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42 results on '"Leventhal, Andrew"'

24. Fontan-Associated Dyslipidemia.

Author

Lubert, Adam M., Alsaied, Tarek, Palermo, Joseph J., Anwar, Nadeem, Urbina, Elaine M., Brown, Nicole M., Alexander, Craig, Almeneisi, Hassan, Fred Wu, Leventhal, Andrew R., Aldweib, Nael, Mendelson, Michael, Opotowsky, Alexander R., and Wu, Fred

Published
2021
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29. MUTUALS: AN AREA OF LEGAL CLIMATE CHANGE.

Author

MUTH, KARL T. and LEVENTHAL, ANDREW

Subjects
STOCK ownership, ORGANIZATIONAL structure, DECISION making in business, LAW firms, BANKING industry
Abstract

Underappreciated in its importance and often-misunderstood in its implications, the choice between a company limited by shares and a company organized as a mutual is an important decision in sectors ranging from agriculture to banking to insurance. Adding gravity to this particular decision is the difficulty and enormous cost of corporate metamorphosis between company types later in the company's life. The authors examine the history of the mutual form, its popularity's rise and fall during the twentieth century, and its advantages and disadvantages in today's environment. [ABSTRACT FROM AUTHOR]

Published
2018

35. A "Cat"‐astrophic Case of Bartonella henselae infective endocarditis followed by cardiac transplantation salvage therapy.

Author

Kuan, William, Dulnuan, Kenneth, Guglin, Maya E., El Haddad, Hanine, Kolodziej, Andrew R., Leventhal, Andrew, and Rajagopalan, Navin

Subjects
BARTONELLA henselae, SALVAGE therapy, HEART transplantation, TRANSPLANTATION of organs, tissues, etc., INFECTIVE endocarditis, INTRACELLULAR pathogens
Abstract

To our knowledge, no cases of Bartonella henselae endocarditis leading to subsequent heart transplantation salvage therapy have been published. We present a case of a 29‐year‐old man with cat‐inflicted B henselae endocarditis and concurrent worsening heart failure, who then underwent successful heart transplantation 50 days following diagnosis. Treatment and monitoring strategies used in this patient are discussed. Furthermore, we review literature related to heart transplantation salvage therapy for endocarditis due to other intracellular pathogens. [ABSTRACT FROM AUTHOR]

Published
2019
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37. INCIDENCE OF OBESITY, ACQUIRED CARDIOVASCULAR RISK FACTORS & ADVERSE CLINICAL OUTCOMES IN PATIENTS WITH REPAIREDTETRALOGY OFFALLOT(TOF)INCIDENCE OF OBESITY, ACQUIRED CARDIOVASCULAR RISK FACTORS & ADVERSE CLINICAL OUTCOMES IN PATIENTS WITH REPAIREDTETRALOGY OF FALLOT

Author

Harrell, Justin M., Sorrell, Vincent, Leventhal, Andrew, Ramachandran, Preeti, and Schneider, Douglas

Subjects
*CARDIOVASCULAR diseases risk factors, *TREATMENT effectiveness, *OBESITY, *TETRALOGY of Fallot
Published
2021
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42. Acid sphingomyelinase promotes lipoprotein retention within early atheromata and accelerates lesion progression.

Author

Devlin CM, Leventhal AR, Kuriakose G, Schuchman EH, Williams KJ, and Tabas I

Subjects
Animals, Aorta, Thoracic enzymology, Apolipoproteins E genetics, Apolipoproteins E metabolism, Atherosclerosis etiology, Atherosclerosis pathology, Atherosclerosis prevention & control, Dietary Fats, Disease Models, Animal, Disease Progression, Female, Foam Cells metabolism, Male, Mice, Mice, Inbred C57BL, Mice, Knockout, Microscopy, Fluorescence, Receptors, LDL genetics, Receptors, LDL metabolism, Sphingomyelin Phosphodiesterase deficiency, Sphingomyelin Phosphodiesterase genetics, Atherosclerosis enzymology, Lipoproteins metabolism, Sphingomyelin Phosphodiesterase metabolism
Abstract

Objective: The key initial step in atherogenesis is the subendothelial retention of apolipoprotein B-containing lipoproteins. Acid sphingomyelinase (acid SMase), an enzyme present extracellularly within the arterial wall, strongly enhances lipoprotein retention in model systems in vitro, and retained lipoproteins in human plaques are enriched in ceramide, a product of SMase. We now sought to test a direct causative role for acid SMase in lipoprotein retention and atherogenesis in vivo., Methods and Results: We studied atherogenesis and lipoprotein retention in Asm(-/-) versus Asm(+/+) mice on the Apoe(-/-) and Ldlr(-/-) backgrounds. Asm(-/-);Apoe(-/-) mice had a approximately 40% to 50% decrease in early foam cell aortic root lesion area compared with Asm(+/+);Apoe(-/-) mice (P<0.05) despite no difference in plasma cholesterol or lipoproteins. To assay lipoprotein retention in vivo, the two groups of mice were injected with fluorescently labeled Apoe(-/-) lipoproteins. Early foam cell lesions of Asm(-/-);Apoe(-/-) mice showed a striking 87% reduction in lipoprotein trapping (P<0.0001) compared with Asm(+/+);Apoe(-/-) lesions. Similar results were obtained with Ldlr(-/-) mice, including an 81% reduction in lipoprotein retention within Asm(-/-);Ldlr(-/-) lesions compared with Asm(+/+);Ldlr(-/-) lesions (P<0.0005)., Conclusions: These findings support a causal role for acid SMase in lipoprotein retention and lesion progression and provides further support for the response-to-retention model of atherogenesis.

Published
2008
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