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Some clinical studies report that I T i SB Br sb increases after brain injury and some patients undergo interventions to achieve a "normal" I T i SB Br sb . If I T i SB Br sb rhythms are similarly affected, it will be critical to establish how normal I T i SB Br sb variation interacts with the daily neural molecular clockwork before inferring a role for I T i SB Br sb disruption in disease. Daily variations in I T i SB Br sb are demonstrable in rodents and non-human primates; in the latter, I T i SB Br sb is consistently higher than carotid artery, aortic arch, and abdominal cavity temperature, and exhibits its own spatial gradient [[1]]. To address this gap, we undertook a retrospective analysis of I T i SB Br sb measured directly in patients with traumatic brain injury, alongside a prospective study of I T i SB Br sb measured non-invasively in healthy adults using magnetic resonance spectroscopy (MRS) [[1]]. [Extracted from the article]