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Background: Hispanics are the fastest growing minority group in the United States; however, minimal information is available on their cancer risks from exposures to hazardous air pollutants (HAPs) and how these risks compare to risks to non-Hispanic whites. Methods: We estimated the personal exposure and cancer risk of Hispanic and white adults who participated in the Relationships of Indoor, Outdoor, and Personal Air (RIOPA) study. We evaluated 12 of the sampled volatile organic compounds and carbonyls and identified the HAPs of most concern and their possible sources. Furthermore, we examined sociodemographic factors and building characteristics. Results: Cumulative cancer risks (CCRs) estimated for Hispanics (median = 519 × 10-6, 90th percentile = 3,968 × 10-6) and for whites (median = 443 × 10-6, 90th percentile = 751 × 10-6) were much greater than the U.S. Environmental Protection Agency (EPA) benchmark of 10-6. Cumulative risks were dominated by formaldehyde and p-dichlorobenzene (p-DCB) and, to a lesser extent, by acetaldehyde, chloroform, and benzene. Exposure to all of these compounds except benzene was primarily due to indoor residential sources. Hispanics had statistically higher CCRs than did whites (p ⩽ 0.05) because of differences in exposure to p-DCB, chloroform, and benzene. Formaldehyde was the largest contributor to CCR for 69% of Hispanics and 88% of whites. Cancer risks for pollutants emitted indoors increased in houses with lower ventilation rates. Conclusions: Hispanics appear to be disproportionately affected by certain HAPs from indoor and outdoor sources. Policies that aim to reduce risk from exposure to HAPs for the entire population and population subgroups should consider indoor air pollution. [ABSTRACT FROM AUTHOR]

We measured volatile organic compound (VOC) exposures in multiple locations for a diverse population of children who attended two inner-city schools in Minneapolis, Minnesota. Fifteen common VOCs were measured at four locations: outdoors (0), indoors at school (S), indoors at home (H), and in personal samples (P). Concentrations of most VOCs allowed the general pattern O &ap; S < P < H across the measured microenvironments. The S and O environments had the smallest and H the largest influence on personal exposure to most compounds. A time-weighted model of P exposure using all measured microenvironments and time-activity data provided little additional explanatory power beyond that provided by using the H measurement alone. Although H and P concentrations of most VOCs measured in this study were similar to or lower than levels measured in recent personal monitoring studies of adults and children in the United States, p-dichlorobenzene was the notable exception to this pattern, with upper-bound exposures more than 100 times greater than those found in other studies of children. Median and upper-bound H and P exposures were well above health benchmarks for several compounds, so outdoor measurements likely underestimate long-term health risks from children's exposure to these compounds. [ABSTRACT FROM AUTHOR]