1. [Sensory syndrome and aphasia after left insular infarct].
- Author
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Peskine A, Galland A, Chounlamountry AW, and Pradat-Diehl P
- Subjects
- Adult, Aphasia pathology, Cerebral Infarction pathology, Female, Functional Laterality physiology, Humans, Magnetic Resonance Imaging, Paresis etiology, Sensation Disorders pathology, Stroke pathology, Subarachnoid Hemorrhage complications, Thalamus pathology, Tomography, X-Ray Computed, Aphasia etiology, Aphasia psychology, Cerebral Infarction complications, Cerebral Infarction psychology, Sensation Disorders etiology, Sensation Disorders psychology, Stroke etiology, Stroke psychology
- Abstract
Introduction: Sensorial impairment without hemiplegia is usually caused by a thalamic lesion., Case Report: A 28-year-old woman presented with hemianesthesia associated with aphasia following a left insular lesion, subsequent to subarachnoid hemorrhage. Brain MRI Flair sequence revealed a high intensity signal in the left insular and frontal subcortical regions. Insular infarct was diagnosed, associated with hemorrhagic sequelae., Discussion/conclusion: Study of the normal and pathologic insular cortex suggest several implications of the region in somatosensory and language functions. However, the insular cortex has been mainly associated with central pain. Lasting objective hypoesthesia has been very rarely documented. The left insular cortex has also been implicated in speech apraxia but our patient presented with fluent aphasia mostly affecting the rhythm of speech, as it has been observed in thalamic aphasia.
- Published
- 2008
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