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Start Over Descriptor "gastrointestinal manifestations" Language spanish; castilian
4 results on '"gastrointestinal manifestations"'

1. Physical therapy of gastrointestinal non-motor manifestations of Parkinson's disease according to coprological methods of nutrition assessment

Author

Andriy Labinsky

Subjects
parkinson's disease, gastrointestinal manifestations, physical therapy, Education, Sports, GV557-1198.995, Medicine
Abstract

Topicality. Determination of the level of efficiency of using physical therapy with nutritional correction of nutrition in gastrointestinal non-motor manifestations of Parkinson's disease. Objectives of the study: to analyze and systematize modern scientific and methodological knowledge and results of practical experience of domestic and foreign researchers on the use of physical therapy with nutritional correction of nutrition in gastrointestinal non-motor manifestations of Parkinson's disease - to study and evaluate the status of patients by nutritional methods non-motor manifestations of Parkinson's disease. Research results. The results of the examination of patients and their nutritional status revealed a significant improvement in the condition of patients after the course of physical therapy with the appointment of a special diet for biorhythms with the onset of persistent hunger. The results were confirmed by scopological studies, during which they found a significant reduction in the number of undigested substances in the stool microscopy of patients after a course of physical therapy against the background of nutritional correction. Conclusions. The analysis of the obtained results shows that physical therapy against the background of nutritional correction improves the condition of patients with gastrointestinal non-motor manifestations of Parkinson's disease according to the data of coprological study. The developed program of physical therapy against the background of a special diet is effective for the correction of gastrointestinal non-motor manifestations of Parkinson's disease and may be recommended for wider introduction.

Published
2017
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2. Manifestaciones Extra pulmonares de la infección por SARS-CoV-2 en Pediatría. Revisión

Author

Ricardo Meza Brítez, Ana Buongermini, Leticia Ramírez Pastore, Celia Martínez de Cuellar, María Lezcano, Zoilo Morel Ayala, Mabel Zacur de Jiménez, and Junior Greco

Subjects
Pediatrics, medicine.medical_specialty, Pediatric Multisystemic Inflammatory Syndrome, Kawasaki disease, business.industry, dermatological manifestations, Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2), SARS-Cov-2, Síndrome Inflamatorio Multisistémico pediátrico, manifestaciones neurológicas, General Medicine, Extra pulmonary, manifestaciones dermatológicas, manifestaciones gastrointestinales, gastrointestinal manifestations, Enfermedad de Kawasaki, neurological manifestations, medicine, business, Covid-19
Abstract

RESUMEN Las manifestaciones clínicas del SARS-Cov-2 en niños difieren a la de los adultos, con afección respiratoria, gastrointestinal, dermatológica y/o cardiovascular. La mayoría de los niños son asintomáticos o presentan síntomas leves de la infección por COVID-19. Sin embargo, en los últimos meses se ha identificado un pequeño número de niños que desarrollan respuesta inflamatoria sistémica significativa. A continuación, realizamos una revisión sobre las manifestaciones extrapulmonares del SARS-Cov-2. ABSTRACT The clinical manifestations of SARS-Cov-2 in children differ from that of adults, with respiratory, gastrointestinal, dermatological and / or cardiovascular conditions. Most children are asymptomatic or have mild symptoms of COVID-19 infection. However, in recent months, a small number of children have been identified who develop a significant systemic inflammatory response. We review the extrapulmonary manifestations of SARS-Cov-2.

Published
2020

3. [Infectious and inflammatory gastrointestinal manifestations of chronic granulomatous disease].

Author

Toledo M, Campos A, Scheffler-Mendoza S, León-Lara X, Onuma-Zamayoa H, Espinosa S, Yamazaki-Nakashimada MA, and Blancas-Galicia L

Subjects
Humans, Inflammation etiology, Macrophages, NADPH Oxidases, Neutrophils, Granulomatous Disease, Chronic
Abstract

Chronic granulomatous disease (CGD) is an inborn error of immunity caused by a defect in one of the components of the NADPH oxidase complex, which is responsible for generating reactive oxygen species (ROS) during the respiratory burst in phagocytes. The absence of ROS produced by NADPH oxidase in neutrophils and in macrophages leads to greater susceptibility to certain bacterial and fungal infections, and also to inflammatory manifestations due to a deregulated inflammatory response, which suggests that the ability to adequately regulate inflammatory signaling depends on ROS produced by NADPH oxidase. The disease course in patients with X-linked CGD is more severe, with recurrent invasive infections; in contrast, patients with non-classic CGD do not present invasive bacterial or fungal infections, but have more prominent inflammatory manifestations. The most frequent gastrointestinal manifestations are stomatitis, gingivitis, chronic diarrhea, liver abscesses that are similar to inflammatory bowel disease (IBD), and granulomas that can cause obstruction or stenosis in the esophagus, stomach or intestine. It has been observed that the deficiency of p40phox and ROS (non-classic CGD) are associated with greater susceptibility to colitis and the development of severe inflammation; therefore, it is presented that these proteins participate in the resolution of inflammation. In general, the inflammatory findings in CGD, including gastrointestinal manifestations, are seldom described. In international cohorts, manifestations that are similar to IBD are reported in up to 58% of patients with CGD; however, in the only Mexican cohort, its finding is described in only 4 out of 93 patients (4.3%). In this review, we summarize the gastrointestinal clinical findings of CGD, including infectious and inflammatory manifestations, emphasizing on the latter.

