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1. Molecular basis of the attenuation exhibited by molecularly cloned highly passaged chicken anemia virus isolates.

Author

Todd D, Scott AN, Ball NW, Borghmans BJ, and Adair BM

Subjects
Animals, Antibodies, Monoclonal, Capsid genetics, Capsid Proteins, Chicken anemia virus immunology, Chicken anemia virus isolation & purification, Chickens, Chimera genetics, Chimera immunology, Circoviridae Infections immunology, Circoviridae Infections prevention & control, Cloning, Molecular, DNA, Viral genetics, Genome, Viral, Molecular Sequence Data, Vaccines, Attenuated genetics, Viral Vaccines genetics, Virulence genetics, Chicken anemia virus genetics, Chicken anemia virus pathogenicity
Abstract

Chimeric virus experiments indicated that the pathogenicity and monoclonal antibody reactivity differences between two molecularly cloned, highly passaged chicken anemia virus isolates could be attributed to the VP1 amino acid change at residue 89. The introduction of this change into a pathogenic cloned low-passage isolate was not sufficient to cause attenuation.

Published
2002
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