141 results on '"Bezzerri, Valentino"'
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2. COVID‐19 disease in patients with chronic neutropenia: The experience from the European Network for Innovative Diagnosis and Treatment of Chronic Neutropenias.
3. Growth Charts for Shwachman–Diamond Syndrome at Ages 0 to 18 Years.
4. Shwachman-Diamond Syndrome: Molecular Mechanisms and Current Perspectives
5. Does cystic fibrosis constitute an advantage in COVID-19 infection?
6. Is cellular senescence involved in cystic fibrosis?
7. The Molecular and Genetic Mechanisms of Inherited Bone Marrow Failure Syndromes: The Role of Inflammatory Cytokines in Their Pathogenesis.
8. Counteracting the Common Shwachman–Diamond Syndrome-Causing SBDS c.258+2T>C Mutation by RNA Therapeutics and Base/Prime Editing.
9. Immunogenicity and Safety of the BNT162b2 COVID-19 Vaccine in Patients with Cystic Fibrosis with or without Lung Transplantation.
10. Pseudomonas aeruginosa reduces the expression of CFTR via post-translational modification of NHERF1
11. Uptake by human glioma cell lines and biological effects of a peptide-nucleic acids targeting miR-221
12. Anti-inflammatory effect of miglustat in bronchial epithelial cells
13. Expression of microRNA-93 and Interleukin-8 during Pseudomonas aeruginosa-Mediated Induction of Proinflammatory Responses
14. Novel Translational Read-through–Inducing Drugs as a Therapeutic Option for Shwachman-Diamond Syndrome.
15. Effects of decoy molecules targeting NF-kappaB transcription factors in Cystic fibrosis IB3–1 cells: Recruitment of NF-kappaB to the IL-8 gene promoter and transcription of the IL-8 gene
16. Modulators of Sphingolipid Metabolism Reduce Lung Inflammation
17. A polymorphism in the 5′ UTR of the DEFB1 gene is associated with the lung phenotype in F508del homozygous Italian cystic fibrosis patients
18. Decoy oligodeoxyribonucleotides and peptide nucleic acids-DNA chimeras targeting nuclear factor kappa-B: Inhibition of IL-8 gene expression in cystic fibrosis cells infected with Pseudomonas aeruginosa
19. Study of Angiogenic Potential and Oxidative Metabolism in Mesenchymal Stromal Cells Derived from Shwachman-Diamond Syndrome Patients
20. Deletion of eif6 Partially Rescues Survival, but Not Neutropenia, of Zebrafish Model of Shwachman-Diamond Syndrome: The Roles of Sbds and Eif6 in Perturbing Lipid Metabolism
21. Pyrogallol, an active compound from the medicinal plant Emblica officinalis, regulates expression of pro-inflammatory genes in bronchial epithelial cells
22. Transcription Factor Oligodeoxynucleotides to NF-κB Inhibit Transcription of IL-8 in Bronchial Cells
23. MPB-07 Reduces the Inflammatory Response to Pseudomonas aeruginosa in Cystic Fibrosis Bronchial Cells
24. PLCB3 Loss-of-function Reduces P. aeruginosa-dependent IL-8 Release in Cystic Fibrosis
25. Proinflammatory signal transduction in epithelial cells: the model of cystic fibrosis lung disease
26. Effects of analogues of 4,6,4'-trymethylangelicin on erythroid differentiation and NF-kB activity
27. Ataluren‐driven restoration of Shwachman‐Bodian‐Diamond syndrome protein function in Shwachman‐Diamond syndrome bone marrow cells.
28. PSEUDOMONAS AERUGINOSA MODULATES THE PHOSPHORYLATION CHANGES OF SCAFFOLDING PROTEINS AND APICAL EXPRESSION OF CFTR IN AIRWAYS
29. IMINOSUGAR-BASED INHIBITORS OF CERAMIDE GLUCOSYL-TRANSFERASE (GLCCERT) AND NONLYSOSOMAL GLUCOSYLCERAMIDASE (GBA2) REDUCE THE INFLAMMATORY RESPONSE TO P. AERUGINOSA IN CF BRONCHIAL EPITHELIAL CELLS
30. Phospholipase C- 3 Is a Key Modulator of IL-8 Expression in Cystic Fibrosis Bronchial Epithelial Cells
31. PLCB3 cooperates with the Toll-like receptors' signaling cascade enhancing P.aeruginosa-dependent IL-8 expression in bronchial epithelial cells
32. MPB-07 Reduces the Inflammatory Response to Pseudomonas aeruginosa in CF Bronchial Cells
33. Differential Effects of Angelicin Analogues on NF-κB Activity and IL-8 Gene Expression in Cystic Fibrosis IB3-1 Cells.
34. An antisense peptide nucleic acid against Pseudomonas aeruginosa inhibiting bacterial-induced inflammatory responses in the cystic fibrosis IB3-1 cellular model system.
35. β-Sitosterol Reduces the Expression of Chemotactic Cytokine Genes in Cystic Fibrosis Bronchial Epithelial Cells.
36. Transient Receptor Potential Ankyrin 1 Channels Modulate Inflammatory Response in Respiratory Cells from Patients with Cystic Fibrosis.
37. Further Insights into the Hematological Disorders Observed in Shwachman-Diamond Syndrome: mTOR and STAT3 Hyper-Phosphorylation in CD4+ and CD8+ T Cells and Efficacy of Rapamycin Treatment
38. Regulation of IL-8 gene expression in gliomas by microRNA miR-93.
39. Antibacterial and anti-inflammatory activity of a temporin B peptide analogue on an in vitro model of cystic fibrosis.
40. GBA2-Encoded β-Glucosidase Activity Is Involved in the Inflammatory Response to Pseudomonas aeruginosa.
41. Trimethylangelicin promotes the functional rescue of mutant F508del CFTR protein in cystic fibrosis airway cells.
42. Effects of decoy molecules targeting NFkappaB transcription factors in cystic fibrosis IB3-1 cells.
43. Mapping the Transcriptional Machinery of the IL-8 Gene in Human Bronchial Epithelial Cells.
44. Phospholipase C-β3 Is a Key Modulator of IL-8 Expression in Cystic Fibrosis Bronchial Epithelial Cells.
45. Trimethylangelicin reduces IL-8 transcription and potentiates CFTR function.
46. Virtual Screening against p50 NF-κB Transcription Factor for the Identification of Inhibitors of the NF-κB-DNA Interaction and Expression of NF-κB Upregulated Genes.
47. Enhanced p53 Levels Are Involved in the Reduced Mineralization Capacity of Osteoblasts Derived from Shwachman–Diamond Syndrome Subjects.
48. Docking of molecules identified in bioactive medicinal plants extracts into the p50 NF-kappaB transcription factor: correlation with inhibition of NF-kappaB/DNA interactions and inhibitory effects on IL-8 gene expression.
49. Induction of IL-6 gene expression in a CF bronchial epithelial cell line by Pseudomonas aeruginosa is dependent on transcription factors belonging to the Sp1 superfamily
50. Nonsense Suppression Therapy: New Hypothesis for the Treatment of Inherited Bone Marrow Failure Syndromes.
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