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A 18 year girl presented with an asymptomatic solitary nodule on the lateral aspect of the right below. Biopsy proved it to be Pilomatricoma. The lesion was excised. Pilomatricoma is rare, benign tumour commonly occurring on the face and extremities and composed of cells resembling those of hair matrix, which undergo ‘mummification†and may calcify.

Trichoscopy as an investigative tool is revolutionizing the diagnosis of hair disorders. The use of a trichoscope has unveiled a plethora of signs which not only helps in decoding the underlying tricoscopic condition but also acts as prognostic markers. Herein, we present a new trichoscopic sign, "i hair" in alopecia areata and trichotillomania. "i hair" are short hairs with an accentuated distal end. There may be a thin hypopigmented shaft just beneath the darker distal end, thus making them resemble the alphabet "i.", Competing Interests: There are no conflicts of interest. What is new? i hair has not yet been reported in trichotillomania and alopecia areata. Its trichoscopic role as a prognostic marker in these conditions has not been visited before.

Stability is taken as the most important parameter before opting for any transplantation technique to treat vitiligo. But, simultaneous donor site repigmentation and depigmentation of grafts at the recipient site has been noted. Similarly donor site depigmentation with complete repigmentation of the recipient area with pigment growing out from each graft has been observed. Successful repigmentation after regrafting in previous punch failure cases has also been reported. Koebner's phenomenon from history (Kp-h) and test grafting were the only available indicators to assess stability. It is quite ironic to note that even after four decades of experience in vitiligo surgery, there seems to be little consensus among workers regarding the optimal required period of stability. Moreover, the exact concept of stability in vitiligo is itself still not transparent and defined beyond doubt Overdependence on KpH or TG may be sometimes misleading in vitiligo. These two reveal the apparent clinical stability only and that may not be the true reflection of stability status of the disease at the molecular level. Antimelanocyte cytotoxic reactivity was observed among CD8+ TCC isolated from perilesional biopsies of patients with vitiligo. An attempt should be made to clearly fathom and define stability, not merely only on clinical ground but along with electron microscopy and histoenzymological analysis of the perilesional and nonlesional skin of vitiligo patients. Probably some growth factors which are responsible for both mitogenic and melanogenic stimulation of melanocytes should also be taken into account. Some serological test(s) could guide us to measure these growth factors.