Search

Your search keyword '"Bhattacharyya, P. N."' showing total 60 results
Start Over Author "Bhattacharyya, P. N." Language english

Refine your results

60 results on '"Bhattacharyya, P. N."'

24. Abstracts

Author

Basu, J., Rohatgi-Mukherjee, K. K., Chatterjee, S., Sarkar, S., Bhattacharyya, S. N., Dasmahapatra, G. K., Wolff, Thomas, Von Bunau, Gunther, Dhawan, S. N., Sen, Dipankar, Bera, Subhash Ch, Gupta, S. C., Yadav, N. S., Sahu, Manabendra, Saha, Manjubikash, Masilamani, V., Sastikumar, D., Behera, Pradipta Kumar, Mishra, Ashok Kumar, Muthusamy, S., Ramakrishnan, V. T., Ramamurthy, P., Sampath, S., Narayan, R., Manickam, M. C. Durai, Srinivasan, C., Pitchumani, K., Roja, M., Rajagopal, S., Ray, Reba, Mukherjee, Samaresh, Samanta, A., Natarajan, E., Selvaraj, S., Natarajan, P., Talwar, S. S., Viswanathan, K., Natarajan, P., Dhathathreyan, Aruna, Dhathathreyan, K. S., Biswal, Basanti, Song, Pill-Soon, Das, S., Muneer, M., Haridas, A., Manilal, V. B., Jayakumar, R., Ramasami, T., Julliard, M., Chanon, M., Joshi, P. N., Biswal, U. C., Joshy, C. L., Purushothaman, E., Maiya, Bhaskar G., Ganesan, S., Nagarajan, B., Narayanan, V., Mohanty, Prasanna, Shubin, V., Murthy, S. D. S., Karapetyan, N. V., Ramaraj, R., Gobi, K. V., Sankareswari, V. Gomathi, Ranganayaki, S., Bahadur, K., Kumar, Sajal, Sonawane, H. R., Kulkarni, D. G., Ayyangar, N. R., Sukumar, N., Venkitachalam, T. V., Rao, A. S., Ali, S. I., Kaur, Harpal, Anbalagan, V., Srivastava, T. S., Kumar, Awadhesh, Chowdhury, P. K., Rao, K. V. S. Rama, Mittal, J. P., Balasubramanian, N., Balasubramanian, S., Dutt, G. B., Doraiswamy, S., Manna, Bidyut Kumar, Rohatgi-Mukherjee, K. K., Chandra, A., Bagchi, B., Arunachalam, S., Devadason, J. R., Dhanasekaran, T., Prabahar, K. Joseph, Mathew, Lizamma, Ramakrishnan, V., Rajaram, J., Rao, V. R. S., Narayanan, P., Pillai, V. N. Sivasankara, Priyadarsini, K. I., Rajendran, T., Palaniandavar, M., Kaneko, M., Kira, A., Sidhu, M. S., Chopra, Arti, Gill, Barinder, Baby, Thomas, and Nampoori, V. P. N.

Published
1992
Full Text
View/download PDF

32. Microbial biocides - Viable alternatives to chemicals for tea disease management.

Author

SARMAH, S. R., BHATTACHARYYA, P. N., and BAROOAH, A. K.

Subjects
BIOCIDES, CONTROL of phytopathogenic microorganisms, FUSARIUM solani, TEA plantations, FUNGICIDES, CROP yields
Abstract

Tea plantations in North east India are mostly rain fed and the cropping season needs moist climate with alternating wet and dry periods that eventually attracts diverse tea pathogens like Fusarium solani, Cephaleuros spp., Corticium spp. Poria hypobrunnea, Ustulina zonata, Fomes lamaoensis etc., known to cause several serious diseases in tea and thereby make enormous losses to crop yield and quality, influencing world tea market, adversely. Tea disease management using chemical fungicides alone is, however, prohibitive since the latter is known for destabilizing soil fertility and thereby directly affects the native microbial populations present in soil. Application of new fungicide molecules at its low doses along with exploitation of biological substances (more especially, microbial biocontrol agents) and adoption of Integrated Disease Management (IDM) schedule has been practiced in the present investigation for controlling certain devastating diseases in tea plantation of North East India. Multilocational field trials has been made throughout the investigations using locally isolated microorganisms such as Azotobacter, Azospirillum, Bacillus, Pseudomonas, Streptomyces, Trichoderma etc. (@ 2%, 5% and 10% spore concentrations), along with crude extracts of botanicals (@ 5% concentration) prepared from Amphineuron opulentum, Cleome gynandra, Ipomea convolvulus, Polygonum hydropiper etc. against dominant tea diseases such as black rot, red rust and Fusarium die back respectively. Maximum emphasis has been made to reduce the load of chemicals and move towards alternative strategies of tea disease management as to promote sustainable tea cultivation. Biological control measures (especially microbial) have potential to reduce the disease severity (up to 78.2% disease reduction) and thereby brought impending perspective for the use of effective biopesticides in tea ecosystem. The potential microbial strains were identified using morphological, biochemical as well as molecular characterization and deposited at NCBI GenBank database with accession numbers. The application technology of microbial biocides has also being popularized among the tea growers throughout the region to accelerate the use of these beneficial microorganisms in tea. [ABSTRACT FROM AUTHOR]

