8 results on '"Kramer, L"'
Search Results
2. Infections II
- Author
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Guillaume, C., Godard, J., Mohammedi, I., Vedrinne, J. M., Bui-Xuan, B., Reverdy, M. E., Motin, J., Bonten, M., Van Tiel, F., Gaillard, C., Stobberingh, E., Van Der Geest, S., Faurisson, F., Peytavin, G., Allaouchiche, B., Torres, A., Ferrer, M., Aznar, E., Gatell, J. M., El-Biary, M., Puig, J., Gonzalez, J., Rodriguez-Roisin, R., Gratadour, P., Mardiquian, N., Di Roio, C., Godard, J., Inglis T. J. J., Sherratt M. J., Sproat L. J., Gibson J. S., Hawkey P. M., Sirvent, J. M., Verdaguer, R., Ferrer, M. J., Carratalá, J., Armengol, J., Bonet, A., Nouira, S., Elatrous, S., Bchir, A., Jaafoura, M., Abroug, F., Bouchoucha, S., Holzapfel, L., Chastang, Cl., Blanc, P. L., Carton, M. J., Legras, A., Schoch, P., Moret, G., Boiteau, R., Timsit, J. F., Garrait, V., Misset, B., Goldstein, F. W., Dumay, M. F., Carlet, J., Seller, G., Nieto, J. M. Sánchez, Carrillo, A., Rubí, J. A. Gómez, Climent, C., Gómez, J. Ruiz, Sola, J., Vaury, F. W. G. Ph., Francoual, S., Marquette, Ch. -H., Hérengt, F., Saulnier, F., Mathieu, D., Nevierre, R., Courcol, R., Ramon, Ph., Meunier, G., Gaussorgues, P., Sab, J. M., Nageotte, A., Doré, P., Robert, R., Grollier, G., Rouffineau, J., Lanquetot, H., Charrière, J. M., Elsman, B. H. P., Legemate, D. A., van Leeuwen, M. S., Feldberg, M. A. H., Obertop, H., Carducci, P., Annetta, M. G., Mignani, V., Rumi, C., Clemente, A., Wrenger, K., Baier, J., Mortion, B., Torwesten, E., Finke, W., Neumann, H., Puchstein, C., Nys, M., Damas, P., Kleinschmidt, R., Wolf, C., Möller, H., Spannbrucker, N., Madl, C., Koppensteiner, R., Kramer, L., Kranz, A., Lenz, K., Ehringer, H., Antonelli, M., Lanore, J. J., Raponis, G. M., Dhainaut, J. F., Martino, P., Rosa, G., Mancinis, C., Segneri, M., D’Errico, R. R., Gaaparetto, A., Capellier, G., Balvay, P., Boillot, A., Tissot, M., Raccado, E., Dupont, M. J., Barale, F., Davidson, J. A. H., Zhang, P., Boom, S. J., Blyth, A., Ramsay, G., Ramsay, G., Sanchez, M., Cambronero, J. A., Lopez, J., Cerda, E., Rodriguez, J. M., Rubio, J., Rogero, S., Nunez Reiz, A., De La Fuente O’Connor, E., Talou, M. Daguerre, García, M. Sánchez, Galache, J. A. Cambronero, Marin, S. Rogero, Reiz, A. Nuñez, Alvarez, B., Muñoz, F., Alvarez, F., Lopez, M. J., Maravi, E., Alvarez-Lerma, F., Tapia, V., Masdeu, G., Garrido, S., Vázquez-Sánchez, A., Nolla, J., Solsona, J. F., Gallet, E., Cacheux, P. Le, Beck, A., Her, B., Lecoutour, X., and Charbonneau, P.
- Published
- 1992
- Full Text
- View/download PDF
3. Detection of subclinical brain dysfunction by sensory evoked potentials in patients with severe diabetic ketoacidosis
- Author
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Eisenhuber, E., Madl, C., Kramer, L., Ratheiser, K., and Grimm, G.
- Published
- 1997
- Full Text
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4. Poster Discussions
- Author
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Purday, J. P., Taylor, S. J., Fettes, S. B., Manara, A. R., Raff, T., German, G., Barthold, U., Finnis, Mark E., Moran, John L., Leppard, Phil, Herman, Benjamin A., Rhodes, A., Malagon, I., Lamb, F. J., Newman, P., Grounds, R. M., Bennett, E. D., Rowan, Kathy, Beck, D. H., Taylor, B. L., Smith, G. B., Dequin, P. F., Capuzzo, M., Pavoni, V., Valpondi, V., Verri, M., Gritti, G., Ragazzi, R., MacKirdv, F. N., Livingston, B. M., Howie, J. C., Millar, B. W., Rué, M., Valero, C., Quintana, S., Artigas, A., Madl, C., Sterz, F., Kramer, L., Eisenhuber, E., Woolard, R. H., Gervais, H., Domanovits, H., Grimm, G., Steering Group and participants of the EURICUS I study, and Association des Réanimateurs du Centre-Ouest (ARCO).
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- 1996
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5. Metabolism II
- Author
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Pinilla, J., Phang, T., Baetz, M., Selvan, S., Hattori, S., Turani, F., Colella, D. F., Zupancich, E., Dauri, M., Actis Dato, G. M., Falco, M., Sabato, A. F., Planas M., Masclads J. R., Porta I., Iglesias R., Benejo B., de Latorre F. J., Schneeweiss, B., Pammer, J., Ratheiser, K., Madl, Ch., Kramer, L., Kranz, A., Fischer, M., Grimm, G., Lenz, K., Laterre, P. F., Hanique, G., Dugernier, Th., Andresen, M., Dougnac, A., Roeseler, J., Reynaert, M. S., Griffiths, R. D., Maclennan, P. A., Helliwell, T., and Macmillan, R. R.
