understanding of Altered Gut Microbiome in Crohn’s Disease: Cause or Consequence
26. Toward the Understanding of Altered Gut Microbiome in Crohn's Disease: Cause or Consequence.
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