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3 results on '"Wang, Ti"'

1. Aspartate all-in-one doping strategy enables efficient all-perovskite tandems

Author

Zhou, Shun, primary, Fu, Shiqiang, additional, Wang, Chen, additional, Meng, Weiwei, additional, Zhou, Jin, additional, Zou, Yuanrong, additional, Lin, Qingxian, additional, Huang, Lishuai, additional, Zhang, Wenjun, additional, Zeng, Guojun, additional, Pu, Dexin, additional, Guan, Hongling, additional, Wang, Cheng, additional, Dong, Kailian, additional, Cui, Hongsen, additional, Wang, Shuxin, additional, Wang, Ti, additional, Fang, Guojia, additional, and Ke, Weijun, additional

Published
2023
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2. Regulation of synaptic responses to high-frequency stimulation and LTP by neurotrophins in the hippocampus

Author

Figurov, Alexander, Pozzo-Miller, Lucas D., Olafsson, Petur, Wang, Ti, and Lu, Bai

Subjects
Hippocampus (Brain) -- Research, Neural stimulation -- Research, Environmental issues, Science and technology, Zoology and wildlife conservation
Abstract

The brain-derived neurotrophic factor (BDNF) stimulates long-term potentiation induced by theta-burst (LTP(sub T)) by increasing the capability of the hippocampal synapses to respond to high-frequency stimulations. Hippocampal slices from postnatal day 12-13 are unable to respond to tetanic stimulation in the absence of BDNF. In the presence of BDNF, the slices show LTP(sub T). A TrkB-IgG fusion protein that removes BDNF suppresses synaptic responses to tetanus and LTP(sub T) level.

Published
1996

3. Nitric oxide mediates activity-dependent synaptic suppression at developing neuromuscular synapses

Author

Wang, Ti, Xie, Zuoping, and Lu, Bai

Subjects
Neuromuscular junction -- Research, Nitric oxide -- Physiological aspects, Environmental issues, Science and technology, Zoology and wildlife conservation
Abstract

Nitric oxide (NO) acts as a retrograde signal in the activity-dependent synaptic inhibition at neuromuscular synapses. Spontaneous and invoked synaptic currents are blocked by NO donors and activators of the cyclic GMP channel. NO released from a postsynaptic myocyte induces synaptic suppression. The NO-binding protein hemoglobin and NO synthase inhibitors avert the synaptic suppression triggered by postsynpatic depolarization.

Published
1995

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