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Your search keyword '"de Cristofaro, V."' showing total 6 results
Start Over Author "de Cristofaro, V." Journal nephrology dialysis transplantation
6 results on '"de Cristofaro, V."'

4. Rate dependence of acute PTH release and association between basal plasma calcium and set point of calcium-PTH curve in dialysis patients.

Author

De Cristofaro, V, Colturi, C, Masa, A, Comelli, M, and Pedrini, L A

Abstract

In vivo, the control of calcium-mediated acute PTH release during induced hypo- or hypercalcaemia is linked not only to plasma calcium concentration per se but also to the rate and direction of calcium change. In fact, during induced hypocalcaemia, the predominant mechanism that causes PTH to be released is the reduction of plasma Ca(2+) irrespective of the absolute starting concentration of ionized calcium. This mechanism, which is rate-dependent and even activated in conditions of hypercalcaemia, may be involved in the association, reported in several papers, between the basal Ca(2+) and the set point of the calcium-PTH curve.

Published
2001
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5. Acquired Immunodeficiency Syndrome Transmitted by Transplanted Kidney: Clinical Course During Maintenance Haemodialyis.

Author

Imbasciati, E., De Cristofaro, V., Samà, F., Pagliari, B., and Baretta, A.

Abstract

We describe the clinical course of a 39-year-old man who developed AIDS during maintenance haemo dialysis. The infection had been transmitted by cadaveric kidney transplantation from a donor with a history of i.v. drug abuse. Fever, splenomegaly, hypergammaglobulinaemia and thrombocytopenia were the first signs, and appeared when haemodialysis was resumed 26 months after transplantation. Twenty-eight months later the patient developed a cerebral abscess due to toxoplasma infection. A striking improvement was obtained with cotrimoxazole, but the treatment had to be stopped because of severe leukopenia. The patient died due to relapse of the cerebral abscess. End-stage renal failure does not seem to have modified the clinical course of AIDS in our patient. [ABSTRACT FROM PUBLISHER]

Published
1988

6. Sudden death and associated factors in a historical cohort of chronic haemodialysis patients

Author

Andrea Stella, Simonetta Genovesi, Emanuela Rossi, Antonio Vincenti, Daniela Pogliani, Maria Grazia Valsecchi, Irene Acquistapace, Vincenzo De Cristofaro, Genovesi, S, Valsecchi, M, Rossi, E, Pogliani, D, Acquistapace, I, De Cristofaro, V, Stella, A, and Vincenti, A

Subjects
Adult, Male, medicine.medical_specialty, medicine.medical_treatment, Sudden death, atrial fibrillation, diabetes, haemodialysis, hyperkalemia, sudden death, Sudden cardiac death, Diabetes Complications, Death, Sudden, Young Adult, Renal Dialysis, Risk Factors, Cause of Death, Internal medicine, Atrial Fibrillation, Humans, Medicine, Cumulative incidence, Prospective Studies, Prospective cohort study, Aged, Cause of death, Transplantation, business.industry, Incidence, Incidence (epidemiology), Middle Aged, Prognosis, medicine.disease, Surgery, C-Reactive Protein, Nephrology, Cohort, Hyperkalemia, Female, Hemodialysis, business
Abstract

Background In haemodialysis patients, deaths due to cardiovascular causes constitute a large proportion of total mortality and sudden cardiac deaths account for approximately 22% of all deaths. The aim of this study was to evaluate the incidence of sudden cardiac death and associated risk factors in a cohort of haemodialysis patients. Methods and results The 3-year cumulative incidence of death in a cohort of 476 patients on chronic haemodialysis treatment was 34.3% (SE 2.3). Sudden death had a 6.9% (SE 1.2) cumulative incidence, with 32 events representing 19.2% of all deaths, while cardiovascular not sudden death and noncardiovascular death accounted for a 3-year cumulative incidence of 7.3% (SE 1.2) and 20.1% (SE 1.9), respectively. According to Cox multivariate analysis, significant risk factors for sudden death were the presence of atrial fibrillation, diabetes mellitus, predialytic hyperkalaemia, haemodialysis mode and C-reactive protein level, which were associated with a 2.9 (CI(95%) 1.3-6.4), 3.0 (CI(95%) 1.3-7.2), 2.7 (CI(95%) 1.3-5.8), 4.5 (CI(95%) 1.3-15.5) and 3.3 (CI(95%) 1.2-8.8)-fold increase in the risk of sudden death, respectively. Sudden death was significantly more frequent during the first 24 h of the first short interdialytic interval and during the last 24 h of the long interval, i.e. immediately before and immediately after the first weekly haemodialysis session (P = 0.02). Conclusions Our data show that the incidence of sudden death in haemodialysis patients is high and that atrial fibrillation, diabetes, hyperkalaemia, haemodialysis mode and C-reactive protein play an important role in developing fatal arrhythmia. Further studies will be necessary to define which interventions could be helpful in reducing this cause of mortality.

Published
2009
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