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Start Over Author "Yahyaoui M" Journal revue neurologique
128 results on '"Yahyaoui M"'

39. Hypertension intracrânienne bénigne

Author

Errguig, L., primary, Benomar, A., additional, Aitbenhaddou, E., additional, Mouti, O., additional, Regragui, W., additional, Benaaboud, B., additional, Yahyaoui, M., additional, and Chkili, T., additional

Published
2004
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46. [Multiple sclerosis associated with antiphospholipid syndrome: diagnostic and therapeutic difficulties].

Author

Ahbeddou N, Ait Ben Haddou E, Hammi S, Slimani C, Regragui W, Benomar A, and Yahyaoui M

Subjects
Anti-Inflammatory Agents therapeutic use, Antiphospholipid Syndrome diagnosis, Brachial Plexus Neuritis etiology, Brachial Plexus Neuritis therapy, Brain pathology, Facial Paralysis etiology, Facial Paralysis therapy, Female, Humans, Image Processing, Computer-Assisted, Magnetic Resonance Imaging, Methylprednisolone therapeutic use, Middle Aged, Multiple Sclerosis diagnosis, Optic Neuritis etiology, Optic Neuritis therapy, Paraparesis etiology, Paraparesis therapy, Vision Tests, Antiphospholipid Syndrome complications, Antiphospholipid Syndrome therapy, Multiple Sclerosis complications, Multiple Sclerosis therapy
Abstract

Strokes are the main neurological manifestation of antiphospholipid syndrome. Other clinical presentations are possible and may mimic classic symptoms of multiple sclerosis (MS). A 46-year-old woman, with a history of two miscarriages, presented four subacute neurological episodes (optic neuritis, right facial paralysis, paraparesis of the thigh, and right brachial monoparesis). Using McDonald criteria, the diagnosis of multiple sclerosis was retained. Because of the occurrence of thrombocytopenia during a final relapse, we reconsidered the diagnosis of MS. Search for antiphospholipid antibodies was positive. All clinical manifestations and complementary tests were compatible with the diagnosis of antiphospholipid syndrome associated with multiple sclerosis. Given the great similarity of clinical, radiological and biological findings in the two diseases, non-thrombotic neurological manifestations of antiphospholipid syndrome can be difficult to distinguish from MS associated with antiphospholipid syndrome., (Copyright © 2011 Elsevier Masson SAS. All rights reserved.)

Published
2012
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47. [Psychotic disorder revealing epilepsy linked to a dysembryoma of the left hippocampus].

Author

Belaizi M, Mehssani J, Yahyaoui M, and Fajri A

Subjects
Acute Disease, Adult, Anticonvulsants therapeutic use, Antipsychotic Agents therapeutic use, Diagnosis, Differential, Electroencephalography, Epilepsies, Partial drug therapy, Epilepsies, Partial etiology, Hallucinations etiology, Hippocampus physiopathology, Humans, Magnetic Resonance Imaging, Male, Paranoid Disorders etiology, Psychomotor Agitation etiology, Psychotic Disorders drug therapy, Supratentorial Neoplasms complications, Teratoma complications, Valproic Acid therapeutic use, Epilepsies, Partial diagnosis, Hippocampus pathology, Psychotic Disorders etiology, Supratentorial Neoplasms diagnosis, Teratoma diagnosis
Abstract

Introduction: Psychotic symptoms are a rare but well-known complication of epilepsy. The prevalence is estimated between 4 and 9%., Patient: We report a case of a 40-year-old patient, unrecognized epileptic, who presented an acute psychotic syndrome which seemed to be of functional origin, the EEG performed during the episode, and the cerebral CT scan being normal. Nevertheless, the clinical presentation, especially the sudden ending of delusions, led to further investigations. Careful history taking and repeated EEG recordings allowed the diagnosis of partial epilepsy that had begun 17 years earlier and symptomatic of a dysembryoplastic tumour of the left hippocampus revealed by MRI., Discussion and Conclusion: Search for an epileptic origin of an acute psychotic syndrome must always be undertaken by systematic EEG. The possibility of a symptomatic temporal tumor must not be overlooked., (Copyright © 2010 Elsevier Masson SAS. All rights reserved.)

Published
2011
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48. [Stroke revealing granulomatosis].