Published
2021
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4. GASTROINTESTINAL MICROBIOTA AEROBIC IN CHILDREN WITH AUTISM SPECTRUM DISORDERS. PRELIMINARY STUDY

Author

Moreno, Xiomara, Santamaria, Giancarlo, Sánchez, Roger, e la Torre, Beatriz, Garcés, Fátima, Hernández, Celsy, Martínez, Clara, Márquez, Ana, and López, Karolina

Subjects
Autism Spectrum Disorders, Microbiota Gastrointestinal aeróbica, Neurodevelopmental Disorders, Disbiosis, Manifestaciones gastrointestinales, Dysbiosis, Gastrointestinal Manifestations, robic Microbiota Gastrointestinal, Trastornos del Espectro Autista, Alteraciones en el Neurodesarrollo
Abstract

Introducción: la alteración de la microbiota intestinal o Dis- biosis ha sido implicada en los cambios de comportamiento del neurodesarrollo y problemas gastrointestinales en pacientes con trastorno del espectro autista (TEA). Objetivo: evaluar la micro- biota intestinal aeróbica (MGIA) y clasificarla en beneficiosa, transitoria y enteropatógena en niños con TEA en la Unidad de Autismo-Maternidad Concepción Palacios. Pacientes y métodos: desde el 26/02/2015 al 12/05/2015 se estudiaron de forma experimental y prospectiva 39 niños diagnosticados con TEA; en el estudio de la MGIA se utilizaron muestras de heces. Se aplicó una encuesta para recopilar datos epidemiológicos, clínicos y comportamientos del neurodesarrollo. Se propone la clasificación de severidad de la disbiosis en grado I,II,III o ausente para la evaluación de la MGIA. Resultados: Fueron 27 niños (69,23%) y 12 niñas (30,77%), con una edad media de 6,3. Disbiosis 31 (79,5%), Disbiosis ausente 8 (20,5%). Según el grado de disbiosis, 5 (16,13%) Grado I, 7 (22,58%) Grado II, 19 (61,29%) Grado III. Los principales agentes causales de disbiosis fueron Klebsiella spp. 16, Proteus mirabilis 8, Streptococcus sp, 6, Serratia marcensces 5, Candida spp. 4. Dos niños presentaron Campylobacter coli como MGIA patógena. Manifestaciones gastrointestinales: 25,80% dolor abdominal, 16,13% diarrea y 38.7% estreñimiento. Trastornos del neurodesarrollo: 50% aleteos, 34% autoagresión, 61% berrinches, insomnio un 34.3%. Conclusiones: Se hace necesario comparar esta investigación con un grupo de niños sin TEA para confirmar que la presencia de disbiosis como causante de alteración de la MGIA se presenta con más frecuencia en niños con TEA. Background: altering the intestinal microbiota or Dysbiosis has been implicated in the changes the behavior of neurodevelopmen- tal and gastrointestinal problems in patients with autism spectrum disorder (ASD). Objective: To evaluate aerobic intestinal micro- biota (AMGI) and rank it beneficial, transitory and enteropathogenic in children with ASD, the Autism Unit-Maternidad Concepcion Palacios. Patients and Methods: From 26/02/2015 to 05/12/2015 were studied experimentally and prospectively 39 children diagnosed with ASD; in this study the AMGI stool samples were used. A survey to collect epidemiological, clinical and neurodevelopmental behavior was applied. Severity classification dysbiosis proposed in grade I, II, III or absent for evaluating the AMGI. Results: There were 27 kids (69.23%) and 12 girls (30.77%) with a mean age of 6.3. Dysbiosis 31 (79.5%), Dysbiosis absent eight (20.5%). Depending on the degree of dysbiosis, 5 (16.13%) Grade I, 7 (22.58%) Grade II, 19 (61.29%) Grade III. The main causative agents of dysbiosis were Klebsiella spp. 16, Proteus mirabilis 8, Streptococcus sp. 6, Serratia marcensces 5, Candida spp. 4. Two children presented MGIA pathogenic Campylobacter coli. Gastrointestinal symptoms: 25,80% abdominal pain, 16.13% diarrhea and 38.7% constipation. Neurodevelopmental disorders: 50% flapping, 34% self-harm, 61% tantrums and 34.3% insomnia. Conclusion: It is necessary to compare this research with a group of children without ASD to confirm the presence of dysbiosis to cau- se impaired MGIA occurs most often in children with ASD.

Published
2015

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