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Your search keyword '"Teixeira, Mauro M."' showing total 10 results
Start Over Author "Teixeira, Mauro M." Journal plos one
10 results on '"Teixeira, Mauro M."'

1. Tumor Necrosis Factor, but Not Neutrophils, Alters the Metabolic Profile in Acute Experimental Arthritis

Author

Oliveira, Marina C., primary, Tavares, Luciana P., additional, Vago, Juliana P., additional, Batista, Nathália V., additional, Queiroz-Junior, Celso M., additional, Vieira, Angelica T., additional, Menezes, Gustavo B., additional, Sousa, Lirlândia P., additional, van de Loo, Fons A. J., additional, Teixeira, Mauro M., additional, Amaral, Flávio A., additional, and Ferreira, Adaliene V. M., additional

Published
2016
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2. Nanocomposite Treatment Reduces Disease and Lethality in a Murine Model of Acute Graft-versus-Host Disease and Preserves Anti-Tumor Effects

Author

Bernardes, Priscila T. T., primary, Rezende, Bárbara M., additional, Resende, Carolina B., additional, De Paula, Talles P., additional, Reis, Alesandra C., additional, Gonçalves, William A., additional, Vieira, Elias G., additional, Pinheiro, Maurício V. B., additional, Souza, Danielle G., additional, Castor, Marina G. M., additional, Teixeira, Mauro M., additional, and Pinho, Vanessa, additional

Published
2015
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3. Lithothamnion muelleri Treatment Ameliorates Inflammatory and Hypernociceptive Responses in Antigen-Induced Arthritis in Mice

Author

Costa, Vivian V., primary, Amaral, Flavio A., additional, Coelho, Fernanda M., additional, Queiroz-Junior, Celso M., additional, Malagoli, Bruna G., additional, Gomes, Jose Hugo S., additional, Lopes, Fernando, additional, Silveira, Kátia D., additional, Sachs, Daniela, additional, Fagundes, Caio T., additional, Tavares, Lívia D., additional, Pinho, Vanessa, additional, Silva, Tarcilia A., additional, Teixeira, Mauro M., additional, Braga, Fernão C., additional, and Souza, Danielle G., additional

Published
2015
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5. Treadmill Exercise Induces Neutrophil Recruitment into Muscle Tissue in a Reactive Oxygen Species-Dependent Manner. An Intravital Microscopy Study

Author

Nunes-Silva, Albená, primary, Bernardes, Priscila T. T., additional, Rezende, Bárbara M., additional, Lopes, Fernando, additional, Gomes, Elisa C., additional, Marques, Pedro E., additional, Lima, Paulo M. A., additional, Coimbra, Cândido C., additional, Menezes, Gustavo B., additional, Teixeira, Mauro M., additional, and Pinho, Vanessa, additional

Published
2014
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8. Induction of Eosinophil Apoptosis by the Cyclin-Dependent Kinase Inhibitor AT7519 Promotes the Resolution of Eosinophil-Dominant Allergic Inflammation

Author

Alessandri, Ana L., primary, Duffin, Rodger, additional, Leitch, Andrew E., additional, Lucas, Christopher D., additional, Sheldrake, Tara A., additional, Dorward, David A., additional, Hirani, Nik, additional, Pinho, Vanessa, additional, de Sousa, Lirlândia Pires, additional, Teixeira, Mauro M., additional, Lyons, John F., additional, Haslett, Christopher, additional, and Rossi, Adriano G., additional

Published
2011
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10. Role of the chemokine receptors CCR1, CCR2 and CCR4 in the pathogenesis of experimental dengue infection in mice

Author

Mauro M. Teixeira, Rafael Elias Marques, Bernhard Ryffel, Anne-Gaelle Besnard, Danielle G. Souza, Caio T. Fagundes, Rodrigo Guabiraba, Immunologie et Embryologie Moléculaires (IEM), Université d'Orléans (UO)-Centre National de la Recherche Scientifique (CNRS), Fondation pour la Recherche Medicale en France (FRM), Centre Nationale de la Recherche Scientifique (CNRS, France), Conselho Nacional de Deselvovimento Cientifico e Tecnologico (CNPq, Brazil), and Teixeira, Mauro M.

Subjects
CCR1, Male, CCR2, Chemokine, Viral Diseases, lcsh:Medicine, Pharmacologie, Dengue virus, medicine.disease_cause, Ligands, chémokine, Dengue, Chemokine receptor, Mice, 0302 clinical medicine, Immunologie, CCL17, lcsh:Science, 0303 health sciences, Multidisciplinary, biology, Alanine Transaminase, Innate Immunity, 3. Good health, Host-Pathogen Interaction, Infectious Diseases, Cytokines, Medicine, [SDV.IMM]Life Sciences [q-bio]/Immunology, Research Article, Receptors, CCR4, Receptors, CCR2, Immunology, Receptors, CCR1, Immunopathology, Microbiology, CCL5, 03 medical and health sciences, Interferon-gamma, Virology, medicine, Animals, Aspartate Aminotransferases, Biology, Immunity to Infections, 030304 developmental biology, Peroxidase, Pharmacology, Inflammation, Interleukin-6, lcsh:R, Immunity, pays tropical, Dengue Virus, infection, Mice, Inbred C57BL, Immune System, biology.protein, lcsh:Q, CC chemokine receptors, problème de santé publique, 030215 immunology
Abstract

Guabiraba, Rodrigo Marques, Rafael Elias Besnard, Anne-Gaelle Fagundes, Caio T Souza, Danielle G Ryffel, Bernhard Teixeira, Mauro M PLoS One. 2010 Dec 29;5(12):e15680. doi: 10.1371/journal.pone.0015680.; International audience; Dengue virus (DENV), a mosquito-borne flavivirus, is a public health problem in many tropical countries. Recent clinical data have shown an association between levels of different chemokines in plasma and severity of dengue. We evaluated the role of CC chemokine receptors CCR1, CCR2 and CCR4 in an experimental model of DENV-2 infection in mice. Infection of mice induced evident clinical disease and tissue damage, including thrombocytopenia, hemoconcentration, lymphopenia, increased levels of transaminases and pro-inflammatory cytokines, and lethality in WT mice. Importantly, infected WT mice presented increased levels of chemokines CCL2/JE, CCL3/MIP-1α and CCL5/RANTES in spleen and liver. CCR1⁻/⁻ mice had a mild phenotype with disease presentation and lethality similar to those of WT mice. In CCR2⁻/⁻ mice, lethality, liver damage, levels of IL-6 and IFN-γ, and leukocyte activation were attenuated. However, thrombocytopenia, hemoconcentration and systemic TNF-α levels were similar to infected WT mice. Infection enhanced levels of CCL17/TARC, a CCR4 ligand. In CCR4⁻/⁻ mice, lethality, tissue injury and systemic inflammation were markedly decreased. Despite differences in disease presentation in CCR-deficient mice, there was no significant difference in viral load. In conclusion, activation of chemokine receptors has discrete roles in the pathogenesis of dengue infection. These studies suggest that the chemokine storm that follows severe primary dengue infection associates mostly to development of disease rather than protection.

Published
2010
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