Author

Raissouni L, Aitbenhaddou E, Ahbeddou N, El Alaoui KT, Slimani C, Benomar A, and Yahyaoui M

Subjects
Abducens Nerve Diseases etiology, Adult, Antibodies, Antineutrophil Cytoplasmic blood, Cerebral Cortex blood supply, Corpus Striatum blood supply, Cyclophosphamide therapeutic use, Exophthalmos etiology, Female, Granulomatosis with Polyangiitis complications, Granulomatosis with Polyangiitis drug therapy, Granulomatosis with Polyangiitis immunology, Humans, Immunosuppressive Agents therapeutic use, Magnetic Resonance Imaging, Methylprednisolone therapeutic use, Nasal Obstruction etiology, Nerve Compression Syndromes etiology, Optic Nerve Diseases etiology, Paresis etiology, Brain Ischemia etiology, Granulomatosis with Polyangiitis diagnosis
Abstract

Introduction: Central nervous system involvement is rare in Wegener granulomatosis. Stroke is the most common event suggestive of the disease., Comment: A 35-year-old woman, who was followed for rhinitis and mild asthma, described gradual decline of visual acuity in the right eye over two months, persistent nasal obstruction and fronto-orbital headache since a few weeks. She presented left hemiparesis due to a stroke, associated with exophthalmos and deficits of the optic nerve and abducens of the right eye. The otolaryngological examination found signs of crusty rhinitis and right nasal stenosis. The diagnosis of Wegener's granulomatosis was established on the basis of the clinical findings, radiological aspects and the presence of ANCA. The patient was treated by antiplatelet agents and high-dose corticosteroids associated with immunosuppressive drugs including cyclophosphamide in a monthly bolus., Discussion: This case illustrates two of the three pathogenic mechanisms that may account for central nervous system involvement in Wegener granulomatosis: vasculitis, extension by contiguity of granulomatous tissue from the nasal cavity or sinuses, and in situ formation of a granuloma into the brain parenchyma or meninges., (Copyright © 2010 Elsevier Masson SAS. All rights reserved.)

Published
2011
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49. [Cerebral venous thrombosis and acute polyradiculoneuritis revealing systemic lupus erythematosus].

Author

Ahbeddou N, Benomar A, Rasmouni K, Quessar A, Ouhabi H, Ait Ben Haddou E, and Yahyaoui M

Subjects
Adult, Cranial Sinuses pathology, Electrodiagnosis, Female, Humans, Lupus Erythematosus, Systemic complications, Magnetic Resonance Imaging, Motor Neurons physiology, Neural Conduction physiology, Polyradiculoneuropathy complications, Prognosis, Sensory Receptor Cells physiology, Sinus Thrombosis, Intracranial complications, Lupus Erythematosus, Systemic pathology, Polyradiculoneuropathy pathology, Sinus Thrombosis, Intracranial pathology
Abstract

Introduction: Neurological manifestations of systemic lupus erythematosus are frequent and polymorphic. In 40% of cases, lupus can be revealed by neurological symptoms. Cerebral nervous system complications predominate and can be a negative factor for prognosis. Peripheral features are rare and various and can compromise functional prognosis, sometimes with fatal outcome., Case Report: We report the case of a 30-year-old woman who presented a cerebral venous thrombosis of the superior longitudinal sinus. Outcome was favorable with antibiotics and anticoagulants. Four months later, she developed an acute polyradiculoneuritis associated with an inflammatory syndrome and positive tests for antinuclear antibody and antinuclear anti-DNA. The diagnosis of neurolupus was retained on the basis of four criteria of the American college of Rheumatology. The patient was given steroid therapy associated with a course of intravenous immunoglobulin. She has fully recovered her deficit., Conclusion: Cerebral venous thrombosis and acute polyradiculonévrites are rare events in systemic lupus erythematosus. Early diagnosis and management are crucial., (2009 Elsevier Masson SAS. All rights reserved.)

Published
2010
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50. [Primary Gougerot-Sjogren's syndrome presenting as a cerebellar syndrome].

Author

Azeroual A, Benomar A, Harmouche H, Daoudi N, Benhaddou EH, Yahyaoui M, and Maaouni A

Subjects
Adrenal Cortex Hormones therapeutic use, Anti-Inflammatory Agents therapeutic use, Biopsy, Cerebellar Diseases diagnosis, Cyclophosphamide therapeutic use, Humans, Immunoelectrophoresis, Immunoglobulin G immunology, Immunosuppressive Agents therapeutic use, L-Lactate Dehydrogenase blood, Magnetic Resonance Imaging, Male, Middle Aged, Salivary Glands pathology, Sjogren's Syndrome diagnosis, Cerebellar Diseases pathology, Sjogren's Syndrome pathology
Abstract

Primary Sjogren's syndrome is an autoimmune exocrinopathy with various extraglandular complications. Central neurological manifestations were described in 20 percent of cases. Cerebellar syndrome is uncommon in Sjogren's syndrome, but can occur after diagnosis, or rarely as an inaugural sign of the disease. We report the case of a 54-year-old man who presented a cerebellar syndrome with xerostomy and xerophthalmos. The diagnosis of primary Sjorgren's syndrome was confirmed on the accessory salivary gland biopsy. The patient received oral and intravenous corticosteroid therapy associated with cyclophosphamide monthly. Outcome was good.

Published
2007
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