19 results on '"Oka, Hiroshi"'
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2. A multi-center double-blind controlled trial of ursodeoxycholic acid for primary biliary cirrhosis.
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Oka, Hiroshi, Toda, Gotaro, Ikeda, Yusei, Hashimoto, Naoaki, Hasumura, Yasushi, Kamimura, Tomoteru, Ohta, Yasuyuki, Tsuji, Takao, Hattori, Nobu, Namihisa, Toshihiko, Nishioka, Mikio, Ito, Ken'ichi, Sasaki, Hiroshi, Kakumu, Shin'ichi, Kuroki, Tetsuo, Fujisawa, Kiyoshi, and Nakanuma, Yasuo
- Abstract
A multi-center double-blind controlled trial of ursodeoxycholic acid (UDCA) for treatment of primary biliary cirrhosis (PBC) was carried out. Twenty two and 23 patients were treated with 600mg/day UDCA and placebo, respectively, for 24 weeks. In UDCA - treated patients, fall of serum aspartate aminotransferase, alanine aminotransferase, alkaline phosphatase and gamma glutamyltranspeptidase activities started within 4 weeks after start of the trial and continued throughout the trial period. The serum IgM level fell in 7 UDCA-treated patients examined but not in 10 placebo-treated patients examined. Serum bilirubin concentration showed no significant change at the end of the study in either of UDCA- and placebotreated group of patients. There was no significant difference between these two groups with respect to the frequency of improvement of pruritus. In UDCA-treated patients, serum bile acid composition changed markedly, though its concentation showed no significant change. The percentage of total bile acid which ursodeoxycholic acid took up increased, whereas those which cholic acid, chenodeoxycholic acid and deoxycholic acid took up were decreased. [ABSTRACT FROM AUTHOR]
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- 1990
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3. Perifusion of isolated rat pancreatic acini: Carbamylcholine-induced biphasic amylase release.
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Nagai, Masatoshi and Oka, Hiroshi
- Abstract
To solve many problems in other in-vitro methods such as perfusion and static incubation, perifusion of isolated rat pancreatic acini was established. Continuous stimulation by carbamylcholine induced a biphasec secretory response, a sharp initial phase and a slow-varying second phase. The junction of these two phases occurred at almost the same time (in about 7 min of stimulation) at any concentration of carbamylcholine. The slow-rising former part of a second phase indicated that a biphasic pattern is produced by overlapping of two kinds of real phases and the beginning of a real second phase occurs at the time of the junction. The concentration dependence of amylase release showed a bell-shaped pattern and the maximal amylase release at an optimal concentration (10M) was 5.43±0.24% total/30 min. The sustained latter part of a second phase showed a slow decline at optimal or less concentrations but a plateau at supraoptimal concentrations. [ABSTRACT FROM AUTHOR]
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- 1989
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4. A multi-centre double-blind controlled trial of glucagon and insulin therapy for severe acute hepatitis.
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Oka, Hiroshi, Fujiwara, Kenji, Okita, Kiwamu, Ishii, Hiromasa, and Sakuma, Akira
- Abstract
A cooperative study was conducted to determine the efficacy of one week of treatment with infusion of 1 mg glucagon and 10 units insulin twice daily in severe acute hepatitis. Ninty-eight patients with either prothrombin time less than 60% or thrombotest or hepaplastin test less than 50% of normal were randomly assigned to hormone or placebo treatment. SGOT and SGPT values dropped after treatment with a gradual decrease or increase in total serum bilirubin or cholesterol levels similarly in both groups receiving hormone or placebo. Deranged prothrombin time improved more rapidly in the hormone group than in the placebo group. Glucagon and insulin infusion may stimulate recovery of the liver from injury. [ABSTRACT FROM AUTHOR]
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- 1989
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5. Reciprocal changes in serum albumin and alpha-fetoprotein levels in the recovery course of acute viral hepatitis stimulated by glucagon and insulin therapy: Analysis of a double blind controlled trial.
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Fujiwara, Kenji, Ogata, Itsuro, and Oka, Hiroshi
- Abstract
The records of 57 patients with acute viral hepatitis (28 given hormones and 29 placebo) in a double blind controlled trial of one week of glucagon and insulin therapy were analyzed. In the placebo group, SGPT values dropped after treatment with improved prothrombin time and serum levels of total bilirubin and albumin. In the hormone group, they changed similarly, except serum albumin levels which were reversed during treatment (P< 0.05). There was a bottom line of serum albumin levels which preceded a peak of serum alpha-fetoprotein levels within one week in the placebo group, but was around the peak in the hormone group. There were 12 patients in the hormone group in whom serum alpha-fetoprotein levels rose with treatment and decreased after its discontinuation, and 5 in the placebo group (P< 0.05). Such a change in serum alpha-fetoprotein levels was accompanied by decreased serum albumin levels in 6 of 10 patients given hormones and none of the 5 given placebo (P< 0.05). These results indicate reciprocal changes in serum albumin and alpha-fetoprotein levels appearing during the recovery course of acute viral hepatitis, and suggest that this therapy may stimulate its development. Stimulation of liver regeneration by this therapy merits consideration. [ABSTRACT FROM AUTHOR]
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- 1989
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6. Antithrombin III concentrate in the treatment of fulminant hepatic failure.