Published
2020
Full Text
View/download PDF

49. Obstacles and opportunities in the functional analysis of extracellular vesicle RNA – an ISEV position paper

Author

Mateescu, Bogdan, Kowal, Emma J. K., van Balkom, Bas W. M., Bartel, Sabine, Bhattacharyya, Suvendra N., Buzás, Edit I., Buck, Amy H., de Candia, Paola, Chow, Franklin W. N., Das, Saumya, Driedonks, Tom A. P., Fernández-Messina, Lola, Haderk, Franziska, Hill, Andrew F., Jones, Jennifer C., Van Keuren-Jensen, Kendall R., Lai, Charles P., Lässer, Cecilia, Liegro, Italia di, Lunavat, Taral R., Lorenowicz, Magdalena J., Maas, Sybren L. N., Mäger, Imre, Mittelbrunn, Maria, Momma, Stefan, Mukherjee, Kamalika, Nawaz, Muhammed, Pegtel, D. Michiel, Pfaffl, Michael W., Schiffelers, Raymond M., Tahara, Hidetoshi, Théry, Clotilde, Tosar, Juan Pablo, Wauben, Marca H. M., Witwer, Kenneth W., and Nolte-‘t Hoen, Esther N. M.

Subjects
Article, Extracellular vesicles, exosomes, non-coding RNA, mRNA, function, sorting, RNA binding proteins, quantification
Abstract

The release of RNA-containing extracellular vesicles (EV) into the extracellular milieu has been demonstrated in a multitude of different in vitro cell systems and in a variety of body fluids. RNA-containing EV are in the limelight for their capacity to communicate genetically encoded messages to other cells, their suitability as candidate biomarkers for diseases, and their use as therapeutic agents. Although EV-RNA has attracted enormous interest from basic researchers, clinicians, and industry, we currently have limited knowledge on which mechanisms drive and regulate RNA incorporation into EV and on how RNA-encoded messages affect signalling processes in EV-targeted cells. Moreover, EV-RNA research faces various technical challenges, such as standardisation of EV isolation methods, optimisation of methodologies to isolate and characterise minute quantities of RNA found in EV, and development of approaches to demonstrate functional transfer of EV-RNA in vivo. These topics were discussed at the 2015 EV-RNA workshop of the International Society for Extracellular Vesicles. This position paper was written by the participants of the workshop not only to give an overview of the current state of knowledge in the field, but also to clarify that our incomplete knowledge – of the nature of EV(-RNA)s and of how to effectively and reliably study them – currently prohibits the implementation of gold standards in EV-RNA research. In addition, this paper creates awareness of possibilities and limitations of currently used strategies to investigate EV-RNA and calls for caution in interpretation of the obtained data.

Published
2017
Full Text
View/download PDF

50. Leishmania donovani Targets Dicer1 to Downregulate miR-122, Lower Serum Cholesterol, and Facilitate Murine Liver Infection.

Author

Ghosh, June, Bose, Mainak, Roy, Syamal, and Bhattacharyya, Suvendra N.

Subjects
RNA helicase genetics, LEISHMANIA donovani, GENETIC regulation, BLOOD cholesterol, VISCERAL leishmaniasis, PARASITIC diseases, LIVER diseases, LABORATORY mice
Abstract

Summary: Leishmania donovani causes visceral leishmaniasis (VL) where the parasite infects and resides inside liver and spleen tissue macrophages. Given the abnormal lipid profile observed in VL patients, we examined the status of serum lipids in an experimental murine model of VL. The murine VL liver displayed altered expression of lipid metabolic genes, many of which are direct or indirect targets of the liver-specific microRNA-122. Concomitant reduction of miR-122 expression was observed in VL liver. High serum cholesterol caused resistance to L. donovani infection, while downregulation of miR-122 is coupled with low serum cholesterol in VL mice. Exosomes secreted by the infective parasites caused reduction in miR-122 activity in hepatic cells. Leishmania surface glycoprotein gp63, a Zn-metalloprotease, targets pre-miRNA processor Dicer1 to prevent miRNP formation in L. donovani-interacting hepatic cells. Conversely, restoration of miR-122 or Dicer1 levels in VL mouse liver increased serum cholesterol and reduced liver parasite burden. [ABSTRACT FROM AUTHOR]

Published
2013
Full Text
View/download PDF

Catalog

Books, media, physical & digital resources
See catalog results