- Published
- 1992
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6. Brain death and organ donors
- Author
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Rabanal, J. M., Quesada, A., Teja, J. L., Maestre, J. M., López-Espadas, F., Burtin, P., Mertès, P. M., Carteaux, J. P., Pinelli, G., Dopff, C., Villemot, J. P., Burlet, C., Boulangé, M., Depret, J., Graini, L., Berton, Ch., Bensadoun, H., Mercat, A., Teboul, J. -L., Auzépy, Ph., Richard, Ch., Navarro, A., Escalante, J. L., Grimm, G., Madl, Ch., Kramer, L., Sterz, F., Schneeweiss, B., Siostrzonek, P., Lenz, K., Rapoport, J., Teres, D., Lemeshow, S., and the Group of Transplant Co-ordinaton of the Community of Madrid
- Published
- 1992
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7. Time-dependency of sensory evoked potentials in comatose cardiac arrest survivors.
- Author
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Gendo, Alexandra, Kramer, Ludwig, Häfner, Michael, Funk, Georg-Christian, Zauner, Christian, Sterz, Fritz, Holzer, Michael, Bauer, Edith, Madl, Christian, Gendo, A, Kramer, L, Häfner, M, Funk, G C, Zauner, C, Sterz, F, Holzer, M, Bauer, E, and Madl, C
- Subjects
SOMATOSENSORY evoked potentials ,EVOKED potentials (Electrophysiology) ,ELECTROPHYSIOLOGY ,COMA ,LOSS of consciousness ,CARDIAC arrest ,PATIENTS ,COHORT analysis - Abstract
Objective: To assess the validity of early sensory evoked potential (SEP) recording for reliable outcome prediction in comatose cardiac arrest survivors within 48 h after restoration of spontaneous circulation (ROSC).Design and Setting: Prospective cohort study in a medical intensive care unit of a university hospital.Patients: Twenty-five comatose, mechanically ventilated patients following cardiopulmonary resuscitationMeasurements and Results: Median nerve short- and long-latency SEP were recorded 4, 12, 24, and 48 h after ROSC. Cortical N20 peak latency and cervicomedullary conduction time decreased (improved) significantly between 4, 12, and 24 h after resuscitation in 22 of the enrolled patients. There was no further change in short-latency SEP at 48 h. The cortical N70 peak was initially detectable in seven patients. The number of patients with increased N70 peak increased to 11 at 12 h and 14 at 24 h; there was no further change at 48 h. Specificity of the N70 peak latency (critical cutoff 130 ms) increased from 0.43 at 4 h to 1.0 at 24 h after ROSC. Sensitivity decreased from 1.0 at 4 h to 0.83 at 24 h after ROSC.Conclusion: Within 24 h after ROSC there was a significant improvement in SEP. Therefore we recommend allowing a period of at least 24 h after cardiopulmonary resuscitation for obtaining a reliable prognosis based on SEP. [ABSTRACT FROM AUTHOR]- Published
- 2001
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8. Clinical impact of arterial ammonia levels in ICU patients with different liver diseases.
- Author
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Drolz A, Jäger B, Wewalka M, Saxa R, Horvatits T, Roedl K, Perkmann T, Zauner C, Kramer L, Ferenci P, and Fuhrmann V
- Subjects
- Adult, Aged, Austria epidemiology, Biomarkers blood, Case-Control Studies, Disease-Free Survival, Female, Hepatitis diagnosis, Humans, Liver Cirrhosis diagnosis, Liver Failure, Acute diagnosis, Logistic Models, Male, Middle Aged, Multivariate Analysis, Prognosis, Prospective Studies, Ammonia blood, Hepatitis mortality, Liver Cirrhosis mortality, Liver Failure, Acute mortality
- Abstract
Purpose: Increased arterial ammonia levels are associated with high mortality in patients with acute liver failure (ALF). Data on the prognostic impact of arterial ammonia is lacking in hypoxic hepatitis (HH) and scarce in critically ill patients with cirrhosis., Methods: The patient cohort comprised 72 patients with HH, 43 patients with ALF, 100 patients with liver cirrhosis and 45 patients without evidence for liver disease. Arterial ammonia concentrations were assessed on a daily basis in all patients and the results were compared among these four patient groups and between 28-day survivors and 28-day non-survivors overall and in each group., Results: Overall 28-day mortality rates in patients with HH, ALF and cirrhosis and in the control group were 54, 30, 49 and 27 %, respectively. Peak arterial ammonia levels differed significantly between transplant-free 28-day survivors and non-survivors in the HH and ALF groups (p < 0.01 for both). Multivariate regression identified peak arterial ammonia concentrations as an independent predictor of 28-day mortality or liver transplantation in patients with HH and ALF, respectively (p < 0.01). There was no association between mortality and arterial ammonia in patients with liver cirrhosis and in the control group. Admission arterial ammonia levels were independently linked to hepatic encephalopathy grades 3/4 in patients with HH (p < 0.01), ALF (p < 0.05) and cirrhosis (p < 0.05), respectively., Conclusions: Elevated arterial ammonia levels indicate a poor prognosis in acute liver injury and are associated with advanced HE in HH, ALF and cirrhosis. Arterial ammonia levels provide additional information in the risk assessment of critically ill patients with liver disease.
- Published
- 2013
- Full Text
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