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Fujiwara, Kenji, Okita, Kiwamu, Akamatsu, Koichi, Abe, Hirohiko, Tameda, Yukihiko, Sakai, Takahiro, Inoue, Noboru, Kanai, Koichi, Aoki, Nobuo, and Oka, Hiroshi
- Abstract
Twenty-six patients with fulminant hepatic failure were treated with daily infusions of antithrombin III concentrate until recovery of consciousness or death. Seven patients were alive (group A), 7 survived 17 to 47 days after treatment (group B), and 12 died within 9 days (group C). Decreased plasma antithrombin III levels increased on the day after treatment, irrespective of the pretreatment levels in all patients. Continuous or temporary normalization was seen in all patients in groups A and B, but in only 5 in group C patients whose bleeding was extensive (p<0.05). An abrupt drop in peripheral platelet counts occurred when plasma antithrombin III levels were below normal. General bleeding accompanied this drop. These results suggest that maintained normal plasma antithrombin III levels are beneficial for prolonged survival time in fulminant hepatic failure, probably through controlling intravascular coagulation, and that antithrombin III infusion may be useful for such treatment. [ABSTRACT FROM AUTHOR]
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- 1988
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7. A diagnostic marker of autoimmune chronic active hepatitis: Liver plasma membrane antibody in the serum absorbed with particulate fraction of kidney homogenate.
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Toda, Gotaro, Ikeda, Yusei, Hashimoto, Naoaki, Maruyama, Toshiyuki, and Oka, Hiroshi
- Abstract
The sera absorbed with the particulate fraction of rabbit kidney homogenate (RK) were tested for the antibody against liver plasma membrane (LPM-Ab) by the radiometric assay method. After incubation of the isolated rabbit liver plasma membrane (RLPM) with appropriately diluted serum, IgG bound to RLPM (IgG-RLPM) was determined usingI-labelled Staphylococcal protein A. IgC-RLPM in each subject tested was expressed in arbitrary units, that is, the multiple of the mean radioactivity associated with RLPM in 35 control subjects. Thus IgG-RLPM was 1.00 (mean) ± 0.23 (SD) in the control subjects, 1.13 ± 0.30 in 9 patients with chronic persistent hepatitis (CPH), 1.13 ± 0.52 in 15 with chronic active hepatitis (CAH), 1.34 ± 0.38 in 23 with liver cirrhosis (LC) and 3.45 ± 0.73 in 5 with autoimmune CAH. F(ab′) fragments from a patient with autoimmune CAH and a control subject decreased IgG-RLPM by 86.4 ± 6.35 and -5.2 ±14.9%, respectively, in four patients with autoimmune CAH. LPM-Ab was detected in 0, 20.0 and 17.4% in the patients with CPH, CAH and LC, respectively. All of the patients with autoimmune CAH were positive for LPM-Ab. The absorption of the sera positive for LPM-Ab with RK decreased IgG-RLPM in various extents. In two of five patients with autoimmune CAH and two of seven patients with CAH or LC, the majority of LPM-Ab was cross-reactive with RK. [ABSTRACT FROM AUTHOR]
- Published
- 1987
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8. Serum pepsinogens as a screening test of extensive chronic gastritis.
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Miki, Kazumasa, Ichinose, Masao, Shimizu, Akihiro, Huang, Shih, Oka, Hiroshi, Furihata, Chie, Matsushima, Taijiro, and Takahashi, Kenji
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The serum level of pepsinogen I (PG I) and pepsinogen II (PG II), and the PG I/PG II ratio were compared with the surface area of the fundic mucosa, as determined endoscopically by the Congo red staining method. Reduction in the area of the fundic mucosa due to gastritis was associated with stepwise reduction in the PG I levels and the PG I/PG II ratios. Reduction in the area of the fundic mucosa was also associated with decreases in the basal acid output, maximal acid output (MAO), the basal pepsin output and the stimulated pepsin output. The best sensitivity and specificity levels for the diagnosis of normal mucosa and severe gastritis were obtained with the PG I/PG II ratio and the MAO. A retrospective study of 58 patients with gastric cancer and 162 cancer-free patients showed that a PG I/PG II ratio identified 86.2% of all carcinomas and 87.5% of early carcinomas. Although this test gave a positive rate of 36% among the cancer-susceptible age group controls, its use would lower the cost of mass screening by targeting a smaller test population. [ABSTRACT FROM AUTHOR]
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- 1987
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9. Effects of calmodulin antagonists on secretion of bile and bile acid.
