448 results on '"Forconi, Francesco"'
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2. XPO1 inhibition sensitises CLL cells to NK cell mediated cytotoxicity and overcomes HLA-E expression
3. Network analysis reveals a major role for 14q32 cluster miRNAs in determining transcriptional differences between IGHV-mutated and unmutated CLL
4. Activity of venetoclax in patients with relapsed or refractory chronic lymphocytic leukaemia: analysis of the VENICE-1 multicentre, open-label, single-arm, phase 3b trial
5. The essential microenvironmental role of oligomannoses specifically inserted into the antigen-binding sites of lymphoma cells
6. BTK and PLCG2 remain unmutated in one-third of patients with CLL relapsing on ibrutinib
7. Ibrutinib and rituximab versus fludarabine, cyclophosphamide, and rituximab for patients with previously untreated chronic lymphocytic leukaemia (FLAIR): interim analysis of a multicentre, open-label, randomised, phase 3 trial
8. High surface IgM levels associate with shorter response to ibrutinib and BTK bypass in patients with CLL
9. Characterization of metabolic alterations of chronic lymphocytic leukemia in the lymph node microenvironment
10. Consensus opinion from an international group of experts on measurable residual disease in hairy cell leukemia
11. Targeted inhibition of eIF4A suppresses B-cell receptor-induced translation and expression of MYC and MCL1 in chronic lymphocytic leukemia cells
12. Hairy cell leukemia and COVID-19 adaptation of treatment guidelines
13. Changes in Bcl-2 members after ibrutinib or venetoclax uncover functional hierarchy in determining resistance to venetoclax in CLL
14. Acalabrutinib monotherapy in patients with Waldenström macroglobulinemia: a single-arm, multicentre, phase 2 study
15. DC-SIGN binding to mannosylated B-cell receptors in follicular lymphoma down-modulates receptor signaling capacity
16. Genome-wide association study identifies risk loci for progressive chronic lymphocytic leukemia
17. Bidirectional linkage between the B-cell receptor and NOTCH1 in chronic lymphocytic leukemia and in Richter’s syndrome: therapeutic implications
18. BCR signaling contributes to autophagy regulation in chronic lymphocytic leukemia
19. Consensus guidelines for the diagnosis and management of patients with classic hairy cell leukemia
20. Development of DNA vaccines against B-cell neoplasms
21. Surface IgM expression and function are associated with clinical behavior, genetic abnormalities, and DNA methylation in CLL
22. IL-4 enhances expression and function of surface IgM in CLL cells
23. Engagement of the B-cell receptor of chronic lymphocytic leukemia cells drives global and MYC-specific mRNA translation
24. Perturbation of the normal immune system in patients with CLL
25. The PI3K/mTOR inhibitor PF-04691502 induces apoptosis and inhibits microenvironmental signaling in CLL and the Eµ-TCL1 mouse model
26. DNA methylation profiling identifies two splenic marginal zone lymphoma subgroups with different clinical and genetic features
27. Stimulation of surface IgM of chronic lymphocytic leukemia cells induces an unfolded protein response dependent on BTK and SYK
28. Correction: Bidirectional linkage between the B-cell receptor and NOTCH1 in chronic lymphocytic leukemia and in Richter’s syndrome: therapeutic implications
29. CLL-638 Latest Results From an Ongoing Firstin-Human Phase 1a/B Study of NX-5948, a Selective Bruton's Tyrosine Kinase (BTK) Degrader, in Patients With Relapsed/Refractory CLL and Other B-Cell Malignancies
30. Latest Results From an Ongoing First-in-Human Phase 1a/B Study of NX-5948, a Selective Bruton's Tyrosine Kinase (BTK) Degrader, in Patients With Relapsed/Refractory CLL and Other B-Cell Malignancies
31. Combination of Ibrutinib Plus Venetoclax with MRD-Driven Duration of Treatment Results in a Higher Rate of MRD Negativity in IGHV Unmutated Than Mutated CLL: Updated Interim Analysis of FLAIR Study
32. Two main genetic pathways lead to the transformation of chronic lymphocytic leukemia to Richter syndrome
33. Identification in CLL of circulating intraclonal subgroups with varying B-cell receptor expression and function
34. Biological and clinical insight from analysis of the tumor B-cell 2 receptor structure and function in chronic lymphocytic Leukemia
35. S1P1 expression is controlled by the pro-oxidant activity of p66Shc and is impaired in B-CLL patients with unfavorable prognosis
36. Disruption of BIRC3 associates with fludarabine chemorefractoriness in TP53 wild-type chronic lymphocytic leukemia
37. Mutations of NOTCH1 are an independent predictor of survival in chronic lymphocytic leukemia
38. The genetics of Richter syndrome reveals disease heterogeneity and predicts survival after transformation
39. The host genetic background of DNA repair mechanisms is an independent predictor of survival in diffuse large B-cell lymphoma
40. Genome-wide DNA profiling of marginal zone lymphomas identifies subtype-specific lesions with an impact on the clinical outcome
41. The outcome of Chronic lymphocytic leukaemia patients with 97% IGHV gene identity to germline is distinct from cases with <97% identity and similar to those with 98% identity
42. Development of a distributed international patient data registry for hairy cell leukemia.
43. Angiopoietin-2 plasma dosage predicts time to first treatment and overall survival in chronic lymphocytic leukemia
44. Impaired expression of p66Shc, a novel regulator of B-cell survival, in chronic lymphocytic leukemia
45. The normal IGHV1-69–derived B-cell repertoire contains stereotypic patterns characteristic of unmutated CLL
46. Hairy cell leukemias with unmutated IGHV genes define the minor subset refractory to single-agent cladribine and with more aggressive behavior
47. BCL2 Inhibition in Refractory Hairy-Cell Leukemia.
48. Guideline for diagnosis and management of Hairy Cell Leukaemia (HCL) and Hairy Cell Variant (HCL-V) a British Society for Haematology Guideline
49. TP53 Mutations Identified Using NGS Comprise the Overwhelming Majority of TP53 Disruptions in CLL: Results From a Multicentre Study.
50. Promoter methylation patterns in Richter syndrome affect stem-cell maintenance and cell cycle regulation and differ from de novo diffuse large B-cell lymphoma
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