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Brown-Sequard Sendromu; ısıya ve ağrıya duyarlılığın kontralateral kaybının yanı sıra motor işlevinin ve propriyosepsiyonun yüksek oranda ipsilateral kaybı ile karakterize edilen tamamlanmamış omurilik yaralanmalarının bir tipidir. Yaralanma vakaları sonucunda ortaya çıkan dengedeki hasarlar bireyi omur kırıklıklarına veya başka omurilik yaralanmalararına neden olabilecek düşmelere meyilli hale getirebilir. Bu vaka rapor çalışmamızda önce keskin bir bıçakla boyuna hasar alması ile "Brown-Seqard Sendromu" oluşmuş daha sonra ise bu sendromun neden olduğu düşme sonucu LV1 kırığının meydana gelmesinden dolayı bir başka tamamlanmamış omurilik yaralanması ile "Cauda Equina Sendromu" oluşmuş bireyi sunmaktayız. Kırık, pedikül vida ile tespit edildi ve hastaya işlevsel aktiviteleri tek başına yapabilecek maksimum kazancı sağlamak için rehabilitasyon programı uygulandı.

Brown-Séquard Syndrome is a type of Incomplete Spinal Cord Injury characterized by a relatively greater ipsilateral loss of proprioception and motor function, with contralateral loss of pain and temperature sensations. The residual deficits in balance produced by such injury may render a person liable to fall that may result in vertebral fracture and another injury to the spinal cord. We present here a case who initially had Brown-Séquard Syndrome due to penetrating knife injury to the neck and later on developed Cauda Equina Syndrome (another Incomplete Spinal Cord Injury) due to fractured LV1 following a fall. The fracture was fixed through Pedicle Screws and the patient underwent effective rehabilitation to gain maximum achievable independence in functional activities. [Cukurova Med J 2014; 39(2.000): 392-398]

Cauda equina syndrome is a neurological state with bilateral ischiatic pain, lower extremity weakness, and bladder and bowel dysfunction due to multiple compressive neuropathies of the lumbar and sacral nerves. Even though the relationship between cauda equina syndrome and voiding dysfunctions is a well known fact, data on the clinical management of the condition is limited. The aim of this paper is to review the pathophysiological relationship between voiding dysfunctions and cauda equina syndrome and to refresh our knowledge about the clinical approach to this group of patients. [ABSTRACT FROM AUTHOR]