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Hashimoto, Naoaki, Maruyama, Toshiyuki, Toda, Gotaro, Ikeda, Yusei, Sugiyama, Yuichi, and Oka, Hiroshi
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In the isolated perfused rat liver, the effects of calmodulin (CaM) antagonists, chlorpromazine (CPZ), trifluoperazine (TFP), W-7 and W-5, on secretion of bile and bile acid were compared. Without addition of taurocholic acid to the perfusate, TFP (200 μM or higher), CPZ (200 μM) and W-7 (400 μM) decreased the bile flow transiently. In contrast, W-5 did not decrease the bile flow. Taking into consideration the binding of TFP and CPZ to bovine serum albumin in the perfusate, they diminished the bile flow by inhibiting CaM function. This was also supported by the difference between the effects of W-7 and W-5. These findings suggested that CaM was involved in the secretion of bile. Under the constant infusion of taurocholic acid into the perfusate, CaM antagonists decreased the secretion of bile acid. However this might be due to the inhibition of bile acid uptake, because these agents inhibited the uptake into isolated rat hepatocytes. The concentrations required for inhibition of the uptake were near to those which decreased the viability, suggesting that the inhibition was due to their non-specific cytotoxic effect. Future studies must be carried out to determine whether CaM is involved in the secretion of bile acid. [ABSTRACT FROM AUTHOR]
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- 1987
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10. Cyclic amp in gastric juice does not reflect histamine H receptor activity in heidenhain pouch dog.
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Kumagai, Junichi, Oka, Hiroshi, Kaneko, Eizo, and Honda, Nishio
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It is strongly believed that cAMP mediates histamine H receptor activity, but does not mediate gastrin and acetylcholine stimulation of gastric acid secretion. Therefore, cAMP production could be a marker of H receptor activity. Whether endogenous histamine mediates gastrin and/or acetylcholine stimulation, at least partialy, remains to be elucidated. If cAMP in the gastric juice reflects H receptor activity, we can investigate whether endogenous histamine mediates gastrin and/or acetylcholine stimulation in vivo. In this study, we investigated whether cAMP in the gastric juice reflected histamine H receptor activity in the Heidenhain pouch dog in vivo using different kinds of inhibitors of gastric secretion. Our hypothesis was as follows: Upon betazole stimulation, cimetidine, an H receptor antagonist, should decrease cAMP output into the gastric juice, but omeprazole, an H, K -ATPase blocker, should not, because it blocks at a site more peripheral than the H receptor and the production of cAMP. Sixty minutes after betazole administration, 4.0 µmol/kg of cimetidine and 0.18 µmol/kg omeprazole were administered intravenously and they inhibited gastric juice volume to a similar degree, that is, 49.6% and 52.1%, respectively. However, omeprazole caused a greater decrease in cAMP output than cimetidine. Inhibition with 4 /µmol/kg/h of cimetidine or 0.2 µmol/kg of omeprazole from the beginning of betazole stimulation also caused similar decreases in gastric juice volume, 66.6% and 60.6%, respectively. Both inhibitors decreased cAMP output into the gastric juice in a similar fashion in the first two 30 minute periods. These results do not agree with our hypothesis. It seems that cAMP in the gastric juice does not reflect histamine H receptor activity following betazole stimulation, at least as a major component. [ABSTRACT FROM AUTHOR]
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- 1986
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11. Human prolyl hydroxylase. Purification, radioimmunoassay and clinical studies in liver diseases.
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Nagai, Yoshiki and Oka, Hiroshi
- Abstract
Prolyl hydroxylase was purified from human placentae, specific antiserum against it was prepared, and a new radioimmunoassay system employingI-labelled enzyme preparation was established. The molecular weight of the placental enzyme was shown to be 320,000 by gel filtration. SDS-polyacrylamide gel electrophoresis showed two bands of unequal intensity having molecular weights of 60,000 and 130,000. Their amino acid compositions were identical to each other, suggesting the polypeptide with a molecular weight of 130,000 might be a dimer of the polypeptide with a molecular weight of 60,000. The new radioimmunoassay established had a sensitivity of the order of 10 ng/ml, indicating it was more sensitive than previous radioimmunoassay employingH-labelling method. Clinical studies on patients with liver diseases disclosed that the concentrations of serum immunoreactive prolyl hydroxylase were elevated both in cases of hepatocellular damage and in cases of cholestasis. In cases of hepatocellular damage the enzyme behaved like cytoplasmic enzymes such as glutamic oxaloacetic transaminase, glutamic pyruvic transaminase and lactic dehydrogenase, but in cases of cholestasis it resembled biliary enzymes such as alkaline phosphatase and γ-glutamyl transpeptidase. This result might be associated with the peculiar location of the enzyme within the cell, in the membrane of rough endoplasmic reticulum. [ABSTRACT FROM AUTHOR]
- Published
- 1985
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12. Proceedings of the 64th annual meeting from May 22 to 24, 1978-Sapporo, Japan.
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Okayama, Toshikata, Koizumi, Fumiaki, Watanabe, Hideo, Kaneko, Toshio, Oka, Hiroshi, Kaneto, Akio, Yanaihara, Noboru, Nakaya, Scishi, Itoh, Zen, Yamaguchi, Ken, Adachi, Isamu, Zeze, Fujio, Abe, Kaoru, Kameya, Toru, Arai, Saburo, Sato, Haruko, Yanaihara, Chizuko, Sugiyama, Mitsugi, Watanabe, Yozo, and Matsumura, Mitsuhiro
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- 1979
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13. Ultrastructural studies on pathogenesis of intrahepatic cholestasis.
