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Your search keyword '"Zhou, Likun"' showing total 9 results
Start Over Author "Zhou, Likun" Journal oncology reports
9 results on '"Zhou, Likun"'

1. [Retracted] Effects of miR‑138‑5p and miR‑204‑5p on the migration and proliferation of gastric cancer cells by targeting EGFR

Author

Wang, Yi, primary, Zhang, Haiyang, additional, Ge, Shaohua, additional, Fan, Qian , additional, Zhou, Likun, additional, Li, Hongli, additional, Bai, Ming, additional, Ning, Tao , additional, Liu, Rui , additional, Wang, Xia, additional, Deng, Ting, additional, Zhang, Le, additional, Ying, Guoguang, additional, and Ba, Yi, additional

Published
2023
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2. [Corrigendum] miR‑370 regulates cell proliferation and migration by targeting EGFR in gastric cancer

Author

Ning, Tao, primary, Zhang, Haiyang, additional, Wang, Xinyi, additional, Li, Shuang, additional, Zhang, Le, additional, Deng, Ting, additional, Zhou, Likun, additional, Liu, Rui, additional, Wang, Xia, additional, Bai, Ming, additional, Ge, Shaohua, additional, Li, Hongli, additional, Huang, Dingzhi, additional, Ying, Guoguang, additional, and Ba, Yi, additional

Published
2022
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4. Effects of miR‑138‑5p and miR‑204‑5p on the migration and proliferation of gastric cancer cells by targeting EGFR

Author

Wang, Yi, primary, Zhang, Haiyang, additional, Ge, Shaohua, additional, Fan, Qian, additional, Zhou, Likun, additional, Li, Hongli, additional, Bai, Ming, additional, Ning, Tao, additional, Liu, Rui, additional, Wang, Xia, additional, Deng, Ting, additional, Zhang, Le, additional, Ying, Guoguang, additional, and Ba, Yi, additional

Published
2018
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5. miR-370 regulates cell proliferation and migration by targeting EGFR in gastric cancer

Author

Ning, Tao, primary, Zhang, Haiyang, additional, Wang, Xinyi, additional, Li, Shuang, additional, Zhang, Le, additional, Deng, Ting, additional, Zhou, Likun, additional, Liu, Rui, additional, Wang, Xia, additional, Bai, Ming, additional, Ge, Shaohua, additional, Li, Hongli, additional, Huang, Dingzhi, additional, Ying, Guoguang, additional, and Ba, Yi, additional

Published
2017
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6. miR-455 inhibits cell proliferation and migration via negative regulation of EGFR in human gastric cancer

Author

Ning, Tao, primary, Peng, Zhijuan, additional, Li, Shuang, additional, Qu, Yanjun, additional, Zhang, Haiyang, additional, Duan, Jingjing, additional, Wang, Xinyi, additional, Yang, Haiou, additional, Liu, Rui, additional, Deng, Ting, additional, Bai, Ming, additional, Wang, Yi, additional, Si, Yiran, additional, Zhang, Le, additional, Wang, Xia, additional, Ge, Shaohua, additional, Zhou, Likun, additional, Ying, Guoguang, additional, and Ba, Yi, additional

Published
2017
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7. Integrated analysis of the miRNA, gene and pathway regulatory network in gastric cancer

Author

ZHANG, HAIYANG, primary, QU, YANJUN, additional, DUAN, JINGJING, additional, DENG, TING, additional, LIU, RUI, additional, ZHANG, LE, additional, BAI, MING, additional, LI, JIALU, additional, ZHOU, LIKUN, additional, NING, TAO, additional, LI, HONGLI, additional, GE, SHAOHUA, additional, LI, HUA, additional, YING, GUOGUANG, additional, HUANG, DINGZHI, additional, and BA, YI, additional

Published
2015
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9. Integrated analysis of the miRNA, gene and pathway regulatory network in gastric cancer.

Author

Zhang H, Qu Y, Duan J, Deng T, Liu R, Zhang L, Bai M, Li J, Zhou L, Ning T, Li H, Ge S, Li H, Ying G, Huang D, and Ba Y

Subjects
Apoptosis genetics, Cell Differentiation genetics, Cell Division genetics, Cell Transformation, Neoplastic genetics, Humans, Multigene Family, Neoplasm Metastasis, Neoplasm Proteins physiology, Stomach Neoplasms metabolism, Gene Expression Regulation, Neoplastic genetics, MicroRNAs genetics, Signal Transduction genetics, Stomach Neoplasms genetics
Abstract

Gastric cancer is one of the most common malignant tumors worldwide; however, the efficacy of clinical treatment is limited. MicroRNAs (miRNAs) are a class of small non-coding RNAs that have been reported to play a key role in the development of cancer. They also provide novel candidates for targeted therapy. To date, in-depth studies on the molecular mechanisms of gastric cancer involving miRNAs are still absent. We previously reported that 5 miRNAs were identified as being significantly increased in gastric cancer, and the role of these miRNAs was investigated in the present study. By using bioinformatics tools, we found that more than 4,000 unique genes are potential downstream targets of gastric cancer miRNAs, and these targets belong to the protein class of nucleic acid binding, transcription factor, enzyme modulator, transferase and receptor. Pathway mapping showed that the targets of gastric cancer miRNAs are involved in the MAPK signaling pathway, pathways in cancer, the PI3K-Akt signaling pathway, the HTLV-1 signaling pathway and Ras signaling pathway, thus regulating cell growth, differentiation, apoptosis and metastasis. Analysis of the pathways related to miRNAs may provides potential drug targets for future therapy of gastric cancer.

Published
2016
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