30 results on '"Li, Guangbi"'
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2. Attenuation of Obesity‐Induced Glomerular Inflammatory Response by Inhibition of Exosome Biogenesis and Release without Changes in NLRP3 Inflammasome Activation in Podocytes
3. Characterization and Ischemic Response of Telecytes in the Mouse Renal Medulla
4. Autophagic Deficiency and Dedifferentiation in Podocytes of Mice Lacking Acid Ceramidase (Asah1) Gene
5. Exosome‐Mediated Release of NLRP3 Inflammasome Products from Podocytes to Trigger Glomerular Inflammatory Response During Obesity
6. Antihypertensive Action of Exosomes Released from the Renal Medulla after Unclipping of the Renal Artery of 2 Kidney‐1 Clip Hypertension Mice
7. Control of TRPML1 Channel Activity and Lysosome Trafficking by Acid Ceramidase in Mouse Coronary Arterial Endothelial Cells
8. Redox Regulation of Lysosome‐MVB Interaction and Inflammatory Exosome Release during NLRP3 Inflammasome Activation by High Homocysteine in Mouse Podocytes
9. Subendothelial Accumulation of Exosomes and Coronary Microvascular Dysfunction in Mice Lacking Acid Ceramidase
10. Podocyte Injury and Glomerular Inflammation Induced by Urinary Exosomes from Hyperhomocysteinemic Mice
11. Inflammasome Activation and Exosome Release in the Lung during Chronic Obstructive Pulmonary Disease: Potential Therapeutic Target of Acupuncture
12. Contribution of NLRP3 Inflammasome Activation to Glomerular Injury during Hyperhomocysteinemia with and without Enhanced Exosome Secretion
13. Amelioration of Podocytopathy by Smpd1 Gene Deletion in Podocyte‐Specific Asah1 Gene Knockout Mice
14. Control of Instant Membrane Resealing by Lysosomal Acid Ceramidase in Endothelial Cells
15. Lysosome‐MVB Encountering and Inflammatory Exosome Release Controlled by Acid Sphingomyelinase in Podocytes during Hyperhomocysteinemia
16. Regulatory Role of Ceramide/mTOR Signaling in Exosome Secretion from Smooth Muscle Cells during Arterial Stiffening and Medial Calcification
17. Enhanced Exosome Release and Inhibited TRPML1 Channel Activity in Podocytes from Mice with Podocyte‐Restricted Deletion of Asah1 Gene
18. Podocyte‐Specific Rescuing and Deletion of Acid Ceramidase Gene in Mouse Glomeruli
19. Ceramide‐Mediated Enhancement of Inflammatory Exosome Secretion from Podocytes during Hyperhomocysteinemia
20. Redox Regulation of TRPML1 Channel Activity and Lysosome Trafficking in Podocytes
21. Increased Podocyte Exosome Release in Glomerular Injury induced by NLRP3 Inflammasome Activation during Hyperhomocysteinemia
22. D‐Ribose Induced NLRP3 Inflammasome Activation in Podocytes by Increase in Advanced Glycation End‐Products
23. Podocyte‐Specific Deletion of Asah1 Gene Produced Podocytopathy without Sclerotic Pathology: A Potential Mouse Model for Steroid‐Insensitive Minimal Change Disease
24. Lysosomal Ca 2+ Release via TRPML1 Channels Regulated by Acid Ceramidase and Associated Sphingolipids in Podocytes
25. Pannexin‐1 Channel Activity and Associated ATP Release in Podocytes in Response to Adipokines
26. Prostamide‐Mediated Protective Action of Anandamide on Nlrp3 Inflammasome Activation in Glomeruli of Mice with Hyperhomocysteinemia
27. NLRP3 Inflammasome as a Novel Target for Docosahexaenoic Acid and Its Metabolites to Abrogate Glomerular Injury during Hyperhomocysteinemia
28. Enhanced Epithelial‐to‐Mesenchymal Transition Associated with Lysosome Dysfunction in Podocytes: Role of p62/Sequestosome 1 as a Signaling Hub
29. Prevention of High Fat‐induced Podocyte Injury and Glomerular Sclerosis in Mice Lacking Nod‐like Receptor Protein 3: Role of Inflammasome Extinction
30. Enhanced dedifferentiation and injury in mouse podocytes lacking CD38 gene (691.4)
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