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Sasaki, Hiroshi, Sano, Masaju, Ito, Kenishi, Sugahara, Katsuhiko, Mitani, Susumu, Ono, Keiichi, Shimano, Matsuro, Saisho, Hiromitsu, Kimura, Kunio, Mizoguchi, Yasuhiro, Monna, Takeyuki, Yamauchi, Hidemi, Sato, Toshio, Oka, Hiroshi, Toda, Gotaro, Fukazawa, Toshio, Okada, Toshimichi, Abei, Tohru, and Hosaka, Hiroo
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- 1977
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14. Cyclic amp in gastric juice does not reflect histamine H2receptor activity in heidenhain pouch dog
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Kumagai, Junichi, Oka, Hiroshi, Kaneko, Eizo, and Honda, Nishio
- Abstract
It is strongly believed that cAMP mediates histamine H2receptor activity, but does not mediate gastrin and acetylcholine stimulation of gastric acid secretion. Therefore, cAMP production could be a marker of H2receptor activity. Whether endogenous histamine mediates gastrin and/or acetylcholine stimulation, at least partialy, remains to be elucidated. If cAMP in the gastric juice reflects H2receptor activity, we can investigate whether endogenous histamine mediates gastrin and/or acetylcholine stimulation in vivo. In this study, we investigated whether cAMP in the gastric juice reflected histamine H2receptor activity in the Heidenhain pouch dog in vivo using different kinds of inhibitors of gastric secretion. Our hypothesis was as follows: Upon betazole stimulation, cimetidine, an H2receptor antagonist, should decrease cAMP output into the gastric juice, but omeprazole, an H+, K+-ATPase blocker, should not, because it blocks at a site more peripheral than the H2receptor and the production of cAMP. Sixty minutes after betazole administration, 4.0 µmol/kg of cimetidine and 0.18 µmol/kg omeprazole were administered intravenously and they inhibited gastric juice volume to a similar degree, that is, 49.6% and 52.1%, respectively. However, omeprazole caused a greater decrease in cAMP output than cimetidine. Inhibition with 4 /µmol/kg/h of cimetidine or 0.2 µmol/kg of omeprazole from the beginning of betazole stimulation also caused similar decreases in gastric juice volume, 66.6% and 60.6%, respectively. Both inhibitors decreased cAMP output into the gastric juice in a similar fashion in the first two 30 minute periods. These results do not agree with our hypothesis. It seems that cAMP in the gastric juice does not reflect histamine H2receptor activity following betazole stimulation, at least as a major component.
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- 1986
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15. Abstracts of selected papers presented at the 78th general meeting of the Japanese Society of Gastroenterology
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Kusano, Motoyasu, Sekiguchi, Toshikazu, Hanyu, Nobuyoshi, Aoki, Teruaki, Matsushima, Yasuhiro, Okamoto, Eizo, Takeda, Yasuo, Takeda, Ryoyu, Miyachi, Masahiko, Nimura, Yuji, Arai, Taidoh, Masuda, Jun, Tanaka, Masao, Ogawa, Yoshiaki, Ura, Kazuhide, Matsumoto, Teiji, Taniyama, K., Kurosawa, Susumu, Owyang, Chung, Baba, Hiroshi, Fujimura, Masaki, Hamada, Eiji, Shimada, Tadahito, Kabemura, Teppei, Chijiiwa, Yoshiharu, Sato, T., Koito, K., Matsuda, Hiroko, Kawasaki, Tsunehisa, Obara, Katsutoshi, Kasukawa, Reiji, Miyoshi, Hirofumi, Matsumoto, Akio, Yamamoto, Manabu, Ohmasa, Ryouji, Kokubu, Shigehiro, Shibata, Hisao, Tajiri, Takashi, Onda, Masahiko, Hashizume, Makoto, Sugimachi, Keizo, Kobayashi, Kenji, Shiozaki, Hitoshi, Fujisaki, Junko, Shimoda, Tadakazu, Hase, Satoshi, Tsukamoto, Yoshihisa, Atsumi, M., Konishi, H., Nakajo, Shinobu, Fujiyama, Yoshihide, Taruishi, Masaki, Ayabe, Tokiyoshi, Morise, Kimitomo, Yamaguchi, Takeo, Makiyama, Kazuya, Itsuno, Minoru, Matsui, T., Okabe, N., Okabe, Nobuo, Matsui, Toshiyuki, Sakatani, Arata, Koizumi, Koichi, Imai, Yutaka, Sugino, Yoshinori, Masaki, Tadahiko, Sawada, Toshio, Nishigami, Takashi, Satomi, Masamichi, Hatada, Yasumasa, Saito, Hiroshi, Kobayshi, Kiyonori, Katsumata, Tomoe, Sugimoto, Kenji, Itabashi, Tsukasa, Kitagawa, Motoji, Hayakawa, Tetsuo, Okazaki, Kazuichi, Yamamoto, Yasuro, Funakoshi, Akihiro, Miyasaka, Kyoko, Soejima, Kazuhiko, Kanda, Mikio, Sugiyama, Masanori, Kuroda, Akira, Inoue, Hisayuki, Bamba, Tadao, Hamanaka, Y., Suzuki, T., Suzuki, Mamoru, Hanyu, Fujio, Tamura, Katsuhiro, Nakase, Akira, Noguchi, M., Hiwatashi, N., Murata, Yuhji, Suzuki, Kazuo, Ohtani, Haruo, Watanabe, Yoshihisa, Zeniya, Mikio, Aizawa, Yoshio, Masumoto, T., Onji, M., Hamasaki, Keisuke, Nakata, Keisuke, Fukui, Hiroshi, Tsujii, Tadasu, Yamashiki, Masayoshi, Nishimura, Akira, Tsutsui, Hiroko, Mizoguchi, Yasuhiro, Kimura, Fumio, Miyazaki, Masaru, Kiyota, Keisuke, Inokuchi, Hideto, Yamamoto, Yoshihiro, Oka, Hiroshi, Horikoshi, Tsutomu, Sekiguchi, Toshikazu, Takayasu, H., Shirai, T., Hongo, Michio, Okuno, Yo, Okano, H., Aoyama, N., Ohsuki, Masao, Maeda, Kenji, Haruma, Ken, Sumii, Koji, Nakai, Yoshihide, Fukunaga, Mikihiko, Kaneko, Hiroshi, Morise, Kimitomo, Okumura, Toshikatsu, Uehara, Akira, Fukuda, Yoshihiro, Satomi, Masamichi, Joh, Takashi, Itoh, Makoto, Kitagawa, Yuko, Kitajima, Masaki, Iwao, Tadashi, Toyonaga, Atsushi, Nakamura, Masahiko, Oda, Masaya, Kawamura, Yukimitsu, Dohden, Kenji, Kamiyama, Yasuhiko, Matsuno, Seiki, Hirano, Morihisa, Otsuka, Sachio, Ito, Masahiro, Sekine, Ichiro, Ogihara, Tatsuo, Sato, Nobuhiro, Uehara, Akira, Namiki, Masayoshi, Sugiura, Nobuyuki, Ebara, Masaaki, Matsuo, Naoki, Uchida, Hideo, Okada, Shuichi, Okazaki, Nobuo, Tsujii, Hirohiko, Ohsuga, Toshiaki, Abe, M., Nagata, Y., Yamasaki, Susumu, Kosuge, Tomoo, Kitamoto, Mikiya, Nakanishi, Toshio, Ichikawa, Yuzo, Mizoguchi, Yasuhiro, Ohtake, Yoshio, Hirasawa, Hiroyuki, Sugihara, Junichi, Muto, Yasutoshi, Yasunaga, Mitsuru, Okita, Kiwamu, Ukida, Minoru, Tsuji, Takao, Isai, Hideya, Uchino, Junichi, Suzuki, Katsuhiko, Komatsu, Kanji, Ohtomo, Yumiko, Idezuki, Yasuo, Sakuramachi, Shunji, Kimura, Taizo, Kitano, Seigo, Sugimachi, Keizo, Moriyama, Masaaki, Yazaki, Yasuyuki, and Saito, Takashige
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- 1993
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16. Proceedings of the 69th General Meeting from April 11–13, 1983-Osaka, Japan
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Baba, Yoshihiro, Nagata, Kunio, Tsukada, Yoshihisa, Narisawa, Rintaro, Honma, Akira, Hirose, Shinichi, Tomizawa, Mineo, Ozaki, Toshihiko, Ichida, Fumihiro, Tanehiro, Kenji, Kuno, Nobuyoshi, Kurimoto, Kumiko, Yokota, Tetsuo, Kano, Tomoyuki, Kasugai, Tatsuzo, Ueno, Kazuya, Shimakura, Katsuhide, Kakita, Akira, Kambayashi, Masaaki, Takahashi, Tsuyoshi, Sasaki, Eisei, Kasai, Yoichi, Takahashi, Toshihisa, Shimaguchi, S., Ariyama, J., Sumida, Masatoshi, Tsuchiya, Yukihiro, Ohto, Masao, Asagami, Fumio, Fuji, Tadasu, Okabe, Norimasa, Sakurai, Kenji, Miyakawa, Shuichi, Horiguchi, Yuji, Nakano, Hiroshi, Nimura, Yuji, Hayakawa, N., Hasegawa, H., Kamiya, J., Maeda, S., Iyomasa, Y., Matsumoto, Yoshiro, Sugahara, Katsuhiko, Ida, T., Mashimo, R., Wen, Shu Kou, Fujii, H., Wakashiro, S., Isowa, G., Itoh, S., Yamakawa, T., Udaka, Hidenori, Miyamoto, Hideyuki, Tamura, Toshikazu, Hirai, Tsutomu, Okamura, Osamu, Komi, Nobuhiko, Yoshioka, Nobuo, Kawamura, Yoshio, Kojima, Takahiko, Hachiya, Hitoshi, Okumura, Nobuyoshi, Suzuki, Kunihiko, Suzuki, Toshiyuki, Suzuki, Sadasuke, Ozeki, Norishige, Goto, Kazuo, Shiraki, Shigehiro, Takeuchi, Toshihiko, Yamaguchi, Atsumasa, Shibue, Tadashi, Kawaura, Yukimitsu, Iwa, Takashi, Takeda, Isao, Nakano, Satoshi, Kobayashi, Tomoko, Harada, Hideo, Wakabayashi, Tokio, Sawabu, Norio, Naito, Yasuo, Nakazawa, Saburo, Isawa, Tomoaki, Tabata, Ikuo, Sasaki, Yuichi, Yamada, Hideaki, Tatsumi, Shunichi, Kobayashi, Kenzo, Suzuki, Eitaro, Okamoto, Eizo, Matsukawa, Masakatsu, Tabata, Ikuo, Nakanishi, Toshio, Kawakami, Hiroiku, Fujimoto, Sotaro, Nakajima, Masatsugu, Sugihara, Junichi, Saitoh, Yoichi, Matsushiro, Takashi, Nagashima, Hideyuki, Yamamoto, Kyoji, Nakamura, Ryuji, Tanaka, Junichi, Shimizu, Fumito, Toshima, Takashi, Hariu, Tuneo, Nagakawa, Takukazu, Suzuki, Noriyoshi, Takahashi, Wataru, Uematsu, Ikunoshin, Sato, Toshio, Egami, K., Tajiri, T., Aoki, N., Yamaguchi, K., Yamakawa, H., Watanabe, A., Yano, M., Hatta, S., Yoshioka, M., Miki, M., Shirota, A., Morimoto, Koji, Furukawa, Masato, Nakata, Toshinori, Yamada, Ryuhei, Ito, Shinichiro, Maeda, Shigeru, Morinaga, Toshinori, Tanigawa, Makota, Fujio, Toshiyuki, Inui, Hiroshi, Kinoshita, Hiroaki, Yamasaki, Osamu, Nagata, Eiichi, Hirohashi, Kazuhiro, Sakai, Katsuz8i, Takada, Tadahiro, Yasuda, Hideki, Shishikura, Makoto, Uchiyama, Katsuhiro, Ogura, Yoshifumi, Mizumoto, Ryuji, Nakazawa, Kazuomi, Sato, Shunichi, Kaito, Isamu, Suzuki, Hiroshi, Ohtsuki, Masao, Goto, Yoshio, Nomoto, M., Yunoue, K., Soga, K., Ichida, F., Koike, Y., Kiyosawa, K., Akahane, Y., Kamijyo, K., Suzuki, Y., Yamamura, S., Komatsu, T., Nagata, A., Furuta, S., Hayashi, Shigeki, Ohta, Yasuhiko, Fujiwara, Kenji, Sato, Yuzuru, Ogata, Itsuro, Takatsuki, Katsuyoshi, Mishiro, Shunji, Oka, Hiroshi, Furube, Masaru, Yamada, Nobuo, Shibata, Hisao, Shibuya, Akitaka, Hijikata, Eishi, Kokubu, Shigenobu, Ishii, Kohdo, Okabe, Haruya, Sasaki, Kenichi, Takada, Akira, Takeuchi, Jugoro, Ohta, Yasuyuki, Tsujii, Tadashi, Ikegame, Fumiaki, Unoura, Masashi, Furusawa, Akihiko, Tanaka, Nobuyoshi, Kato, Yasuhiro, Kobayashi, Kenichi, Hattori, Nobu, Tameda, Yukihiko, Kakiuchi, Satoshi, Kosaka, Yoshitane, Harihara, Shigeyoshi, Yamamoto, Sukeo, Yamada, Gotaro, Nagashima, Hideo, Onji, Morikazu, Yamashita, Yoshimasa, Horiike, Norio, Ohta, Yasuyuki, Abe, Hirohiko, Hino, Kazuhiko, Kojima, Masaru, Noguchi, Kazunori, Ueda, Hiromu, Aritaka, Tomoki, Maruyama, Naoto, Motoori, Hitoshi, Yamauchi, Kazuaki, Setoyama, Hiroshi, Sata, Michio, Kubo, Yasuhiko, Tanikawa, Kyuichi, Munehisa, Tatsuo, Nakata, Keisuke, Kono, Kenzo, Muro, Toyoichi, Sato, Akira, Furukawa, Ryuji, Ishii, Nobuko, Kusumoto, Yukio, Koji, Toshihiko, Nagataki, Shigenobu, Nakao, Norio, Miura, Kohi, Sato, Y., Ohta, Y., Ogata, I., Hayashi, S., Fujiwara, K., Oka, H., Furui, S., Iio, M., Ikeda, Kenji, Oyake, Eiji, Takeuchi, Kazuo, Kumada, Hiromitsu, Nakajima, Masao, Yoshiba, Akira, Irimoto, Masahiro, Kobayashi, Toshio, Ohto, Masao, Satoh, Morio, Yamada, Ryusaku, Nakatsuka, Haruki, Yamada, Ryusaku, Kubo, Yasuhiko, Hirai, Kenji, Kumagai, Masanobu, Yamaguchi, Genjiro, Tanaka, Masatoshi, Abe, Masanobu, Tanikawa, Kyuichi, Ando, Keijiro, Shingai, Yasushi, Okita, Kiwamu, Takemoto, Tadayoshi, Tarao, Kazuo, Iwamura, Kenichiro, Soga, Kenji, Honma, Akira, Nomoto, Minoru, Takagi, Hitoshi, Ichida, Fumihiro, Ichida, Takafumi, Higashi, Shunsaku, Noguchi, Takashi, Mizumoto, Ryuji, Kanematsu, Takashi, Takenaka, Kenji, Matsumata, Takashi, Sonoda, Takashi, Furuta, Toshiya, Sugimachi, Keizo, Inokuchi, Kiyoshi, Yamasaki, Susumu, Hasegawa, Hiroshi, Makuuchi, Masatoshi, Nakanishi, Yoshimi, Kakita, Akira, Sano, Hidekazu, Konno, Tetsuro, Sano, Fumio, Kasai, Yoichi, Tsuzuki, Toshiharu, Iida, Shuhei, Yamanaka, Naoki, Okamoto, Eizo, Kuwata, Keiji, Toyosaka, Akihiro, Tanaka, Nobutaka, Fujiwara, Shiro, Yamazaki, Hajime, Hirohashi, Kazuhiro, Kinoshita, Hiroaki, Sakai, Katsuji, Ono, Tokio, Yamada, Hiroaki, Tada, Masahiro, Asakura, Hitoshi, Kobayashi, Kensuke, Morishita, Tetsuo, Tsuchiya, Masaharu, Sato, Nobutaka, Hiratsuka, Hideo, Ueda, N., and Harada, K.
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- 1984
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17. Abstracts of selected papers presented at the 30th Annual Meeting of the Japanese Society of Gastroenterology October 20–22, 1988 — Kagoshima, Japan
- Author
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Oda, Ichiro, Ogawa, Kaoru, Okada, Shuichi, Ueno, Keiichi, Itoh, Tohru, Horiguchi, Yuji, Kijima, Hiroshi, Inui, Kazuo, Koga, Akitoshi, Hamamoto, Tetsuro, Tsukada, Hideaki, Tamada, Takashi, Nakamura, Takahiko, Okanoue, T., Koike, Daisuke, Abe, Hirohiko, Miura, Tsutomu, Hashimoto, Shuji, Oda, M., Azuma, T., Nagata, Shigeyuki, Ishii, Hiromasa, Kawada, Norifumi, Mizoguchi, Yasuhiro, Yoshikawa, Yuji, Oka, Hiroshi, Ichida, Takafumi, Ichida, Fumihiro, Ueno, Takato, Tanikawa, Kyuichi, Kokudo, Norihiro, Kawasaki, Seiji, Onji, Morikazu, Miyaoka, Hiroaki, Yokosuka, Osamu, Omata, Masao, Ikeda, Hiroshi, Tsuji, Takao, Hayashi, Shigeki, Fujiwara, Kenji, Yasunaga, Mitsuru, Okita, Kiwamu, Takase, Koujirou, Tameda, Yukihiko, Ohnishi, Hiroo, Sugihara, Jun-ichi, Kojima, Hideo, Kamimura, Tomoteru, Tanaka, Eiji, Yoda, Hidetoshi, Kanno, Atsushi, Ohtsuki, Masao, Miyata, Yasuji, Koga, Shunichi, Unoura, Masashi, Tanaka, Nobuyoshi, Kodama, Takahiro, Nishimura, Hideo, Suzuki, Kazuyuki, Madarame, Takeo, Komatsu, Masafumi, Ookubo, Shunji, Nishiyama, Masataka, Saito, Shozo, Kumashiro, Ryukichi, Tanikawa, Kyuichi, Ikeda, Kenji, Kumada, Hiromitsu, Nishiguchi, Shuhei, Kuroki, Tetsuo, Yoshiba, Makoto, Takeuchi, Yukari, Morito, Takao, Kasukawa, Reiji, Takeda, Isao, Nakano, Satoshi, Matsumura, Kenzo, Imanari, Tomohiro, Lee, Shigeki, Fujita, Naotaka, Mine, Tetsuya, Sato, Eiichi, Suyama, Masafumi, Ariyama, Jo, Takahashi, Toshihisa, Hirano, Masanori, Hirai, Atsushi, Yamakawa, Tatsuo, Kamiya, Junichi, Nimura, Yuji, Yoshimoto, Hideo, Ikeda, Seiyo, Yamasuji, Tadashi, Saeki, Keizo, Itoh, Masaki, Kajiyama, Goro, Seki, Hideichi, Kimura, Ken, Nakamura, Hiroshi, Tsuchiya, Yukihiro, Omura, Ryosuke, Tanaka, Shinya, Shimakura, Katsuhide, Matsuda, Yoshiaki, Wakabayashi, Tokio, Morimoto, Hideo, Kamiya, Yasutaka, Okayama, Yasutaka, Kobayashl, Masao, Fujimoto, Sotaro, Yamaguchi, Atsumasa, Shibue, Tadashi, Akashi, Ryukichi, Hattori, Masahiro, Takehira, Yasunori, Tsugane, Yasutoshi, Tashiro, Yoshinori, Kuwayama, Hajime, Kon, Yoichi, Higuchi, Tsugio, Hirata, Nobuto, Kano, Tomoyuki, Kurimoto, Kumiko, Kubota, Y., Seki, T., Kamura, Yoshimine, Nakayama, Toshimichi, Yoshimura, Hitoshi, Yoshioka, Tetsuya, Akimoto, Kimihiko, Mine, Tetsuya, Sato, Eiichi, Ohnishi, Hirohide, Ogata, Etsuro, Igarashi, Atsushi, Kadowaki, Atsushi, Yaoita, Tsutomu, Sato, Naoki, Monma, Kimitsune, Kogure, Hiroaki, Tajima, Yoshio, Takanashi, Hideki, Tsuchiya, Yukihiro, Haniya, Kazuo, Ohto, Masao, Murata, Ikuo, Tanaka, Takashi, Sakabe, Takashi, Kato, Takashi, Koike, Tadashi, Watanabe, Tetsuya, Nakamura, Tetsuo, Kato, H., Ikai, I., Nishimura, N., Kobayashi, N., Ozawa, K., Ono, Katsuyuki, Sata, Michio, Abe, Hirohiko, Tanikawa, Kyuichi, Kadowaki, Ken, Ikoma, Jiro, Kojima, Yuji, Murata, Tetsuya, Kakehashi, Ryuichi, Ibe, Toshio, Kano, Uichiro, Ito, Tetsuo, Tanaka, Takeshi, Watanabe, Shozo, Suzuki, Shiro, Nakazawa, Osamu, Kohda, Kyuhei, Ohyama, Kohzo, Terada, Shoki, Sazaki, Katsuhiko, Kure, Teikichi, Morita, Koetsu, Shida, Osamu, Muramatsu, Hiroshi, Niitsu, Yoshiro, Tozuka, Shinichi, Sakai, Yoshinori, Ikeda, Takaaki, Koyama, Wataru, Sakamoto, Shigemi, Kanayama, Masaaki, Tanaka, Naohide, Arakawa, Yasuyuki, Matsuo, Yutaka, Takaguchi, Kouichi, Yamada, Gotaro, Matsuura, Kazuharu, Ikeda, Hirosi, Iwasaki, Yoshiaki, Nouso, Kazuhiro, Matsueda, Kazuhiro, Sawahara, Masahiko, Fujiki, Shigeatsu, Mizuno, Motowo, Yama, Shingo Kino, Tsuji, Takao, Nadano, Seijin, Horiike, Norio, Michitaka, Kojiro, Kumon, Izumi, Ogawa, Yasushi, Yamaguchi, Shuji, Onji, Morikazu, Ohta, Yasuyuki, Nishi, Masaaki, Matsuzaki, Yasushi, Tanaka, Naomi, Matsumoto, Hisashi, Chuganji, Yoshimichi, Osuga, Toshiaki, Shoji, S., Kurata, M., Wakashima, M., Itoh, Y., Hagiri, M., Kondou, T., Katayama, M., Nishizawa, K., Takikawa, H., Ohsawa, H., Miyake, M., Yamanaka, M., Ooka, Teruji, Akita, Masahiko, Kim, Hogen, Kato, Michio, Masuzawa, Manabu, Okuyama, Takumasa, Akiyama, Masahiko, Ishigami, Yoshitaka, Tamura, Kazuya, Fukui, Osamu, Abe, Hiroshi, Kamada, Takenobu, Arai, Ken, Shindo, Michiko, Okuno, Tadao, Matsumoto, Masayuki, Takeda, Makoto, Takino, Tatsuro, Sokawa, Yoshihiro, Omata, M., Yokosuka, O., Ito, Y., Hosoda, K., Ohto, M., Fuse, Yoshinobu, Kodama, Tadashi, Sugiyama, Kenji, Nakajo, Shinobu, Ajitsu, Shin, Takahashi, Tsuneo, Kuroe, K., Murata, Y., Ikeda, Hideo, and Tanikawa, Kyuichi
- Published
- 1990
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18. (1) Cytological aspects of gastro-enteric hormone secretion.
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Fujita, Tsuneo, Yanaihara, Noboru, Tachibana, Shinro, Kaneko, Toshio, Oka, Hiroshi, Matsuo, Yutaka, Itoh, Zen, Aizawa, Isamu, and Takeuchi, Shinjin
- Published
- 1976
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19. Thyroid function in liver cirrhosis and effect of thyroxine on collagen metabolism in experimentally induced cirrhotic liver of rats.
- Author
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Oka, Hiroshi and Sakai, Takahiro
- Published
- 1969
- Full Text
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